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Arrhythmias
- Atrial Flutter
- Atrial Fibrillation
- Ventricular Tachycardia
- Ventricular Fibrillation
Atrial Flutter
What is it?
Atrial flutter is a relatively common abnormal heart rhythm where the atria beat quickly, but evenly. This is similar to atrial fibrillation, but the atria beat quickly and unevenly.
Atrial flutter and fibrillation always have some degree of atrial-ventricular (AV) block with a 2:1 pattern meaning the rate of the ventricles is usually 150 beats per minute while the atria beat at 300 beats per minute. In other words, every other atrial beat reaches the ventricle.
Both of these arrhythmias impair blood flow out of the heart (cardiac output) and promote clot (thrombus) formation.
How does a normal heart beat?
Heart muscle cells are activated by electrical impulses that cause them to contract regularly and in sync. This contraction produces a heartbeat, allowing blood to be pumped out to the entire body.
Electrical impulses originate from specialized cells called the sinoatrial (SA) node, which is the hearts natural pacemaker.
The SA node is located in the upper right chamber of the heart, the right atrium. From the SA node, the impulse spreads across the upper chambers of the heart to reach the atrial ventricular (AV) node located between the atria and lower ventricles. After leaving the AV node, impulses spread across the pumping chambers of the heart, the ventricles.
As impulses are spread along the heart, the cardiac muscle cells are stimulated to contract, producing a heartbeat.

How does atrial flutter occur?
Sometimes the electrical impulses ‘short circuit’ and travel across the heart in an abnormal way. Abnormal electrical pathways produce irregular heartbeats and rhythms known as arrhythmias.
Arrhythmias occur when other areas of the heart, other than the SA node, begin sending out their own impulses and take over the pacemaker function of the SA node. The sites that take over could be from any area of the atria, AV node, or ventricles.
The rapid fluttering of the atria does not allow the complete filling of the ventricles, that pumps blood out to the entire body. Symptoms of atrial flutter such as lightheadedness or dizziness may be felt.
Also, some amount of blood remains in the chamber that leads to clot formation.

How is it classified?
Atrial flutter is classified into two types:
- Type I (Typical) - more common, atrial flutter rate 240-340 beats/minute, circular (clockwise or counterclockwise) re-entry of electrical patterns in the atria
- Type II (Atypical) – rare, atrial flutter rate 340-440 beats/minute, different re-entry pathways
Most patients with atrial flutter will have only one of these. Rarely a patient may present with both types, although they can only occur one at a time.
What are the causes?
Atrial flutter can be due to abnormalities or diseases of the heart, or by diseases elsewhere in the body affecting the heart. Atrial flutter may occur after open heart surgery or substance abuse.
Heart abnormalities and diseases include:
- Congenital heart defects (heart defect from birth)
- Coronary artery disease (decreased blood supply to the heart) or atherosclerosis

- High blood pressure
- Heart attack
- Cardiomyopathy and congestive heart failure
- Rheumatic fever and pericarditis (inflammation of the heart)
- Abnormalities of the heart valves, especially mitral stenosis and mitral regurgitation
- Hypertrophy (enlarged heart)
Diseases elsewhere in the body affecting the heart:
- Hyperthyroidism
- Pulmonary embolism (blood clot in lung)
- Chronic obstructive pulmonary disease (COPD) and emphysema
- Sleep apnea
Substance abuse:
- Consuming large amounts of alcohol (binge drinking)
- Cocaine, amphetamines, diet pills, caffeine, or cold medicines
- Carbon monoxide intoxication
Surgery
- First week following open heart surgery
Who is at risk?
The following are at risk of developing atrial flutter:
- Increasing age
- Men are at more risk of developing atrial flutter than women
- Family history of atrial flutter
- Drinking alcohol or binge drinking
- Atrial flutter is greatest when an underlying heart disease is associated with left atrial enlargement, left ventricular or biventricular failure
- Heart failure patients and COPD patients
- Other chronic conditions including thyroid disorders and sleep apnea
What are the symptoms?
Some patients may be unaware of their heart beating abnormally.
Although characteristic symptoms include:
- Palpitations (pounding sensation in the chest)
- Fluttering sensation in the chest
- Shortness of breath
- Anxiety

- Weakness
- Dizziness, light-headedness
- Fainting
- Confusion
- Fatigue
- Intolerance to exercise
- Nausea
- Nervousness
- Swelling of the legs or abdomen
What are the complications?
The complications of atrial flutter include:
- Syncope (fainting)
- Heart failure
- Thrombus (clot formation)
- Stroke
When should I seek medical care?
If you experience symptoms of atrial flutter, call your physician.
If you have already been diagnosed and being treated for atrial flutter, seek medical help at the hospital as soon as possible if you have the following symptoms:
- Severe chest pain
- Feeling faint or light-headed
- Have an actual episode of fainting
How is it diagnosed?
The diagnosis of atrial flutter will be based on your history, physical examination, and certain tests.
- Patient history
Most patients will give a history of characteristic symptoms like palpitations, dizziness, fatigue, shortness of breath, etc.
2. Physical Examination

You will be examined for signs of atrial flutter including:
- Rapid, irregular, fast pulse
- Heart rate of 100-175 (Normal heart rate = 60-100)
- Normal to low blood pressure
- Varying heart sounds heard over chest
3. Tests
Certain tests can be performed to determine the cause and severity of your condition including:
- Blood tests- to check potassium and thyroid hormone levels
- Electrocardiogram- this is a very useful and simple test that records the electrical activity of the heart and reveals atrial flutter or atrial fibrillation. Also shows signs of heart attack, conduction abnormalities, hypertrophy, and electrolyte imbalances.

- Chest X-ray- a simple test performed in our office showing the size and shape of the heart. Also reveals the presence of fluid in the lungs.
- Echocardiogram (ECHO) - a noninvasive test performed in our office to determine the size and function of the heart’s chambers and the structure and function of the heart valves.
- 24-Hour Holter monitor- records the electrical events of the heart during your normal daily activities. This is useful in detecting evidence of arrhythmias.
- Event monitor- If symptoms of arrhythmia are infrequent and do not occur within 24 hours, an event recorder may be ordered for you for up to 30 days.
- Stress test- determines how well the heart works during exercise.
How is it treated?
Atrial flutter treatment goals include restoring the heart to normal rhythm, regulating heart rate, preventing blood clot formation, and treating the underlying cause.
This is done by the use of medications and procedures.
Medications
Some medications are used to slow an irregularly fast heart beat. These decrease the heart rate by slowing the conduction of impulses through the AV node.
These drugs include:
- Beta Blockers
- Calcium channel blockers
- Digoxin
Medications are used to reduce the frequency and duration of atrial flutter and prevent future episodes.
These include:
- Amidarone
- Sotalol
- Propafenone
- Flecainide
Medications are used to prevent the formation of blood clots.
These drugs include:
- Heparin
- Warfarin
- Dabigatran
- Aspirin
- Clopidogrel
Procedures
-
Cardioversion- atrial flutter is sensitive to a low electrical energy direct-current (DC) or medical cardioversion (using an antiarrhythmic drug). DC cardioversion converts atrial flutter into a normal sinus rhythm with high a success rate. Cardioversion may be delayed until starting treatment with an anticoagulant. This medication is usually given for 3-4 weeks, allowing pre-existing clots to resolve. A transesophageal echocardiogram (TEE) is an alternative way of delaying cardioversion, if no blood clots are present, cardioversion may be started. A mild anesthetic is first given, pads are applied over the chest, and then the heart will be shocked into the correct rhythm.

-
Catheter radiofrequency ablation- is a hospital procedure done to destroy areas on the heart that are causing irregular heart rhythms. A thin flexible tube is inserted into a blood vessel to access the heart. Heat is applied to the tip of the catheter to permanently destroy small areas of abnormal heart tissue. The damaged tissue is no longer capable of generating electrical impulses. A pacemaker may be placed after this procedure
-
Pacemaker or ICD- a small implantable device used to regulate the heart rate and rhythm. Therefore, the heart contracts in a regular way. An ICD shocks the heart out of the dangerous rhythm.
What is the prognosis?
Atrial flutter can be controlled with treatment.
With treatment, many patients do well.
How can I prevent this?
Atrial flutter can not always be prevented, but there are ways of reducing the risk of developing atrial flutter.
Leading a healthy lifestyle may reduce the chance of coronary artery disease, which can cause atrial flutter.
Some ways you can do this is by:
- No smoking!
- Be physically active. Engage in moderate physical activity for at least 30 minutes a day
- Eat nutritious foods low in cholesterol and fats
- Maintain a BMI below 25
- Control high blood pressure, cholesterol, and sugar
Updated March 20th, 2012 -JS
For further information please contact Cardiovascular Interventions, PA at 407-894-4880
Atrial Fibrillation
What is it?
Atrial fibrillation is a relatively common abnormal heart rhythm where the atria beat quickly, irregularly, and unevenly.
Atrial fibrillation always has some degree of atrial-ventricular (AV) block with a 2:1 pattern meaning the rate of the ventricles is usually 150 beats per minute while the atria beat at 300 beats per minute. In other words, every other atrial beat reaches the ventricle.
This arrhythmia impairs blood flow out of the heart (cardiac output) and promotes clot (thrombus) formation.
How does a normal heart beat?
Heart muscle cells are activated by electrical impulses that cause them to contract regularly and in sync. This contraction produces a heartbeat, allowing blood to be pumped out to the entire body.
Electrical impulses originate from specialized cells called the sinoatrial (SA) node, which is the hearts natural pacemaker.
The SA node is located in the upper right chamber of the heart, the right atrium. From the SA node, the impulse spreads across the upper chambers of the heart to reach the atrial ventricular (AV) node located between the atria and lower ventricles. After leaving the AV node, impulses spread across the pumping chambers of the heart, the ventricles.
As impulses are spread along the heart, the cardiac muscle cells are stimulated to contract, producing a heartbeat.

How does atrial fibrillation occur?
In atrial fibrillation, chaotic electrical signals travel across the heart in an abnormal way. Abnormal electrical pathways produce irregular heartbeats and rhythms known as arrhythmias.
Arrhythmias occur when other areas of the heart, other than the SA node, begin sending out their own impulses and take over the pacemaker function of the SA node. The sites that take over could be from any area of the atria, AV node, or ventricles.
The rapid fibrillation of the atria does not allow the complete filling of the ventricles, that pumps blood out to the entire body. Symptoms of atrial fibrillation such as lightheadedness or dizziness may be felt.
Also, some amount of blood remains in the chamber that leads to clot formation.

What are the types?
Atrial fibrillation can occur in different patterns.
These include:
- Paroxysmal (intermittent) atrial fibrillation: recurrent (2 or more) episodes, spontaneously terminates and converts back to normal sinus rhythm within a week, lasting for less than 24 hours
- Persistent atrial fibrillation: occurs in episodes, does not spontaneously convert back to sinus rhythm, and is unrelated to a reversible cause.
- Permanent atrial fibrillation: the heart is always in atrial fibrillation and is unrelated to a reversible cause.
What are the causes?
Atrial fibrillation can be due to abnormalities or diseases of the heart, or by diseases elsewhere in the body affecting the heart. Atrial fibrillation may occur after open heart surgery or substance abuse.
Heart abnormalities and diseases include:
- Congenital heart defects (heart defect from birth)

- Coronary artery disease (decreased blood supply to the heart) or atherosclerosis
- High blood pressure
- Heart attack
- Cardiomyopathy and congestive heart failure
- Rheumatic fever and pericarditis (inflammation of the heart)
- Abnormalities of the heart valves, especially mitral stenosis and mitral regurgitation
- Hypertrophy (enlarged heart)
- Sick sinus syndrome
Diseases elsewhere in the body affecting the heart:
- Hyperthyroidism
- Pulmonary embolism (blood clot in lung)
- Chronic obstructive pulmonary disease (COPD) and emphysema
- Sleep apnea
Substance abuse:
- Consuming large amounts of alcohol (binge drinking)
- Cocaine, amphetamines, diet pills, caffeine, or cold medicines
- Tobacco
- Carbon monoxide intoxication
Surgery
- First week following open heart surgery
Medications
- Theophylline
Who is at risk?
The following are at risk of developing atrial fibrillation:
- Increasing age
- Men are at more risk of developing atrial fibrillation than women
- Family history of atrial fibrillation
- Drinking alcohol or binge drinking
- Atrial fibrillation is greatest when an underlying heart disease is associated with left atrial enlargement, left ventricular or biventricular failure
- Heart failure patients and COPD patients
- Other chronic conditions including thyroid disorders and sleep apnea
What are the symptoms?
Some patients may be unaware of their heart beating abnormally.
Although characteristic symptoms include:
- Palpitations (pounding sensation in the chest)
- Chest pain
- Shortness of breath
- Anxiety

- Weakness
- Dizziness, light-headedness
- Fainting
- Confusion
- Fatigue
- Intolerance to exercise
- Nausea
- Nervousness
- Swelling of the legs or abdomen
What are the complications?
The complications of atrial fibrillation include:
- Syncope (fainting)
- Heart failure
- Thrombus (clot formation)
- Stroke
When should I seek medical care?
If you experience symptoms of atrial fibrillation, call your physician.
If you have already been diagnosed and being treated for atrial fibrillation, seek medical help at the hospital as soon as possible if you have the following symptoms:
- Severe chest pain
- Feeling faint or light-headed
- Have an actual episode of fainting
How is it diagnosed?
The diagnosis of atrial fibrillation will be based on your history, physical examination, and certain tests.
1. Patient history
Most patients will give a history of characteristic symptoms like palpitations, dizziness, fatigue, shortness of breath, etc.
2. Physical Examination
You will be examined for signs of atrial fibrillation including: 
- Rapid, irregular, fast pulse
- Heart rate of 100-175 (Normal heart rate = 60-100)
- Normal to low blood pressure
- Varying heart sounds heard over chest
3. Tests
Certain tests can be performed to determine the cause and severity of your condition including:
- Blood tests- to check potassium and thyroid hormone levels
- Electrocardiogram- this is a very useful and simple test that records the electrical activity of the heart and reveals atrial fibrillation. Also shows signs of heart attack, conduction abnormalities, hypertrophy, and electrolyte imbalances.

-
Chest X-ray- a simple test performed in our office showing the size and shape of the heart. Also reveals the presence of fluid in the lungs.
-
Echocardiogram (ECHO) - a noninvasive test performed in our office to determine the size and function of the heart’s chambers and the structure and function of the heart valves.
-
24-Hour Holter monitor- records the electrical events of the heart during your normal daily activities. This is useful in detecting evidence of arrhythmias.
- Event monitor- If symptoms of arrhythmia are infrequent and do not occur within 24 hours, an event recorder may be ordered for you for up to 30 days.
- Stress test- determines how well the heart works during exercise.
How is it treated?
Atrial fibrillation treatment goals include restoring the heart to normal rhythm, regulating heart rate, preventing blood clot formation, and treating the underlying cause.
This is done by the use of medications and procedures.
1. Medications
Some medications are used to slow an irregularly fast heart beat. These decrease the heart rate by slowing the conduction of impulses through the AV node.
These drugs include:
- Beta Blockers
- Calcium channel blockers
- Digoxin
Medications are used to reduce the frequency and duration of atrial fibrillation and prevent future episodes.
These include:
- Amidarone
- Sotalol
- Propafenone
- Flecainide
Medications are used to prevent the formation of blood clots.
These drugs include:
- Heparin
- Warfarin
- Dabigatran
- Aspirin
- Clopidogrel
2. Procedures
-
Cardioversion- atrial fibrillation is sensitive to a low electrical energy direct-current (DC) or medical cardioversion (using an antiarrhythmic drug). DC cardioversion converts atrial fibrillation into a normal sinus rhythm with high a success rate. Cardioversion may be delayed until starting treatment with an anticoagulant. This medication is usually given for 3-4 weeks, allowing preexisting clots to resolve. A transesophageal echocardiogram (TEE) is an alternative way of delaying cardioversion, if no blood clots are present, cardioversion may be started. A mild anesthetic is first given, pads are applied over the chest, and then the heart will be shocked into the correct rhythm.

-
Catheter radiofrequency ablation- is a hospital procedure done to destroy areas on the heart that are causing irregular heart rhythms. A thin flexible tube is inserted into a blood vessel to access the heart. Heat is applied to the tip of the catheter to permanently destroy small areas of abnormal heart tissue. The damaged tissue is no longer capable of generating electrical impulses. A pacemaker may be placed after this procedure
-
Pacemaker or ICD- a small implantable device used to regulate the heart rate and rhythm. Therefore, the heart contracts in a regular way. An ICD shocks the heart out of the dangerous rhythm.
What is the prognosis?
Atrial fibrillation can be controlled with treatment.
With treatment, many patients do well.
However, atrial fibrillation tends to return and get worse. It may come back even with treatment.
How can I prevent this?
Atrial fibrillation can not always be prevented, but there are ways of reducing the risk of developing atrial fibrillation.
Leading a healthy lifestyle may reduce the chance of coronary artery disease, which can cause atrial fibrillation.
Some ways you can do this is by:
- No smoking!
- Be physically active. Engage in moderate physical activity for at least 30 minutes a day
- Eat nutritious foods low in cholesterol and fats
- Maintain a BMI below 25
- Control high blood pressure, cholesterol, and sugar
Updated March 22nd, 2012 -JS
For further information please contact Cardiovascular Interventions, PA at 407-894-4880
Ventricular Tachycardia
What is it?
Ventricular tachycardia is an abnormal heart rhythm where the ventricles beat quickly, but evenly. This is similar to ventricular fibrillation, but the ventricles beat quickly and unevenly. The term tachycardia refers to a fast heart rate.
Ventricular tachycardia may develop as a complication of a heart attack because scar tissue can form in the ventricle muscle within days, months, and or years after a heart attack. Additionally, it can occur in patients with cardiomyopathy, heart failure, heart surgery, myocarditis, and valvular heart disease.
In ventricular tachycardia abnormal electrical signals originate in the ventricles, instead of the normal pathway from the atria down to the ventricles. If left untreated, ventricular tachycardia may lead to a dangerous and life-threatening ventricular fibrillation.
Both of these arrhythmias impair blood flow out of the heart (cardiac output) and are both medical emergencies.
How does a normal heart beat?
Heart muscle cells are activated by electrical impulses that cause them to contract regularly and in sync. This contraction produces a heartbeat, allowing blood to be pumped out to the entire body.
Electrical impulses originate from specialized cells called the sinoatrial (SA) node, which is the hearts natural pacemaker. The SA node is located in the upper right chamber of the heart, the right atrium. From the SA node, the impulse spreads across the upper chambers of the heart to reach the atrial ventricular (AV) node located between the atria and lower ventricles. After leaving the AV node, impulses spread across the pumping chambers of the heart, the ventricles. As impulses are spread along the heart, the cardiac muscle cells are stimulated to contract, producing a heartbeat.

How does ventricular tachycardia occur?
Sometimes the electrical impulses ‘short circuit’ and travel across the heart in an abnormal way. Abnormal electrical pathways produce irregular heartbeats and rhythms known as arrhythmias. Arrhythmias occur when other areas of the heart, other than the SA node, begin sending out their own impulses and take over the pacemaker function of the SA node. The sites that take over could be from any area of the atria, AV node, or ventricles, in this case the ventricles.
The rapid rate does not allow the complete filling of the ventricles to pump blood out to the entire body. Symptoms of ventricular tachycardia such as lightheadedness or dizziness may be felt.

How is it classified?
Ventricular tachycardia can be classified in several ways.
Based on the morphology:
- Monomorphic ventricular tachycardia: all the beats appear to match each other on an electrocardiogram (EKG). i.e. right ventricular outflow tract (RVOT)
- Polymorphic ventricular tachycardia: beat to beat variability seen on an EKG. i.e. torsades de pointes (“twisting of the points”)
By the duration of the episode:
- Non-sustained ventricular tachycardia- a fast heart rhythm which terminates within 30 seconds
- Sustained ventricular tachycardia- a fast heart rhythm that lasts for more than 30 seconds
On the basis of symptoms:
- Pulseless ventricular tachycardia- no effective cardiac output and no effective pulse, causing cardiac arrest. Pulseless VT is a shockable rhythm.
- Idiopathic ventricular tachycardia- occurs in younger individuals; cause of ventricular tachycardia is unknown, but presumed to be due to congenital causes
What are the causes?
Ventricular tachycardia can be due to abnormalities or diseases of the heart, or by diseases elsewhere in the body affecting the heart. Ventricular tachycardia may occur after open heart surgery, substance abuse, or medications.
Heart abnormalities and diseases include:
- Congenital heart defects (heart defect from birth)
- Coronary artery disease (decreased blood supply to the heart) or atherosclerosis

- High blood pressure
- Heart attack
- Cardiomyopathy and congestive heart failure
- Rheumatic fever and pericarditis (inflammation of the heart)
- Abnormalities of the heart valves, especially mitral stenosis and mitral regurgitation
- Hypertrophy (enlarged heart)
Diseases elsewhere in the body affecting the heart:
- Hyperthyroidism
- Pulmonary embolism (blood clot in lung)
- Chronic obstructive pulmonary disease (COPD) and emphysema
- Sleep apnea
- Electrolyte imbalances (low potassium/ magnesium levels)
Substance abuse:
- Consuming large amounts of alcohol (binge drinking)
- Cocaine, amphetamines, diet pills, caffeine, or cold medicines
- Tobacco
- Carbon monoxide intoxication
Surgery
- First week following open heart surgery
Medications
- Amiodarone
- Sotalol
Who is at risk?
The following are at risk of developing ventricular tachycardia:
- Increasing age
- Family history of ventricular tachycardia
- Drinking alcohol or binge drinking
- Use of recreational drugs
- Electrolyte imbalances
- Heavy caffeine use
- Heart failure patients and COPD patients
- Pericarditis (inflammation of the heart) and autoimmune diseases
- Other chronic conditions including thyroid disorders and sleep apnea
What are the symptoms?
Symptoms of ventricular tachycardia occur because the heart rate is beating so fast, blood pressure falls; therefore the heart can not pump enough oxygen rich blood to the body causing symptoms.
Some patients may be unaware of their heart beating abnormally. Although characteristic symptoms include:
- Dizziness
- Shortness of breath
- Lightheadedness
- Sensation of neck fullness
- Anxiety

- Rapid pulse rate
- Palpitations
- Chest pain (angina)
- Fainting (syncope)
- Confusion
- Fatigue
- Intolerance to exercise
- Nausea
- Nervousness
- Swelling of the legs or abdomen
What are the complications?
The complications of ventricular tachycardia include:
- Syncope (fainting)
- Heart failure
- Thrombus (clot formation)
- Stroke
- Sudden cardiac arrest
- Death
When should I seek medical care?
If you experience symptoms of ventricular tachycardia, call your physician.
If you faint, have difficulty in breathing, or have chest pain lasting more than a few minutes, call emergency care or call 911. These are urgent symptoms of a potentially fatal ventricular tachycardia and must seek emergency care immediately to avoid the risk of cardiac arrest and death.
How is it diagnosed?
The diagnosis of ventricular tachycardia will be based on your history, physical examination, and certain tests.
1. Patient history
Most patients will give a history of characteristic symptoms like palpitations, dizziness, fatigue, shortness of breath, etc.
2. Physical Examination
You will be examined for signs of ventricular tachycardia including: 
- Weak or no pulse
- Low blood pressure
- Rapid breathing
- Varying heart sounds heard over chest
3. Tests
Certain tests can be performed to determine the cause and severity of your condition including:
- Blood tests- to check potassium, magnesium, and thyroid hormone levels
- Electrocardiogram- this is a very useful and simple test that records the electrical activity of the heart and reveals ventricular tachycardia. Also shows signs of heart attack, conduction abnormalities, hypertrophy, and electrolyte imbalances.

- Chest X-ray- a simple test performed in our office showing the size and shape of the heart. Also reveals the presence of fluid in the lungs.
- Echocardiogram (ECHO) - a noninvasive test performed in our office to determine the size and function of the heart’s chambers and the structure and function of the heart valves.
- 24-Hour Holter monitor- records the electrical events of the heart during your normal daily activities. This is useful in detecting evidence of arrhythmias.
- Event monitor- If symptoms of arrhythmia are infrequent and do not occur within 24 hours, an event recorder may be ordered for you for up to 30 days.
- Stress test- determines how well the heart works during exercise.
How is it treated?
The treatment goals for tachycardias are to slow a fast heart rate when it occurs, prevent future episodes, minimize complications, and treat the underlying cause.
1. Stop a fast heart rate:
A fast heart rate may terminate spontaneously, or you may be able to slow your heart rate using simple physical methods.
Medications and procedures may be required in addition to these methods.
- Vagal maneuvers: this maneuver can be done during an episode of a fast heart beat. This includes coughing or bearing down as if having a bowel movement.
- Medications: anti-arrhythmic medication can be used to restore a normal heart rate. These medications include flecainide or propafenone.
- Cardioversion: if pulse is present, a shock is delivered to your heart through pads attached to the chest wall. The current interrupts the electrical impulses in the heart and restores a normal rhythm.
2. Preventing episodes tachycardia:
-
Catheter ablation: is a hospital procedure done to destroy areas on the heart that are causing irregular heart rhythms. A thin flexible tube is inserted into a blood vessel to access the heart. Heat is applied to the tip of the catheter to permanently destroy small areas of abnormal heart tissue. The damaged tissue is no longer capable of generating electrical impulses. A pacemaker may be placed after this procedure
-
Medications: anti-arrhythmic medications may prevent tachycardia, when taken regularly. These medications include: calcium channel blockers such as diltiazem and verapamil or beta blockers such as metoprolol and esmolol or anti-arrhythmic such as lidocaine and amidarone.
-
Pacemaker or ICD- a small implantable device used to regulate the heart rate and rhythm. Therefore, the heart contracts in a regular way. An ICD shocks the heart out of the dangerous rhythm.
3. Treating the underlying disease:
Treatment of an underlying condition attributing to tachycardia such as some form of heart disease or hyperthyroidism may prevent or minimize tachycardic episodes.
How can I prevent this?
Ventricular tachycardia can not always be prevented, but there are ways of reducing the risk of developing ventricular tachycardia.
Leading a healthy lifestyle may reduce the chance of coronary artery disease, which can cause ventricular tachycardia.
Some ways you can do this is by:
- No smoking!
- Drink in moderation

- Limit caffeine
- Avoid recreational drugs
- Be physically active. Engage in moderate physical activity for at least 30 minutes a day
- Eat nutritious foods low in cholesterol and fats
- Maintain a BMI below 25
- Control high blood pressure, cholesterol, and sugar
- Control stress
- Have regular physical exams and report any signs or symptoms to your physician
Updated April 2, 2012 -JS
For further information please contact Cardiovascular Interventions, PA at 407-894-4880
Ventricular Fibrillation
What is it?
Ventricular fibrillation is an abnormal heart rhythm that occurs when the heart beats so rapidly and irregularly that the heart muscle quivers and does not beat at all.
In ventricular fibrillation the ventricles produce many abnormal circuits, instead of following the normal electrical pathway from the atria down to the ventricles. This usually occurs due to damaged heart muscle.
The ventricles beat very quickly and irregularly, so blood is not pumped out of the heart to vital organs of the body. If the heart is not able to pump at all (cardiac arrest), death may occur if emergency treatment is not given to return the heart rhythm to normal.
How does a normal heart beat?
Heart muscle cells are activated by electrical impulses that cause them to contract regularly and in sync. This contraction produces a heartbeat, allowing blood to be pumped out to the entire body.
Electrical impulses originate from specialized cells called the sinoatrial (SA) node, which is the hearts natural pacemaker. The SA node is located in the upper right chamber of the heart, the right atrium. From the SA node, the impulse spreads across the upper chambers of the heart to reach the atrial ventricular (AV) node located between the atria and lower ventricles. After leaving the AV node, impulses spread across the pumping chambers of the heart, the ventricles. As impulses are spread along the heart, the cardiac muscle cells are stimulated to contract, producing a heartbeat.

How does ventricular fibrillation occur?
In ventricular fibrillation electrical impulses can become chaotic after the heart muscle has been damaged. Damage usually occurs after a first heart attack or due to problems from a scar in the heart muscle from a previous heart attack. Electrical activity is not passed through these scarred areas. The ventricles begin sending out their own rapid and chaotic impulses and take over the pacemaker function of the SA node.
Ventricular fibrillation may also develop if ventricular tachycardia is left untreated.
Rapid, chaotic electrical impulses cause the ventricles to quiver instead of pumping the blood out of the heart. This causes low blood pressure and no blood supply to the vital organs of the body including the brain. Most people lose consciousness within seconds and require immediate cardiopulmonary resuscitation (CPR).
The survival rate in ventricular fibrillation is better if CRP is initiated immediately until the heart can be shocked back into rhythm with the use of a defibrillator. If these measures are not taken immediately, death can occur within minutes.

What are the causes?
Ventricular fibrillation is usually caused after damage to the heart muscle from a previous heart attack.
Ventricular fibrillation can occur whenever the heart does not receive enough oxygen or due to abnormalities or diseases of the heart, or by diseases elsewhere in the body affecting the heart. Ventricular tachycardia may occur after open heart surgery, substance abuse, or medications.
Heart abnormalities and diseases include:
- Congenital heart defects (heart defect from birth)
- Coronary artery disease (decreased blood supply to the heart) or atherosclerosis

- High blood pressure
- Heart attack
- Cardiomyopathy and congestive heart failure
- Rheumatic fever and pericarditis (inflammation of the heart)
- Abnormalities of the heart valves, especially mitral stenosis and mitral regurgitation
- Hypertrophy (enlarged heart)
Diseases elsewhere in the body affecting the heart:
- Hyperthyroidism
- Pulmonary embolism (blood clot in lung)
- Chronic obstructive pulmonary disease (COPD) and emphysema
- Sleep apnea
- Electrolyte imbalances (low potassium/ magnesium levels)
Substance abuse:
- Consuming large amounts of alcohol (binge drinking)
- Cocaine, amphetamines, diet pills, caffeine, or cold medicines
- Tobacco
- Carbon monoxide intoxication
Surgery
- First week following open heart surgery
Medications
- Amiodarone
- Sotalol
Who is at risk?
Several factors may increase the risk of ventricular fibrillation.
These include:
- Men
- Increasing age
- Congenital heart disease
- Family history of ventricular tachycardia or ventricular fibrillation
- Previous episode of ventricular fibrillation
- Previous heart attack
- Hypertension
- Excessive alcohol consumption
- Electrolyte imbalance
- Heavy caffeine use
- Smoking
- Substance abuse i.e. cocaine
- Heart failure patients and COPD patients
- Pericarditis (inflammation of the heart) and autoimmune diseases
- Other chronic conditions including thyroid disorders and sleep apnea
What are the symptoms?
An episode of ventricular fibrillation presents with unconsciousness or sudden collapse. This is due to a lack of oxygen rich blood to the brain and other vital organs.
The following symptoms may occur within 1 hour before the collapse:
- Chest pain
- Dizziness
- Nausea
- Rapid heartbeat (tachycardia)
- Shortness of breath
When should I seek medical care?
If you experience symptoms of ventricular fibrillation, seek emergency care immediately.
If you faint, have difficulty in breathing, or have chest pain lasting more than a few minutes, call emergency care or call 911. These are urgent symptoms of a life-threatening ventricular fibrillation and must seek emergency care immediately to avoid the risk of cardiac arrest and death.
Steps to follow:
- Call 9-1-1
- Cardiopulmonary resuscitation must be initiated immediately from someone trained in CPR. This can be given by a family member or medical personnel. Chest compressions (30) must be started immediately followed by rescue breaths (2). This cycle is to be repeated until a shock or defibrillator can be given.
What are the complications?
The most common complication of ventricular fibrillation is sudden death. This occurs within 1 hour of symptoms.
Complications in those who survive include:
- Neurological damage
- Reduced mental perception
- Coma
How is it diagnosed?
Ventricular fibrillation is a medical emergency and is not typically diagnosed before hand unless an episode of collapse occurs within the doctor’s office.
Your physician will know if you are in ventricular fibrillation based on history, physical examination, and tests.
1. History
The patient will give history of sudden loss of consciousness or collapse.
History of warning symptoms of fibrillation may be given such as chest pain, dizziness, nausea, fast heart rate, or difficulty in breathing.
2. Physical Examination
After collapse:
- Unconscious patient
Before collapse:
- Weak or no pulse
- Low blood pressure
- Rapid breathing
- Varying heart sounds heard over chest
3. Tests
Certain tests can be performed to determine the cause and severity of your condition including:
- Blood tests- to check potassium, magnesium, and thyroid hormone levels and certain cardiac enzymes
- Electrocardiogram- this is a very useful and simple test that records the electrical activity of the heart and reveals ventricular fibrillation. Also shows signs of heart attack, conduction abnormalities, hypertrophy, and electrolyte imbalances.
-
Chest X-ray- a simple test performed in our office showing the size and shape of the heart. Also reveals the presence of fluid in the lungs.
-
Echocardiogram (ECHO) - a noninvasive test performed in our office to determine the size and function of the heart’s chambers and the structure and function of the heart valves.
How is it treated?
Ventricular fibrillation is a medical emergency and must be treated immediately.
The goal of treatment is to restore blood flow as quickly as possible to prevent damage to vital organs.
Once blood flow is restored, treatment is given to prevent future episodes.
- Cardiopulmonary Resuscitation (CPR) - this mechanically helps to restore blood flow to vital organs by mimicking the pumping action of the heart.
- Call for emergency help immediately.
- CPR can be preformed by anyone trained in CPR including a family member until medical personnel arrive.
- As per new regulations, chest compressions must be initiated first rather then giving rescue breaths. Count 30 chest compressions that must be given hard and fast over the chest. Allow the chest to rise completely between compressions.
After 30 chest compressions are given, mouth-to-mouth breathing must be given with 2 rescue breaths with the person’s neck in an extended position to allow airflow through the airways.
Repeat chest compressions and rescue breaths until help arrives.
- Defibrillation- this is a device used to deliver a quick electric shock through the chest wall. This is given to ‘shock’ the heart out of the chaotic rhythm and usually allows the heart to resume a normal heart rhythm.
A shock may be given by medical personnel or may be given through a public-use defibrillator. A public defibrillator can be located in public places including airports, schools, malls, and community centers. A public defibrillator is called an Automated External Defibrillator (AED).
Once a person collapses and CPR is initiated, an AED must be retrieved. Turn the device on by pushing the power button. In the AED kit, two pads will be provided that must be matched up accordingly to the areas over the chest to the pictures given on the pads. The device will detect if the heart is to be shocked or not. If the heart requires a shock, stand clear and press the “Shock” button. CPR must be resumed until help arrives or until the person begins to move.
It is important to take a CPR class especially in those with family members who are at risk.
- Cardioversion: if pulse is present, in a hospital setting a shock is delivered to your heart through pads attached to the chest wall. The current interrupts the electrical impulses in the heart and restores a normal rhythm.
Treatment to prevent future episodes
If structural damage has occurred in the heart after ventricular fibrillation episode, medications or a medical procedure may be recommended to reduce the risk of a future episode.
1. Medications
Various anti-arrhythmic drugs may be used in the long term treatment of ventricular fibrillation.
Different classes of medications may be used including beta blockers, angiotensin-converting enzyme (ACE) inhibitors, calcium channel blockers, and anti-arrhythmics (amiodarone).
Since there is a stagnation of blood in the heart during fibrillation, blood clots commonly develop in the heart. Medications are used to prevent the formation of blood clots.
These drugs include:
- Heparin
- Warfarin
- Dabigatran
- Aspirin
- Clopidogrel
2. Implantable cardioverter-defibrillator (ICD)
After fibrillation has been stabilized, an ICD implantation may be recommended.
This is a small implantable device used to detect abnormal heart rhythms and shock the heart out of the dangerous rhythm.
3. Catheter ablation:
is a hospital procedure done to destroy areas on the heart that are causing irregular heart rhythms.
A thin flexible tube is inserted into a blood vessel to access the heart. Heat is applied to the tip of the catheter to permanently destroy small areas of abnormal heart tissue.
The damaged tissue is no longer capable of generating electrical impulses.
What is the prognosis?
With early detection and effective CPR given before a defibrillator is given, up to 25% of victims are able to leave the hospital without neurological damage.
If ventricular fibrillation occurs within the hospital with a heart attack, defibrillation has a 95% success rate.
If shock and heart failure occur, even with defibrillation only 30% of those shocked will return to a normal heart rate.
The survival rate for a person who has an attack of ventricular fibrillation outside the hospital ranges between 2-25%.
How can I prevent this?
Ventricular fibrillation can not always be prevented, but there are ways of reducing the risk of developing ventricular fibrillation. Leading a healthy lifestyle may reduce the chance of coronary artery disease, which can cause ventricular tachycardia and fibrillation.
Some ways you can reduce the risk is by:
- No smoking!
- Drink in moderation
- Limit caffeine
- Avoid recreational drugs
- Be physically active. Engage in moderate physical activity for at least 30 minutes a day
- Eat nutritious foods low in cholesterol and fats
- Maintain a BMI below 25
- Control high blood pressure, cholesterol, and sugar
- Control stress
- Have regular physical exams and report any signs or symptoms to your physician.
- Take a CPR course if a family member or friend is at risk of ventricular fibrillation.
Updated April 4th, 2012 -JS
For further information please contact Cardiovascular Interventions, PA at 407-894-4880
Cholesterol
- Hyperlipidemia
- Advanced Lipid Profile
-
Lipoprotein (a)
-
Atherosclerosis
-
Types of Dietary Fats
Advanced Lipid Profile
What is it?
An advance lipid profile is a test used for the identification of additional risk factors of coronary artery disease that may not be detected on a standard lipid profile, despite a normal result.
Early detection of elevated lipid levels can easily be reversed with diet modification and medications. An advanced lipid profile is a successful tool in the detection and management of high cholesterol that can cause serious heart problems.
Early risk factor identification and modification can drastically reduce the morbidity and mortality of heart disease.
Why do I need it?
An advanced lipid profile is usually recommended at your initial visit to our clinic in addition to a standard lipid profile and is intermittently checked throughout treatment.
Standard cholesterol tests may not completely represent the risk for heart attack and strokes. This test is especially useful in patients with diabetes, insulin resistance, or cardiovascular disease who continue to have progression of cardiovascular disease even when their LDL (Lousy cholesterol) and total cholesterol is at their target goal.
This test helps determine the most accurate target number which is best for you, based on your risk factors.
What is it?
An advanced lipid profile measures a variety of markers for cholesterol.
The two tests commonly done are:
- Apolipoprotein B (ApoB)
- LDL particle number (LDL-P)
Other tests include:
- Apolipoprotein A1 (ApoA1)
- HDL particle number (HDL-P)
- Small dense LDL (sdLDL)
- HDL-2
- Lipoprotein (a) Mass (Lp (a))
- Lipoprotein-associated phospholipase (Lp-PLA2)
- Apolipoprotein E (Apo E) Genotype
- Omega 3 fatty acid
- Omega 6 fatty acid
An accurate advanced lipid profile is achieved after a 12 hour fast.*
Medications are to be taken as normally the morning of your test. If you are taking diabetic medications, do not take your medications the morning of your test. We will schedule an early appointment in these patients.
You may drink plenty of water during this fast; it will not affect the results of the test.
Apolipoprotein B (ApoB) and LDL particle number (LDL-P)

Since cholesterol can not dissolve in blood, it needs to attach to a lipoprotein to transport cholesterol in blood. HDL particles are known as Apo A and the particles causing atherosclerosis (fatty plaque build up in arteries) are known as Apo B. The majority of circulating Apo B particles is LDL-P.
If there is an increased amount of triglycerides and cholesterol in the blood, an increased amount of Apo B will be driven into the walls of the arteries. A type of white blood cell known as a macrophage comes to the area of Apo B deposition and ingests this foreign material. This process is the hallmark of atherosclerosis.
LDL-P is the most significant predictor of cardiovascular morbidity and mortality. This gives the most accurate number of Apo B particles in circulation; therefore assess the true risk factor for atherosclerosis.
A standard lipid profile shows the overall outlook of cholesterol, it may even be normal, but an advanced lipid profile measures the most accurate LDL levels that can lead to atherosclerosis.
This shows the accurate target number that is required for the management of your cholesterol.
Your physician may advise a combination of diet and drug therapy to prevent the onset and progression of cardiovascular disease.
Apolipoprotein A1 (ApoA1)
HDL particles (Healthy cholesterol) are known as Apolipoprotein A1. Apo A1 helps to clear out cholesterol from your arteries. 
High levels of Apo A is associated with decreased risk of cardiovascular disease.
HDL particle number (HDL-P)
An increased amount of HDL-P decreases the risk of cardiovascular disease.
To further increase HDL, diet modification, exercise, and medications can be tried.
Small dense LDL (sdLDL)
Small dense LDL can produce more damage to the arterial wall than just regular LDL.
An LDL level in a standard lipid panel may be normal, but the sdLDL in an advanced lipid will assess your risk.
To lower your risk, diet, and exercise can help reduce sdLDL.
HDL-2
HDL-2 is the best type of HDL which removes cholesterol build up in the arteries.
An increased amount of HDL-2 decreases the risk of cardiovascular disease.
Lipoprotein (a) Mass (Lp (a))
Lipoprotein A is a genetic marker for cardiovascular disease.
An increased level of Lp (a) can increase the risk of cardiovascular risk at an early age.
Lipoprotein-associated phospholipase (Lp-PLA2)
Lp-PLA2 is a specific marker for vascular inflammation which can make fatty plaque build up within the artery prone to rupture.
Elevated Lp-PLA2 is a powerful predictor of heart attack and stroke.
Apolipoprotein E (Apo E) Genotype
Apolipoprotein E is a genetic marker for those who are more prone to high cholesterol levels.
Apo E comes in different forms including Apo E2, Apo E3, and Apo E4.
Apo E2 and Apo E3 patients respond well to statin drugs.
Fish oil is known to benefit Apo E2 and Apo E3 patients.
Apo E4 patients typically do not respond well to statin drugs, but respond well with diet modification.
Elevated Apo E levels increase the risk of cardiovascular disease, especially in Apo E4 patients.
This is also useful in the prediction of a patient’s responsiveness to cardiac-related environmental factors, diet, and drug therapy.
Omega 3 fatty acid
Omega 3 fatty acids reduce the risk of cardiovascular disease.
A good source of omega 3 fatty acids come from oily fish and fish oils.
Omega 6 fatty acid
Omega 6 fatty acid reduces the risk of cardiovascular disease.
Oily fish and fish oil are a good source of omega 6 fatty acid.
Results:
The results of your test will return within 7 days.
Your test results will be discussed with you during your next scheduled visit. Diet or drug therapy may be advised or altered.
Your test results will be reflected in a chart form similar to the one below.
Your test result may fall under a optimal range, intermediate risk range, or high risk range.
|
Optimal Range |
Intermediate Risk Range |
High Risk Range |
Apo B (mg/dL) |
<60 |
60 - 79 |
≥ 80 |
LDL-P (nmol/L) |
< 1000 |
1000 - 1299 |
≥ 1300 |
Apo A1 (mg/dL) |
≥ 151 |
130 - 150 |
< 130 |
HDL-P (µmol/L) |
≥ 35 |
28 - 34 |
<28 |
sdLDL (mg/dL) |
< 20 |
21 - 30 |
≥ 31 |
HDL-2 (mg/dL) |
≥ 17 |
13 - 16 |
≤ 12 |
Lp (a) Mass (mg/dL) |
< 30 |
|
≥ 30 |
Lp-PLA2 (ng/mL) |
< 200 |
200 - 234 |
≥ 235 |
Apo E Genotype |
2/2 – 2/3 |
2/3 |
3/4 – 4/4 |
Omega- 3 Total |
0.1% - 14.1% |
Omega- 6 Total |
28.6% - 44.5% |
Updated March 6th, 2012 -JS
For further information please contact Cardiovascular Interventions, PA at 407-894-4880
Lipoprotein (a)
What is it?
Lipoprotein (a), also called Lp (a), is a modified form of LDL cholesterol bound to the protein apolipoprotein.
Elevated levels of lipoprotein (a) may promote atherosclerosis which is a risk factor for coronary artery disease.
There is a fundamental relationship of lipoprotein (a) excess with the risk of heart attack.

How am I at risk of a heart attack?
Lp (a) is a modified LDL (lousy cholesterol) in which a large protein, apolipoprotein (a) is bound to apolipoprotein B. Apolipoprotein (a) is a specific marker of Lp (a).The apolipoprotein (a) chain has five domains. One of the domains is similar to a fibrin-binding domain of plasminogen that dissolves clots. Because of the similar structure with plasminogen, Lp (a) interferes with the protein that dissolves clots.
Lp (a) also binds to a type of white blood cell that promotes the formation of fatty cells in the arterial walls called foam cells. Lp (a) deposits cholesterol into atherosclerotic plaques that narrow blood vessels in the heart. Inflammation begins within these plaques and leads to a thickening of the arterial wall. This further narrows the blood vessels depriving the heart muscle and body tissue of oxygen rich blood.
Therefore, plaque formation, inflammation and altered plasminogen activity increase the risk of heart attack.
Both genetic and environmental factors including age, sex, race, and ethnic background influence the risk of coronary artery disease.
What diseases can occur with high Lp (a) levels?
High Lp (a) levels in blood increases the risk for:
- Coronary artery disease (CAD)
- Cerebrovascular disease (CVD)
- Atherosclerosis
- Thrombosis
- Stroke
Who should be screened?
Patients with a moderate or high risk of cardiovascular disease are recommended to have their Lp (a) levels checked.
Any patient with the following risk factors should be screened.
- Premature cardiovascular disease
- Familial hypercholesterolemia
- Family history of premature cardiovascular disease
- Family history of elevated lipoprotein (a)
- Recurrent cardiovascular disease despite cholesterol lowering treatment with a statin drug
- ≥10% 10-year risk of fatal and/or non-fatal cardiovascular disease
There are no signs or symptoms of elevated lipoprotein (a), although Lp (a) levels are assessed by performing a simple blood test for an advanced lipid profile.
Even those who are not at risk are recommended to have an advanced lipid profile done at least once to assess accurate lipid levels.
How is it treated?
High Lp (a) levels are hard to treat, yet some medications are available to lower these levels.
The medications available to lower elevated Lp (a) levels are: 
- Niacin (vitamin B3)
- Fibrate drugs (gemfibrozil/ fenofibrate)
Niacin 1-3 grams daily can reduce lipoprotein (a) levels by 20-30%.
Atorvastatin, aspirin, coenzyme Q10, L-carnitine, gingko biloba may be beneficial as well.
When elevated Lp (a) levels are discovered in a patient, attention towards other more easily treatable risk factors should be increased. These include high LDL cholesterol levels, smoking, and leading a sedentary lifestyle.

Lipoprotein (a) levels:
- Desirable < 14 mg/dL
- Borderline risk: 14-30 mg/dL
- High risk: 31-50 mg/dL
- Very high risk: >50 mg/dL
Updated April 18, 2012 -JS
For further information please contact Cardiovascular Interventions, PA at 407-894-4880
Atherosclerosis
What is it?
Atherosclerosis is the build up of fat and cholesterol filled plaques inside the artery walls, which can restrict blood flow.
These plaques have the potential to rupture leading to the development of a clot. A clot may restrict blood flow further or dislodge and obstruct another vessel.
Atherosclerosis increases the risk of cardiovascular disease including heart attack and stroke.
Atherosclerosis can affect any artery in the body and is a preventable and treatable condition.
How does it occur?
At a young age, fat can start to deposit within the blood vessel walls. Fat build up over the years can cause injury to the blood vessel walls.
Not only fat, but a number of other substances in blood including inflammatory cells, cellular waste products, proteins, and calcium begins sticking to the inside of vessel wall. All of these substances collectively form what is called Plaque.



Many of the plaque deposits have a soft inner center and hard exterior. If the hard outer surface breaks or tears, the soft inner center will become exposed to our body’s platelets.
The platelets will come to the area of breakage and form a clot around the plaque. This further narrows the artery and restricts blood flow.
A clot may dislodge and permit blood flow again, but the dislodging of a clot can also be dangerous.
What are the symptoms?
Atherosclerosis can affect any artery in the body. When this condition affects the vessels of the heart it is known as coronary artery disease. Atherosclerosis affecting all other arteries excluding the coronary arteries, it is known as peripheral vascular disease.
Peripheral vascular disease can affect the arteries of the neck, arms, kidneys, legs, etc. Symptoms depend on the part of the body that is affected although; more than one area can be affected.
Coronary artery disease: Symptoms may include chest pain (angina), shortness of breath, dizziness, palpitations, sweating, extreme weakness, fast heart beat, or nausea.
Peripheral vascular disease:
- Peripheral Vascular Disease: includes symptoms of claudication (pain in the legs while walking), cold feet, tingling or numbness in the legs, slow healing of cuts or sores over the feet and legs, pale, shiny, or bluish skin, brittle and slow growing nails, loss of hair over the limbs, absent or weak pulse in the legs or feet, or erectile dysfunction
- Carotid Artery Disease (narrowing of artery in the neck): symptoms of syncope or fainting, dizziness, or blurring of vision.
- Renal Artery Disease (narrowing of arteries in kidney): symptoms of high blood pressure.
What are the causes of atherosclerosis?
-
Smoking
-
High blood pressure
-
High cholesterol
-
Diabetes
What are the risk factors?
Non-modifiable risk factors:
-
Age: Old age
-
Sex: Men have a greater risk of atherosclerosis than women. The risk increases in woman after menopause due to less hormone production of estrogen
-
Race: African Americans have more severe high blood pressure than Caucasians
-
Family history of medical diseases associated with cardiovascular disease
Modifiable risk factors:

-
Smoking
-
Excessive alcohol consumption
-
High blood pressure
-
High blood cholesterol levels
-
Uncontrolled diabetes (HbA1c >7.0)
-
Sedentary lifestyle- lack of physical activity is associated with atherosclerosis
-
Obesity
-
Obstructive sleep apnea
-
Chronic inflammation- even having the flu can be a risk because it is a form of inflammation. C-reactive protein is a marker of inflammation and high levels of this can show an increased risk of atherosclerosis.
-
Stress
-
Homocysteine – is an amino acid in the body that helps make protein and maintain tissue. Elevated levels of this can increase the risk of atherosclerosis.
-
Fibrinogen- is a protein in the body that aids in blood clotting. An increase in this can increase platelet clumping leading to the formation of clots.
What are the complications?
Complications of atherosclerosis depend on the location of the narrowed or blocked artery.
Coronary artery disease: Complications of coronary artery disease include chest pain (angina), heart attack, congestive heart failure, arrhythmia (abnormal heart rhythm), or pericarditis (inflammation of a layer of the heart).
Peripheral vascular disease:
- Peripheral Vascular Disease: complications include permanent tingling, numbness, or weakness in the legs or feet, permanent burning or aching pain, leg or foot ulcers, or gangrene (death of a part of the body due to lack of blood supply. Treatment requires amputation of the affected part)
- Carotid Artery Disease: transient ischemic attack (TIA) or stroke
- Renal Artery Disease (narrowing of arteries in kidney): kidney failure
How is it diagnosed?
Dr. Jamnadas will have a better idea if you have atherosclerosis by:
- Discussing with you about your symptoms, medical and family history, and risk factors.
- Physical examination- blood pressure will be recorded, peripheral pulses will be felt, and the physician may listen to your arteries for an abnormal whooshing sound called a bruit that may indicate poor blood flow.
- Blood tests- to assess increased levels in cholesterol or elevated blood sugar levels.
- Performing diagnostic tests including:
Electrocardiogram (EKG) – records the electrical activity of the heart. This may show evidence of a current or previous heart attack.
Ankle Brachial Index (ABI) – this is a non-invasive test measuring the ratio of blood pressure in the ankle to your arm. If the value is lower than expected, this may indicate a vascular problem. You may be required to walk on a treadmill and have readings taken before and after exercise.
Ultrasound Doppler Test- helps to evaluate the blood flow through a vessel and identify the site of blocked or narrowed arteries.
Echocardiogram (ECHO) - shows the images of the heart which determines whether the heart walls and pumping activity are normal or performing weakly.
Stress test- helps access the blood flow to the heart at rest and during stress. Will detect if any areas receiving less blood flow.
Chest x-ray- reveals signs of congestive heart failure.
Angiogram- used to locate the exact anatomical site of blockage by injecting a dye within the arteries, which is visualized on x-ray showing the amount of blood flow to an area, and the number, size, and location of any blockages.
Computerized tomography (CT) - helps visualize your arteries. Sometimes an electron beam computerized tomography (EBCT) is offered to detect calcium within fatty deposits in narrowed arteries.
How is it treated?
The treatment for atherosclerosis may include lifestyle changes, medications, or procedures.
It is very important to reduce any risk factors in order to manage the symptoms and progression of atherosclerosis.
Lifestyle changes
-
Quit smoking!!
-
Limit alcohol intake if you chose to drink.

-
Lower blood sugar levels
-
Maintain a normal blood pressure and cholesterol
-
Eat a well balanced diet.
-
Eat foods high in fiber such as whole grain cereals, oatmeal, and figs. Eat plenty of fruits such as apples, bananas, prunes, oranges, and pears. Include fish and legumes such as beans or chickpeas.
-
Lower your salt intake.
-
Avoid fatty foods including bad saturated fat that is found in some meats, dairy products, chocolates, baked goods, and deep-fried food.
-
Exercise regularly for 30 minutes at least 3-4 times a week and be more physically active.

-
Maintain a healthy weight with a BMI below 25.
-
If you have diabetes, practice proper foot care and prevent injury to the foot. Avoid walking barefoot and wear proper shoes. Maintain toenails and skin care. Be sure to inspect the soles of your feet regularly.
-
Relax and reduce stress.
For some people these changes may be the only treatment needed.
Medications
If lifestyle changes are not enough to control the effects of atherosclerosis, medications may be prescribed to you including:
- Cholesterol lowering medications- these act by decreasing the amount of cholesterol in blood, especially Lowering your low density lipoprotein (LDL- your Lousy cholesterol) and keep your high density lipoprotein (HDL- your Healthy cholesterol) High.
- Aspirin – is an anticoagulant that prevents platelets from clumping together. This reduces the chances of a blood clot narrowing or obstructing the arteries. This is not appropriate for all patients including patients with bleeding disorders or already taking an anticoagulant.
- Beta blockers- slow your heart rate and reduce blood pressure which decreases the oxygen demand of the heart.
- Angiotensin converting enzyme (ACE) inhibitors and Angiotensin receptor blockers (ARBs) - help relax the blood vessels and lower the blood pressure so the heart does not have to work as hard and improve the lining of arteries.
- Calcium Channel Blockers- medication that helps relax the muscles surrounding the coronary arteries and cause the vessels to open leading to an increase in blood flow to the heart and lowering blood pressure.
Supplements
Food and supplements rich in omega 3 fatty acids help reduce inflammation throughout the body, lower blood pressure, and the risk of heart attack.
- Fish and Fish Oil- are high in omega 3 fatty acids, especially in fish like salmon, herring, and tuna. Fish oil supplements are also high in omega 3 fatty acids.
- Flax and flaxseed oil- contain omega 3 fatty acids and fiber.
- Greensoul- is a natural supplement that helps to maintain a healthy cholesterol level, supports the body’s immunity, and the ability to fight free radicals.
- Coenzyme Q10- helps maintain a healthy cholesterol level, boosts immunity, and energy.
- Perfusion SR- helps enhance blood flow, maintain a normal blood pressure, and enhance the elasticity of large arteries.
Procedures to restore blood flow
Angioplasty and stent placement: this is a procedure considered as non-surgical because it is done by Dr. Jamnadas or Dr. Kelly, who accesses the artery by inserting a long, thin tube (catheter) into the narrowed part of your artery while being visualized under x-ray.
A wire with a deflated balloon is passed through the catheter to the narrow area. The balloon is then inflated, compressing the plaque against the artery wall and widening it, so it no longer restricts blood flow.

Blocked arteries in the coronary, renal, iliac, and legs can easily be treated with stenting or endarterectomy.
Unlike stenting done in the coronary arteries, only bare metal stents are used in the peripheries. Bare metal stents act as a scaffolding to keep an artery open but suffer a 30% re-stenosis rate.
This is due to a process called intimal hyperplasia which is attributed to a keloid. A keloid is an overgrowth of the inner lining of the blood vessel that covers over the stent.
However, this reparative process lasts no more than 6 minutes. After 6 minutes, if an artery has not re-narrowed at the site of the stent, it is unlikely to re-narrow.
If symptoms were to reoccur, it would be due to New blockages at another site within the arteries.
**Patients will need to improve risk factor modifications to prevent New blockage formation.

- Endarterectomy: is a procedure where a catheter is used to scrape the inner lining of an artery. That material which is blocking the artery is extracted and then taken out of the artery using the catheter. The procedure is Best suited for blocked arteries in the legs. Laser arterectomy has been used in the legs also, but Dr. Jamnadas does not favor it at this time due to the high re-stenosis rate.
Bypass Surgery: is performed by removing a portion of a small blood vessel and sewing or ‘grafting’ one end of the bypass proximal to the area of blockage and the other end beyond the area of blockage, therefore bypassing the affected area.
-
Enhanced external counterpulsation (EECP): This is for patients who have angina (chest pain) and have already exhausted the standard
treatments without successful results or for those who do not qualify for the other treatment regimens. This is a non-invasive procedure which promotes the formation of collaterals to bypass the clogged arteries as another means of getting oxygen rich blood to those areas of the heart that are not getting it. This is carried out in our office daily for 7 weeks.
How can I prevent this?
-
Do not smoke!
-
Maintain a healthy weight with a BMI <25
-
Maintain a normal blood pressure, cholesterol and sugar levels.
-
Eat nutritious, low fat foods and avoid foods high in cholesterol.
-
Exercise regularly for 30 minutes at least 3-4 times a week.
Updated February 27th, 2012 -JS
For further information please contact Cardiovascular Interventions, PA at 407-894-4880
Reducing Your Risk of Coronary Heart Disease
It’s the Type of Fat – Not the Amount of Fat
Although many people believe that reducing their intake of dietary fat will lower their risk of coronary heart disease (CHD), recent studies show that the type of fat—not the amount of fat—consumed influences cardiovascular risk.(1) Despite a decrease in total fat consumption in the United States, there has not been a decline in total caloric intake; additionally, the prevalence of type 2 diabetes and obesity has increased, suggesting that there is another dietary factor influencing cardiovascular risk.1 This factor is the type of fat consumed; different types of fats have different effects on health.
Challenging the Low-Fat Diet
Results of a prospective cohort study assessing dietary choices of 80,082 women with no cardiovascular disease at baseline showed that each 5% energy intake from saturated fat was associated with a 17% increase in the risk of coronary disease.2 The study concluded that replacing saturated and trans fats with unhydrogenated, monounsaturated, and polyunsaturated fats is more effective at preventing CHD than is following a low-fat diet.(2) This is because low-fat, high-carbohydrate diets raise HDL cholesterol levels and raise fasting triglyceride levels, which increases cardiovascular risk.2
Why is Saturated Fat Bad?
Saturated fatty acids increase total cholesterol and LDL cholesterol levels; elevated lipid levels are a risk factor for cardiovascular disease.1 In the Nurses’ Health Study, there was an increase in cardiovascular disease risk with each intake of saturated fatty acids.1 Both saturated and trans fat intake are associated with higher LDL levels and lower HDL levels, which increase cardiovascular disease risk. Additionally, trans fat consumption increases lipoprotein(a) levels, which are also associated with cardiovascular disease.
What is Monounsaturated Fat?
Both monounsaturated and polyunsaturated fats are liquid at room temperature. Polyunsaturated fats help your body to rid itself of newly synthesized cholesterol, keeping blood cholesterol levels low. Both fats keep your blood cholesterol levels low when substituted in place of saturated fats.
Studies have shown an inverse relationship between consumption of monounsaturated fats and total mortality.1 In fact, mortality rate of cardiovascular heart disease is very low in traditional Mediterranean populations that consume olive oil (a monounsaturated fatty acid source) as their primary source of fat.1 In metabolic studies, substituting carbohydrates with monounsaturated fats raises HDL levels without affecting the LDL levels. This may also improve glucose tolerance and insulin sensitivity among diabetic patients.
Now What?
In your diet, you should replace saturated and trans fats with monounsaturated and polyunsaturated fats. This will lower your lipid profile as well as your risk of CHD. Also, you should exercise at least 20 minutes a day, most days of the week, to ensure optimal cardiovascular health. In fact, a combination of diet and lifestyle changes is associated with decreased progression of atherosclerosis. The Lifestyle Heart Study assigned 28 MI patients to an intervention consisting of a low-fat, vegetarian diet, exercise, and yoga.1 The other 20 individuals were assigned to usual care group.1 After one year, the blood cholesterol was lowered by 19% in the intervention group, compared with the control group.1 This shows the benefits of both a healthy diet and exercise.
Dietary Sources of Trans Fats: Stick margarine, commercially baked products, deep-fried foods, partially hydrogenated vegetable oils (cookies, crackers, French fries, donuts)
Dietary Sources of Saturated Fats: some plant oils (palm, palm kernel, and coconut oils), foods from animals (whole milk, ice cream, lard, meats).
Dietary Sources of Polyunsaturated Fats: nuts (walnuts, almonds, peanuts, and other nuts), certain plant oils (safflower, sesame, soy, and others)
Dietary Sources of Monounsaturated Fats: certain plant oils (olive, canola, peanut), avocados
- Please refer to our Heart Healthy recipes under the Educational Resource Center for healthy recipes for all occasions.
Updated February 12, 2009 - MJ
Hyperlipidemia
What is it?
Hyperlipidemia refers to elevated levels of lipids (fats) in blood. These lipids include cholesterol and triglycerides. Elevated levels of lipids can significantly increase your risk of heart disease.
Elevated lipids in blood deposit into the walls of blood vessels and form plaque. Constant elevation of these levels can increase the deposits laid in the vessel walls. These plaques restrict the blood flowing through the vessels depriving tissues of the oxygen and nutrients it needs. This plaque has a potential of rupturing. Once the plaque ruptures, a clot will form and further narrow the blood flow to tissues.
This narrowing of the arteries is called atherosclerosis. When atherosclerosis affects the heart it is called coronary artery disease. This causes symptoms of chest pain and can lead to a heart attack. Atherosclerosis of the peripheries is known as peripheral vascular disease which causes symptoms of leg pain while walking called claudication. Atherosclerosis can even narrow the vessels supplying the brain causing dizziness. A clot may dislodge and block a vessel in the brain causing stroke.
Hyperlipidemia can significantly increase a person’s risk of heart attack and stroke!


What causes this?
-
Genetic- hyperlipidemia runs in the family due to a genetic mutation.
- High saturated fatty diet
- Obesity
- Sedentary lifestyle- lack of physical activity
- Endocrine disease- Diabetes, hypothyroidism, and adrenal disease
- Kidney disease
- Liver disease
What are the different types of lipids?
Lipids are made up of cholesterol and triglycerides. Lipids are transported in blood as parts of larger complexes known as lipoproteins.
Blood tests measure the level of your lipoproteins. This blood test is performed here in our clinic with same day results. The measurement of these lipids is known as your lipid profile.
The standard lipid profile measures:
- Total Cholesterol
- Low Density Lipoprotein (LDL)
- High Density Lipoprotein (HDL)
- Triglycerides
-
Total Cholesterol- this measures all the overall cholesterol components in blood.
This level includes your good and bad cholesterol; therefore this is not an indicator to start medications.
Total cholesterol levels can be checked at any time of the day and does not require fasting.
Normal levels: Below 200 mg/dl
- Low Density Lipoprotein (LDL) - this is sometimes referred to as your Lousy cholesterol. LDL cholesterol builds up in the arteries making them narrow. Your LDL is the most accurate component for checking the risk of heart disease.
An accurate LDL level is achieved after a 12 hour fast.
Normal levels: Below 70mg/dl in high risk patients; below 100mg/dl with risks; below 120mg/dl in non-risk patients
- High Density Lipoprotein (HDL) - this is sometimes referred to as your Healthy cholesterol. HDL picks up excess cholesterol and takes it back to the liver instead of it accumulating in the arteries. – That’s a good thing!
Like total cholesterol, HDL can be checked at any time of the day without fasting.
Normal levels: Above 60mg/dl
- Triglycerides- this is found in a type of lipoprotein called very low density lipoprotein. Triglycerides accumulate in the arteries and can be atherogenic.
Triglycerides should be checked with a 12 hour fast for accurate levels.
Normal levels: Below 150mg/dl
Who is at risk?
Positive Risk Factors Negative Risk Factors
Family history of heart disease High HDL levels
Increasing age
Low HDL levels
Smoking
High Blood Pressure (Hypertension)
Obesity
Poor Diet
Diabetes
Lack of exercise
When should I get my lipid profile done?
Screening should be started at the age of 35 in patients with no risk factors
With risk factors, screening should be started at the age of 20-25.
You must have your lipid profile checked at least once by the age of 45.
Depending on your risk factors and lipid levels, you may be screened every 5 years or more frequently, if risk factors are present.
How is it treated?
Lifestyle changes:
The first line of treatment is lifestyle changes. Lifestyle changes not only treat, but prevent hyperlipidemia.
A few ways you can modify your lifestyle are by:
Quit smoking if you are a smoker.
Reduce the amount of saturated fats and cholesterol you take. Saturated fats include cheese, butter, milk, and red meats.
Add more fruits and vegetables to your diet.
Try eating fish like cod, salmon, or tuna. These are low in fat and have healthy omega 3 fatty acids.

Increase fiber in your diet. This helps in lowering cholesterol levels. Eat more whole grain foods like brown rice and oatmeal.
Reduce your weight. Maintain a BMI below 25.
Exercise for 30 minutes at least 3-4 times a week and be more physically active.
Medications:

Medications are to be used along with lifestyle changes.
Dr. Jamnadas or Dr. Kelly will chose a medication or combination of medications depending on age, risk factors, your present health, and possible side effects of the drug.
After initial therapy, your lipid profile will be monitored at 6 week intervals until target levels are reached. Monitoring will then be done every 6-12 months.
Lipid lowering medications include:
-
Statins- statins are extremely useful in lowering your lousy LDL by 25- 60%. They are also used in the prevention of heart disease. Statins act by blocking a step in cholesterol synthesis in the liver and enhances the clearance of LDL from circulation.
Statins include: Simvastatin (Zocor), Atorvastatin (Lipitor), Lovastatin (Altoprev, Mevacor), Pravastatin (Pravachol), Rosuvastatin (Crestor), and Fluvastatin (Lescol).
- Bile acid sequestrants- these agents block the absorption of bile acids from the gut that the liver uses to make cholesterol. These help in lowering only mild to moderately elevated LDL and slightly increase HDL.
These may interfere with fat soluble vitamin absorption including vitamins A, D, K, and E. The effects of this can be minimized by taking the drug at different times of the day.
Bile acid sequestrants include: Cholestyramine (Prevalite, Questran), Colestipol (Colestid), and Colesevelam (WelChol).
- Ezetimibe (Zetia) - this blocks the absorption of cholesterol from the food in your diet.
- Ezetimibe-Simvastatin (Vytorin) - this is a combination of a statin and ezetimibe. This lowers the absorption of cholesterol from your diet and lowers the synthesis of cholesterol from the liver.
- Niacin (Niaspan) - is a vitamin that decreases triglyceride levels by limiting the synthesis of LDL and VLDL.
- Fibrates- these lower the synthesis of LDL moderately and increase HDL by 10-15%.
Fibrates include: Gemfibrozil (Lopid) and Fenofibrate (Tricor, Triglide).
Supplements:
Food and supplements rich in omega 3 fatty acids help reduce triglyceride levels and lower the risk of heart disease.
-
Fish and Fish Oil: are high in omega 3 fatty acids, especially in fish like salmon, herring, and tuna. Fish oil supplements are also high in omega 3 fatty acids.
- Flax and flaxseed oil: contain omega 3 fatty acids and fiber.
- Greensoul: is a natural supplement that helps to maintain a healthy cholesterol level, supports the body’s immunity, and the ability to fight free radicals.
- Coenzyme Q10: helps maintain a healthy cholesterol level, boosts immunity, and energy.
What are the complications?
High lipid levels can lead to atherosclerosis. Atherosclerosis can affect any artery in the body and may lead to complications including:
-
Coronary artery disease: can lead to chest pain due to narrowing of the arteries in the heart
- Heart attack: the complete obstruction of an artery in the heart due to rupture of plaque and clot formation.
- Peripheral vascular disease: narrowing of the arteries in the legs can lead to leg pain while walking called claudication.
- Stroke: the complete obstruction of an artery in the brain due to a dislodged clot from a ruptured plaque.
How can this be prevented?
Healthy lifestyle changes can lower your chances of having hyperlipidemia. Set goals for yourself and stick to it. Some ways you can help prevent having high lipid levels are:
- Quit Smoking

- Limit alcohol to a maximum of one glass a day if you chose to drink.
- Eat a low-fat diet with plenty of whole grain foods, fruits, and vegetables
- Lose excess weight! Maintain a BMI below 25.
- Exercise for 30 minutes at least 3-4 times a week and be more physically active.
Updated February 7, 2012- JS
For further information please contact Cardiovascular Interventions, PA at 407-894-4880
Coronary Artery Disease
What is it?
Coronary artery disease is the narrowing or blockage of the coronary arteries caused by atherosclerosis.
Atherosclerosis occurs due to cholesterol and plaque build up in the walls of the arteries. Plaque build up can restrict blood flow to the heart muscle and deprive the heart of the oxygen and nutrients that it needs.
How does it occur?
At a young age, fat can start to deposit within the blood vessel walls. Fat builds up over the years that can cause injury to the blood vessel walls.
Not only fat, but a number of other substances in blood like inflammatory cells, cellular waste products, proteins, and calcium begin sticking to the inside of vessel wall. All of these substances collectively form what is called Plaque.



Many of the plaque deposits have a soft inner center and hard exterior. If the hard outer surface breaks or tears, the soft inner center will become exposed to our body’s platelets.
The platelets will come to the area of breakage and form a clot around the plaque. This causes the artery to narrow even more.
A clot may dislodge and permit blood flow again, but the dislodging of a clot can also be dangerous.
What are the symptoms?
Some people may have no signs or symptoms at all. This is known as Silent Coronary Artery Disease, which is seen more often in diabetic patients. Silent coronary artery disease can lead to sudden heart attack, congestive heart failure, or arrhythmia.
When there are symptoms, the following may be noticed:
-
Chest pain (Angina) - Angina is the most common symptom of coronary artery disease. It can be described as chest pain, discomfort, heaviness, tightness, pressure, aching, burning, numbness, fullness, or squeezing. This can be mild or severe. Angina is usually felt in the chest, but can also be felt in the left shoulder, arms, neck, back, and jaw. This can be similar to a heart attack, though angina pain usually lasts for only a few minutes and relieved on rest. If not relieved after a few minutes or on rest, call 9-1-1 for help.
- Shortness of breath
- Palpitations
- Faster heartbeat
- Dizziness
- Nausea
- Extreme weakness
- Sweating
If you are not in an emergency situation, call Dr. Jamnadas and inform him of any symptoms you are experiencing.
If you have been prescribed Nitroglycerin and experience angina: Stop what you are doing and rest. Take one nitroglycerin tablet sublingually and wait 5 minutes. If you still have angina after 5 minutes, call 9-1-1 for emergency assistance.
For patients diagnosed with Chronic Stable Angina:
- If you experience angina, take 1 nitroglycerin tablet sublingually.
- Wait 5 minutes
- If you are still experiencing angina, take another tablet.
- Wait 5 minutes
- If angina still persists, take a final sublingual tablet.
- Wait 5 minutes
- After a total of 3 nitroglycerin tablets and a total of 15 minutes of angina, call 9-1-1 for emergency help.
What causes coronary artery disease?
- Smoking
- High blood pressure
- High cholesterol
- Diabetes
What are the risk factors?
Non-modifiable risk factors:
-
Age: Old age
- Sex: Men have a greater risk of coronary artery disease than women. The risk increases in woman after menopause due to less hormone production of estrogen.
- Race: African Americans have more severe high blood pressure than Caucasians therefore have a higher risk of heart disease.
- Family history of heart disease.
Modifiable risk factors:

-
Smoking
- Excessive alcohol consumption
- High blood pressure
- High blood cholesterol levels
- Uncontrolled diabetes (HbA1c >7.0)
- Sedentary lifestyle- lack of physical activity is associated with coronary artery disease.
- Obesity
- Obstructive sleep apnea
- Chronic inflammation- even having the flu can be a risk because it is a form of inflammation. C-reactive protein is a marker of inflammation and high levels of this can show an increased risk of coronary artery disease.
- Stress
- Depression- loneliness can be a risk factor of coronary artery disease.
- Homocysteine – is an amino acid in the body that helps make protein and maintain tissue. Elevated levels of this can increase the risk of coronary artery disease.
- Fibrinogen- is a protein in the body that aids in blood clotting. An increase in this can increase platelet clumping leading to the formation of clots.
What are the complications of coronary artery disease?
-
Chest pain (Angina) - this is due to the narrow arteries limiting oxygen enriched blood from supplying the heart muscle. The demand is greater during physical activity and exercise leading to chest pain and shortness of breath.
-
Heart attack- this is due to the rupture of plaque and formation of a blood clot completely obstructing an artery.
-
Congestive heart failure- when the heart muscle is chronically deprived from oxygen and the blood flow it needs, the heart muscle can become too weak to pump adequately.
-
Arrhythmia- abnormal heart rhythm that can cause inadequate blood pressure and black outs or sudden death.
-
Pericarditis- is inflammation of a layer of the heart
-
Stroke- occurs when a dislodged clot gets taken to the brain and blocks part of an artery in the brain.
-
Peripheral artery disease- can occur due to the weak blood supply to the peripheries or a dislodged clot blocking off the blood supply.
-
Aneurysm- is a weakening in the wall of an artery causing a dilatation or sac within the artery.
-
Mitral regurgitation
-
Shock
What is the prevalence?
Heart disease is the leading cause of death in the United States.
The American Heart Association (AHA) estimates about every 34 seconds an American will have a heart attack.
How is coronary artery disease diagnosed?

Dr. Jamnadas will have a better idea if you have coronary artery disease by:
- Discussing with you about your symptoms, medical and family history, and risk factors.
- Physical examination
- Blood tests- to measure your lipid profile
Performing diagnostic tests including:
- Electrocardiogram (EKG) - records the electrical activity of the heart. This may involve you to wear a 24 hour holter monitor which records the electrical events of the heart during your normal daily activities. It is important to accurately record your activities and symptoms so Dr. Jamnadas can compare them to the holter monitor findings.
- Echocardiogram (ECHO) - shows the images of the heart which determines whether the heart walls and pumping activity are normal or performing weakly.
- Stress test- helps access the blood flow to the heart at rest and during stress. Will detect if any areas receiving less blood flow.
- Chest x-ray- reveals signs of congestive heart failure.
- Coronary Angiogram- allows Dr. Jamnadas to locate the exact anatomical site of blockage by injecting a dye within the coronary arteries showing the amount of blood flow to an area, and the number, size, and location of any blockages.
- Computerized tomography (CT) - helps visualize your arteries. Sometimes an electron beam computerized tomography (EBCT) is offered to detect calcium within fatty deposits in narrowed arteries.
- Magnetic resonance angiogram (MRA) – this is an MRI used with a dye to check for areas of narrowing or blockage in the arteries.
How is coronary artery disease treated?
The goal of treatment is for you to get back to a full and active lifestyle.
Treatment depends on many factors including your age, heart function, and other health problems.
Lifestyle changes
-
First treatment is diet changes, exercise, and weight management.
- Eat foods high in fiber such as whole grain cereals, oatmeal, and figs. Eat plenty of fruits such as apples, bananas, prunes, oranges, and pears. Include fish and legumes such as beans or chickpeas.
- Lower your salt intake.
- Avoid fatty foods including bad saturated fat that is found in some meats, dairy products, chocolates, baked goods, and deep-fried food.
- Maintain a healthy weight with a BMI below 25.

- No smoking.
- Limit alcohol to one glass a day if you chose to drink.
- Be more physically active.
- Relax and reduce stress.
For some people these changes may be the only treatment needed.
Medications

If lifestyle changes are not enough to control your heart disease, medications may be prescribed to you including:
- Cholesterol lowering medications- these act by decreasing the amount of cholesterol in blood, especially Lowering your low density lipoprotein (LDL- your Lousy cholesterol) and keep your high density lipoprotein (HDL- your Healthy cholesterol) High.
- Aspirin – prevents platelets clumping together. This thins the blood and reduces the chances of a blood clot narrowing or obstructing the arteries. This is not appropriate for all patients including patients with bleeding disorders or already taking a blood thinner.
- Beta blockers- slow your heart rate and reduce blood pressure which decreases the oxygen demand of the heart.
- Angiotensin converting enzyme (ACE) inhibitors and Angiotensin receptor blockers (ARBs) - help relax the blood vessels and lower the blood pressure so the heart does not have to work as hard and improve the lining of arteries.
- Calcium Channel Blockers- medication that helps relax the muscles surrounding the coronary arteries and cause the vessels to open leading to an increase in blood flow to the heart and lowering blood pressure.
- Nitroglycerin- either a sublingual tablet, spray, or patch which is used to control chest pain.
Supplements
Food and supplements rich in omega 3 fatty acids help reduce inflammation throughout the body, lower blood pressure, and the risk of heart attack.
- Fish and Fish Oil- are high in omega 3 fatty acids, especially in fish like salmon, herring, and tuna. Fish oil supplements are also high in omega 3 fatty acids.
- Flax and flaxseed oil- contain omega 3 fatty acids and fiber.
- Greensoul- is a natural supplement that helps to maintain a healthy cholesterol level, supports the body’s immunity, and the ability to fight free radicals.
- Coenzyme Q10- helps maintain a healthy cholesterol level, boosts immunity, and energy.
- Perfusion SR- helps enhance blood flow, maintain a normal blood pressure, and enhance the elasticity of large arteries.
Procedures to restore blood flow
-
Angioplasty and stent or drug eluting stent placement: Most coronary blockages are treated with balloon angioplasty and coronary stenting. Angioplasty is the stretching of an artery to widen it, followed by stent placement.
This procedure is considered non-surgical because it is done by Dr. Jamnadas, who accesses the heart by inserting a long, thin tube (catheter) into the narrowed part of your artery.
A wire with a deflated balloon is passed through the catheter to the narrow area. The balloon is then inflated, compressing the plaque against the artery walls so it no longer restricts blood flow. Following this a stent may be placed to prevent restenosis.




There are two types of stents, bare metal stents and most of them today are drug coated called drug eluting stents.
Bare metal stents act as a scaffolding to keep an artery open, but suffer from a 30-40% re-stenosis rate. This is due to a process called intimal hyperplasia which is attributed to a keloid. A keloid is an overgrowth of the inner lining of the blood vessel that covers over the stent.
However, this reparative process lasts no more than 6 minutes. After 6 minutes, if an artery has not re-narrowed at the site of the stent, it is unlikely to re-narrow.
Drug eluting stents have a less aggressive reparative process and therefore the narrowing rate within the stent is less than 5%.
Drug eluting stents are one of the marvels of modern cardiology. Typically if a year has gone by with no evidence of re-narrowing, then that portion of the artery that has been stented will remain patent indefinitely in 99% of patients.
If symptoms of angina reoccur after one year, it is usually due to NEW blockage in a DIFFERENT part of the artery, therefore risk factor modification is needed to prevent new blockages. ** Many people have the misguided understanding that patients who have received multiple stents over the course of years have had stent failures. In the vast majority of these cases, it is a stent in a New location that was needed.
Following stent placement in addition to atherosclerosis risk factor modification, patients need to be put on two antiplatlet therapies. Plavix or Effient + Aspirin are recommended for one uninterrupted year of antiplatlet therapy.
It is important to know that a Plavix sensitivity test is done for those who have a drug eluting stent to assess patients responsive to Plavix. Those who are sensitive and have the drug eluting stent will require Plavix therapy up to 1 year until the stent is covered by the natural lining of the blood vessel wall. This also helps in preventing blood clots from forming.
For those with a bare metal stent applied, 1 month of Plavix therapy is required.

- Coronary artery bypass surgery: This is done in patients with one or more blocked coronary arteries that are bypassed by a blood vessel graft to restore normal blood flow to the heart. In those whom stent replacement is not feasible or if there is left main artery stenosis, a graft is usually taken from another area of your body either in the chest, arm, or leg. This will go around the clogged artery or arteries to create a new pathway for blood to flow to the heart.
-
Enhanced external counterpulsation (EECP): This is for patients who have angina (chest pain) and have already exhausted the standard tre
atments without successful results or for those who do not qualify for the other treatment regimens. This is a non-invasive procedure which promotes the formation of collaterals to bypass the clogged arteries as another means of getting oxygen rich blood to those areas of the heart that are not getting it. This is carried out in our office daily for 7 weeks.
How can I prevent coronary artery disease?
Healthy lifestyle habits can prevent coronary artery disease from developing.
Taking action in leading a healthy lifestyle can help keep your arteries strong, elastic, and free of plaque build up to ensure maximum blood flow.
These healthy habits include:
- NO Smoking
- Limiting alcohol
- Controlling high blood pressure, cholesterol, and diabetes.
- Eating a healthy diet
- Keep physically active with the right kind of exercise.
- Maintain a healthy weight with a BMI <25
- Get an annual flu shot
- Reduce stress!
Updated January 19, 2012- JS
For further information please contact Cardiovascular Interventions, PA at 407-894-4880
Cardiac Medications
- Coumadin
- Tab 2
- Tab 3
- Tab 4
- Tab 5
- Tab 6
- Tab 7
Coumadin (also called Warfarin)
What is it?
Coumadin is a prescribed medication that decreases the clotting ability of blood. This helps to keep blood flowing smoothly by preventing clots from forming in the blood vessels and heart.
Why do I need it?
Coumadin is prescribed to patients to prevent blood clots from forming or growing larger in the blood vessels.
Coumadin is required in patients who have:
-
A prosthetic heart valve
-
Atrial fibrillation (irregular heart rhythm)
-
Clotting disorders (i.e. antiphospholipid syndrome)
-
Had previous stroke or heart attack
-
Had a clot which has travelled to the lung (pulmonary embolism) or leg (deep vein thrombosis)
-
Recent surgeries (hip/knee replacement)
-
An existing clot to prevent it from growing larger
What tests do I need?
The two tests combined in one that are required to determine the clotting time are:
-
Prothrombin time (Protime, PT) – test that measures the time it takes for the clotting mechanism to progress. This is specifically sensitive to clotting factors that are affected by Coumadin.
-
International normalized ratio (INR) - expresses the PT in a standardized way to ensure that results taken from different laboratories can be reliably compared.
These are taken every 1-4 weeks and can alter the dosage of Coumadin you are taking based on the periodic blood tests to maintain the clotting time within a target range.
After taking Coumadin the general effect occurs within 24 hours, but the peak effect may be after 3-4 days. After a single dose of Coumadin the action can last up to 2-5 days. The effects may be more pronounced as the daily doses overlap. Coumadin is completely absorbed after oral administration with a peak concentration within the first 4 hours. It is excreted mostly in urine and the effects of a single dose of Coumadin can wear off in approximately 1 week.
How should I use it?
-
Coumadin should be taken Exactly as directed.
-
Take the dose once a day, around the same time every day.
-
If you forget to take your daily dose at the same time as normally, take the dose as soon as possible on the same day. In case you miss the dose that day, wait until the following day to take the regularly prescribed dose for that next day. DO NOT TAKE A DOUBLE DOSE.
-
If you forget to take your dose for 2 or more days in a row, call Dr. Jamnadas. He may need to change your dose.
-
Dosage ranges from 1-10 mg as scored tablets, making it easier to cut in half. Your dosage will be adjusted according to the results of your blood tests which are taken every 1-4 weeks.
-
Can be taken with or without food.
-
Refill your prescription 1 week before your supply ends to avoid missing a dose.
-
Continue taking your Coumadin as long as Dr. Jamnadas prescribes it.
-
Call our office before making any changes in Coumadin doses, even if suggested by another facility.
Where can I store this?
Keep this medication in the container provided, keeping it tightly closed.
Keep it stored at room temperature and away from excess heat, cold, moisture, and light. Do not store in the bathroom.
Keep out of reach of children.
What precautions should I know?
If you are taking these medications, they may interfere with Coumadin:
-
Amidarone or Digoxin (antiarrhythmic drug)
-
Antibiotics: Cephalosporins, Ciprofloxacin, Erythromycin, Sulfamethoxazole-Trimethoprim etc.
-
Anti-inflammatory: Ibuprofen (Advil or Motrin); Naproxen (Aleve or Naprosyn)
-
Antifungal: Fluconazole
-
Anticoagulants: Heparin, Plavix, or Aspirin
-
Medications for: cancer, cholesterol, colds and allergies, depression, mental illness, diabetes, heartburn or peptic ulcers, gout, seizures, tuberculosis, thyroid problems, birth control.
-
Herbal or botanical products
If you initiate, stop, or start, or change doses of any of these medications it will affect your PT and INR.
.
Do not take two or more medications containing Warfarin.
You are at higher risk of bleeding if you are taking Coumadin and:
- Are 65 years of age or older
- Have history of peptic ulcers
- Have high blood pressure
- Have history of stroke
- Have a serious heart disease
- Have a low blood count or cancer
- Have had trauma or surgery
- Have renal disorders
Let us know if you:
-
Are pregnant or think you might be pregnant or plan on being pregnant while taking Coumadin.
-
Are Breast-feeding
-
Have any bleeding tendencies
-
Are diabetic
-
Are having surgery including dental or medical surgery
-
Are consuming large amounts of alcohol
-
Use an indwelling catheter
-
Are receiving an immunization such as the flu shot
-
Are unwilling to take the medication or non-complaint
-
Wish to start a new medication including over the counter medications such as cold medicine or pain reliever.
What side effects can it cause?
Coumadin therapy may cause:
- Hemorrhage (bleeding): Coumadin can cause major or fatal bleeding. Bleeding is more likely to occur within the first month of use. Bleeding can occur from the nose, gums, or ears.
- Tissue necrosis (death of a tissue): necrosis or gangrene of the skin or other tissues is uncommon, but may lead to removal or amputation of affected part.
- Systemic atheroemboli and cholesterol micro emboli
- Thrombocytopenia: Low platelet counts. Bruises may that appear without reason or become swollen after time. Purplish spots on your skin may also be noticed.
- Hypersensitivity or allergic reactions.
- Vascular disorders like vasculitis (inflammation of the vessels)
- Gastrointestinal disorders: nausea, vomiting, diarrhea, or abdominal pain.
- Respiratory disorders: breathing difficulties or flu-like symptoms.
- Urogenital disorders: reddish or rusty colored urine.
- Skin disorders: rash, itching, or patchy baldness of hair
- Chills
What dietary instructions should I follow?
Follow a normal and healthy diet. Certain foods and beverages can affect the efficiency of Coumadin. It is important to pay attention to what you eat while taking this medication. Vitamin K can reduce the efficiency of Coumadin. Do not restrict the amount of vitamin K intake; just be consistent with the amount normally taken. Eating small amounts of foods that are rich in vitamin K should not cause any problems. Avoid eating or drinking large amounts of:
-
Green leafy vegetables including: collard and mustard greens, kale, parsley, Brussels sprouts, spinach, cabbage, and green tea.
-
Vegetable oils
-
Soybean or canola
-
Cranberry and grapefruit juice
Let Dr. Jamnadas or Dr. Kelly know if you are planning on eating more or less vegetables or decide to follow a special meal plan to lose weight.
Recommendations:
Prevent falls: Falling can increase the risk of bleeding. Try preventing falls in the house by removing loose rugs and electrical cords that can lead to slipping and falling. Ensure adequate lighting in your home, especially in stairways and entrances. Avoid walking on wet floors or any slippery surface or in unfamiliar areas.
Reduce the risk of bleeding: Since there is a tendency of bleeding, some simple changes can decrease the risk by
-
Using a soft bristle toothbrush
- Using waxed dental floss
- Shave with an electrical razor instead of a blade
- Take caution when using knives or scissors
- Avoid walking barefoot.
Dental procedures: including simple extraction of 3 teeth, gingival surgery, crown and bridge procedures, dental scaling, and surgical removal of teeth. It is NOT necessary to change your dose of Coumadin if your INR is below 4. The risks of discontinuing Coumadin, even for a short period of time, may cause irreversible changes due to a dislodged clot (thromboembolism) causing stroke or heart attack. Most dental procedures can be managed using routine local measures to control bleeding. The effects of minor bleeding are reversible. Consider the benefits and risks before discontinuing Coumadin.
Exercise: avoid any activity or contact sport that may cause serious injury.
Avoid excessive alcohol consumption: Alcohol can affect your ability to handle Coumadin. Drinking alcohol can increase the risk of injury and therefore bleeding.
Identification bracelet: Carry an identification bracelet stating that you take Coumadin. On the bracelet list your name, medical problems, medications and dosages, doctor’s name and telephone number.
Carry a list of medications: It is important to keep a written list of the medications you are taking weather it is prescribed or not prescribed (over the counter) and any other vitamins, minerals, or dietary supplements.
Updated January 31st, 2012 -JS
For further information please contact Cardiovascular Interventions, PA at 407-894-4880
Content 7
Heart Failure
What is it?
Heart failure is a condition that results when the heart (particularly the left ventricle) can not pump or fill with enough blood. Therefore, the heart must work harder to deliver oxygen rich blood to the body.
Heart ‘failure’ does not mean the heart has completely stopped or failed.
There are several types of heart failure. As the heart’s pumping action is weakened, blood may back up in and accumulate fluid in parts of the body; this is termed as congestive heart failure. Diminished pumping of the ventricles due to muscle weakening is termed as systolic dysfunction. After each ventricle contracts (systole) the ventricle need to relax (diastole) and fill with blood. During stiffening of the heart muscle, the ventricles are unable to completely relax, preventing the complete filling of blood in the ventricle; this is termed as diastolic dysfunction. The conditions may only affect one side of the heart; either the right or left side termed as right heart failure or left heart failure, although both sides are commonly involved.


What are the causes?
Heart failure is caused by a disease or condition that damages the heart.
Some of the most common causes include:
- Congenital heart disease- defects present from birth
- Hypertension (high blood pressure) - long standing or chronic hypertension forces the heart to work harder to pump blood. This constant stress increases the workload of the heart, causing it to enlarge.
- Coronary artery disease- narrowing of the blood vessels supplying the heart muscle deprives the heart of oxygen. This can lead to a heart attack which causes permanent damage to the heart and impairs the hearts function.
- Valvular Disease – aging, infection, previous heart attacks, or high blood pressure can damage the heart valves. Valvular disease may present as a narrowed or stenosed valve, limiting blood flow through the valve. A leaky or insufficient valve causes a backward flow or regurgitation. Valvular insufficiency and regurgitation may both be present.
- Cardiomyopathy – damage to the heart muscle leading to an enlarged, thick, and rigid heart.
- Other causes- chronic alcohol abuse, thyroid disorders, viral infections, and arrhythmias (irregular heart rhythm).
What are the symptoms?
Heart failure is characterized by specific symptoms and usually starts off slowly. Symptoms first start off during exertion then progress to symptoms at rest. Symptoms may also occur suddenly after a heart attack.
Common symptoms include:
- Easy fatigability

- Weakness, lightheadedness, or dizziness
- Shortness of breath or difficulty in breathing
- Fast or irregular heart rate

- Swelling (edema) of lower legs, feet, or abdomen (ascites)
What are the complications?
The most common complications of heart failure include:
- Cardiomyopathy- a weakened and enlarged heart muscle
- Arrhythmias- irregular heart rhythms that can lead to blood clot formation
- Heart attack
- Stroke
- Death
What is ejection fraction?
Ejection fraction (EF) is the amount of blood pumped out of the ventricles during each contraction.
This is measured to diagnose heart failure and the degree of heart failure.
A normal ejection fraction is 55- 70%. This will be decreased in a heart failure patient.
How is it diagnosed?
Heart failure is diagnosed based on your medical history, physical examination, and certain tests.
- Patient History
A significant history of previous heart attack, high blood pressure, diabetes, or alcohol can be used as clues.
Symptoms of heart failure may be told due to excess fluid accumulation such as shortness of breath, swelling, abdominal pain or distension.
Symptoms can also be due to decreased cardiac output including easy fatigability and weakness.
2. Physical Examination
You will be examined for signs of heart failure including:
- Rapid or irregular heart rate

- Rapid or difficulty in breathing
- Weight gain
- Leg swelling
- Distended neck veins
- Swelling in abdomen (ascites)
- Sounds (crepitations) may be heard over the lungs
3. Tests
A series of test may be performed to determine how well your heart is functioning and the cause of heart failure.
These tests include:
- Blood tests- B-type natriuretic peptide (BNP) is typically elevated from heart failure and can aid in the diagnosis. This is routinely followed for the response to treatment of congestive heart failure.
- Sodium and potassium levels will also be assessed.
- Electrocardiogram (EKG) - a noninvasive test performed in our clinic which determines the electrical activity of your heart. This is test is used to detect conditions associated with an abnormal heart rhythm or previous heart attack that may cause heart failure.
Chest X-ray- shows the size and shape of the heart. Also reveals the presence of fluid in the lungs. This is performed in our office.
- Echocardiogram (ECHO) - a noninvasive test performed in our office to determine the size and function of the heart’s chambers and structure and function of the heart valves.
- Stress test- determines how well the heart works during exercise.
- CT or MRI- is sometimes used to visualize the coronary arteries.
- Cardiac catheterization– a simple invasive procedure that helps measure the hearts function and visualize the arteries of the heart under x-ray guidance by the use of injecting a dye.
How is it treated?
Heart failure can be controlled and treated, but rarely completely goes away.
Treatment involves diet and lifestyle modification, medications, procedures, and sometimes surgery for advanced cases.
Diet and Lifestyle Modification

-
Salt and water restriction- manage the amount of salt in your diet. Eat less than 2000 mg (2 grams) of sodium per day. Read food labels to keep track of how much sodium you eat. Canned food, frozen, and processed foods are high in sodium. Make sure to check the amount of sodium in each serving. Do not add salt to food when you are cooking, instead season food with lemon, pepper, garlic, or onion. Reducing salt intake improves high blood pressure that may be the cause of heart failure or may lead to worsening of heart failure.
- Water intake should be limited. Patients with severe heart failure should drink less than 2 liters of fluid per day, including all fluids. Only drink when you are thirsty and measure drinks in a cup before you drink them. Suck on lemon wedges to quench thirst and suck on hard candy to keep your mouth moist.
Weigh yourself everyday- if there is weight gain of more than 2 pounds in one day or weight increases by 4 pounds in one week, see your physician. Weight gain is a sign that you may be retaining more fluid than you should be.
- Control weight- maintain a healthy weight with a BMI less than 25. Extra weight makes the heart work harder. By reducing your weight you can reduce the strain on your heart.
- Do not smoke- smoking increases the risk of heart attack which can worsen heart failure.
- Limit alcohol- limit alcohol to one serving per day if you choose to drink.
- Exercise- ask your physician which exercise routine is best for you. Exercise for at least 30 minutes a day 5 days a week. This can improve symptoms of shortness of breath and fatigue.
Medications
Most heart failure patients benefit from the use of one or more medications. Medications are used to improve symptoms and prolong survival.
The most common heart failure medications include:
- Diuretics- these are sometimes referred to as ‘water pills’ that is used for patients with leg swelling or fluid in the lungs. This helps excrete excess water and has been demonstrated to impact a long term survival.
- ACE inhibitors or ARBs- dilate blood vessels and lower blood pressure making it easier for the heart to pump, which is especially useful in those with heart muscle weakness. ACE inhibitors sometimes cause dry cough, therefore ARBs may be preferred.
- Beta blockers- this lowers the heart rate and decreases blood pressure.
- Digoxin- this helps the heart muscle to contract with more strength, allowing more blood to be pumped with each beat.
- Anticoagulants- patients with heart failure and with an irregular heart rhythm are at risk for developing a blood clot. To prevent this, aspirin or warfarin (Coumadin) may be started.
Procedures
-
Heart rhythm treatment- for patients with a life-threatening abnormal heart rhythm, a device may be recommended to you. An implantable cardioverter-defibrillator (ICD) is an implantable device that detects abnormal rhythms and shocks the heart to return it to a normal rhythm.
- Cardiac resynchronization therapy (CRT) is a treatment used in patients with an abnormal electrical conduction in the heart whose ventricles contract out of sync. This is a special type of pacemaker used to treat the problem and keep the contraction of the heart in sync.
-
Artery and valvular treatment- procedures used to improve blood flow to help the heart pump better and improve symptoms. This can be done with cardiac catheterization with angioplasty or stenting. Bypass surgery allows blood to flow around a clogged artery.
- Valvular disease may require valve repair or replacement.
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Tissue Valve |
Mechanical Valve |
Treatment in advanced heart failure
Heart transplantation can be helpful in patients with severe heart failure or those who do not respond to other treatments.
You may be recommended a left ventricular assist device (LVAD) instead of transplantation or during the waiting period for transplantation. This is an implantable device used as a temporary mode of support to improve blood flow.
What is the prognosis?
The prognosis of a patient depends on:
- The nature of the underlying disease
- The response to medications
- The degree of involvement of other organ systems
- The degree of symptoms and impairment
The prognosis of heart failure is associated with the patient’s grade of functioning. Heart failure is graded on a scale of I to IV
- Class I- patients with no symptoms or limitation of physical activity
- Class II- patients with symptoms and slight limitation of physical activity
- Class III- patients with symptoms on ordinary activity and limitation of physical activity
- Class IV- patients symptoms at rest
Heart failure is often a progressive condition that may lead to the development and worsening of clinical illness. Some people may be at risk of deadly heart rhythms.
Heart failure can be controlled by following a healthy lifestyle, being compliant with medications and other treatments as well as treating the cause of heart failure (i.e. high blood pressure).
Heart failure may get worse due to:
- Angina (chest pain)
- High salt intake
- Heart attacks
- Infections
- Improper medication usage
How to live with heart failure:
Some guidelines to help manage your condition include:
- See your physician regularly and follow instructions closely.

- Never stop taking medication without consulting your physician. Do not skip doses when you feel better or can not afford medication. Talk to your doctor about other treatment options.
- Try to eat foods without added salt
- Monitor blood pressure, pulse, and weight regularly
- Do not smoke
- Ask your doctor about receiving a flu shot. This can be important in preventing the effects of disease.
- Look for signs of worsening of heart failure like worsened shortness of breath or cough, worsening of leg swelling, weight gain of 2 pounds in one day, or fast or irregular heartbeat.
Updated March 19th, 2012 -JS
For further information please contact Cardiovascular Interventions, PA at 407-894-4880
Devices
- Pacemakers
- Biventricular Pacemakers
- ICD
- Biventricular ICD
- Holter Monitor
- Event Monitor
Pacemaker
What is a pacemaker?
A pacemaker is a small device that sends electrical impulses to the cardiac muscle. This helps to regulate a normal heart rate and rhythm.
Insertion of a pacemaker is known as implantation. A pacemaker is placed just below the skin near your heart to help regulate the heart beat.

Who needs it?
A pacemaker is used to treat patients with:
- Slow Atrial Fibrillation
- Bradycardia (slow heart rate)
- Heart Block (electrical impulses not continuing throughout pathway properly)
- Sick sinus syndrome (alternating slow and fast rhythms of the heart)
- Congestive heart failure
How a normal heart beats:
In a normal heart, the muscles and chambers within the heart synchronize in a regulated type of harmony together. This occurs only when there is a normal rate and rhythm of the heart.
If the heart is beating too slow or too fast, sufficient amount of blood is unable to flow throughout the body. When this happens patients may get symptoms of arrhythmia. These symptoms include fatigue, shortness of breath, fainting, or loss of consciousness.
The electrical signals of the heart follow through a conduction pathway. The origin of this pathway is from the Sinoatrial node that is known as the natural pacemaker of the heart. The impulses are transmitted throughout the heart keeping it beating in sync in rate and rhythm.
What does a pacemaker do?
When the Sinoatrial node is not functioning properly, a pacemaker is needed to help initiate the correct electrical impulses. The pacemaker acts by mimicking the Sinoatrial node, generating a normal heart rate and rhythm.
![]() |
©2012 St. Jude Medical, Inc. |
A pacemaker consists of:
-
Battery: the pacemaker battery supplies the power to the pacemaker. This is a small, sealed, lithium battery that can lasts for years. Battery life usually lasts 5-10 years depending on how active your pacemaker is.
-
Computerized generator: this is a miniature computer inside the pacemaker. The battery transmits electrical impulses that go through this generator to stimulate the heart to beat.
- Case: the battery and computer generator are sealed inside a metal casing.
- Leads: are flexible wires connecting the generator to the walls of the heart.
Pacemakers not only monitor your heart beat, but also your body motion and breathing pattern. This can signal the pacemaker to increase during times of exercise.
What are the different types of pacemakers?
Depending on the condition of the heart, Dr. Jamnadas or Dr. Kelly will determine the number of chambers in your heart that need to be paced.
Pacemakers can be implanted temporarily or permanently.
The different types of pacemakers are:
-
Single Chamber pacemakers: this uses one lead which connects either to the right atrium or ventricle.
- Dual Chamber pacemakers: this uses two leads with one connecting to the right atrium and the second connecting to the right ventricle to make the heart beat more efficiently.
- Biventricular pacemakers: this uses 3 leads, one in the right atrium, and one in each ventricle. This treatment resets the ventricular pumping system and is referred to as Cardiac Resynchronization Therapy (CRT).
What are the risks?
Although risks are very rare, as with any surgical procedure it does carry a small risk of complication.
These complications include:
- Allergy: may produce an allergic reaction to the dye.
- Swelling, bruising, or bleeding at or around the implantation site.
- Formation of a clot (hematoma).
- Infection: can occur where there pacemaker was implanted.
- Damage to blood vessels or nerves around pacemaker site
- Collapse lung
How is this diagnosed?

Dr. Jamnadas or Dr. Kelly will discuss with you about your history and symptoms.
Following this a physical examination will be performed to get a better idea of your condition.
A number of different tests can be done to find out the cause of irregular heartbeats including:
-
Electrocardiogram (EKG) – a simple, non-invasive test that records the electrical activity of the heart and its beating pattern.
- Holter monitor- records the electrical events of the heart during your normal daily activities. It is important to accurately record your activities and symptoms so Dr. Jamnadas or Dr. Kelly can compare them to the Holter monitor findings. If symptoms do not occur within 24 hours, an event recorder may be ordered for you for up to 30 days.
- Echocardiogram (ECHO) - shows images of the heart determining the size and shape of the heart, and shows whether the heart walls and pumping activity are normal or performing weakly.
- Stress test- helps access the blood flow to the heart at rest and during stress. Will detect if any areas receiving less blood flow.
What to expect:
Before the procedure:
Today pacemaker devices are amazingly small and average about 5 x 5cms, weighing only 1 to 3 ounces.
A pacemaker is usually implanted just beneath the skin of your chest, below the collarbone.
Pacemakers are typically placed on the left side of the chest, but this may not be suitable for everyone.
The procedure will take place within a hospital and does not require general anesthesia.
You may or may not stay over night in the hospital.
You will need to arrange for someone to pick you up and drop you off.
During the procedure:
The entire procedure usually takes about one to two hours.
A sedative will be given to help you relax through an i.v. in your hand or arm that may make you sleepy.
Under sterile precautions, Dr. Jamnadas will inject a local anesthetic until you are fully numb in the area where the pacemaker will be inserted.
A needle will be inserted into a large vein to get access to the heart.
A dye will be given to view the heart under x-ray guidance.
To enter the heart, the pacemaker leads will be threaded through the vein and fixed to the wall of the right atrium or ventricle or both, depending on your pacemaker.
The other end of the lead will be secured to the generator box.
The pacemaker settings are programmed to help your heart beat at a rate that is right for you.![]()
Once the leads are fixed, Dr. Jamnadas will place a single incision just below the collar bone to insert the pacemaker underneath the skin of the chest.
After the pacemaker is placed within the chest, it will be tested to make sure it is working properly.
The incision will then be closed and a dressing will be applied.
After the procedure:
After the procedure you may feel some mild pain, tenderness, or swelling in the area of implantation. Full recovery from surgery can take several days to weeks.
On the side of incision, Do NOT raise your arm above your shoulder for a week. This will give the lead time to attach to the heart wall properly. Raising your arm can make the lead separate from the site of attachment.
Do not drive until you know how your pacemaker affects you.
Do not be afraid to exercise. Ask Dr. Jamnadas which exercise plan is best for you and when it is best to start.
Make sure to:
-
Change your dressing as often as instructed and avoid getting the area wet for about a week.
- Take your temperature and check the incision everyday for signs of infection like redness, discharge, or a local rise in temperature.
- Learn how to take your own pulse and keep record of it.
- Avoid putting direct pressure on your pacemaker. For example, do not lie on your chest while sleeping at night.
- Let your primary health care provider or any other healthcare providers as well as dentist know about your pacemaker before receiving any treatment.
- Carry an ID card that contains information about your pacemaker. Your pacemaker may set off a metal detector while going through security. You may need to show this card to security personnel.
- Keep your cellular phone away from your pacemaker. Do not carry your phone in your shirt pocket, even when it is turned off.
Avoid strong magnets including MRI scans and hand held security wands.
- Avoid strong electrical fields such as those made by radio transmitting towers, ham radios, and heavy duty electrical equipment.
- Avoid leaning over the open hood of a running car. A running engine creates am electrical field.
Keep all follow up appointments to check your pacemaker and lead. Following the surgery, an x-ray will be required to make sure the position of the lead is still intact.
Follow up appointments will be scheduled for you in the initial one month, and then the following 2 months, then every 3 months to get your device checked.
What can I use?
The following items are all ok to use:
- Microwave ovens

- Computers
- Hair dryers

- Power tools
- Radios
- Televisions
- Stereos

- Electric blankets
- Vacuum cleaners
- Heating pads
- Cars
Call Dr. Jamnadas if you experience any symptoms of:
-
Dizziness
- Palpitations
- Shortness of breath
- Fatigue
- Fainting spells
- Twitching chest muscles
- Chest pain or pain at pacemaker site
- Fever
- Hiccups that won’t stop
Updated January 27th, 2012- JS
For further information please contact Cardiovascular Interventions, PA at 407-894-4880
Biventricular Pacemaker
What is it?
A biventricular pacemaker a small device used for cardiac resynchronization therapy for patients with heart failure. This delivers small electrical impulses to both the left and right ventricles to help them contract at the same time. This helps the heart to pump blood more efficiently with each beat.

Who needs it?
A biventricular pacemaker is used to treat patients with:
- Moderate to severe heart failure
- Ejection fraction <35% (the amount of blood pumped out of the heart with each beat)
- Ventricular dysynchrony
- Interventricular conduction delay or bundle branch block
- Symptoms of heart failure not corrected with medication at maximum dosage
How a normal heart beats:
In a normal heart, the muscles and chambers of the heart contract and relax in a synchronized regulated type of harmony together. This timed synchronization or rhythm allows the ventricles to relax and fill with sufficient amount of blood then contract, pumping out a sufficient amount of blood throughout the body.
How does it work?
In a patient with heart failure, the chambers of the heart are not in sync with each other. This leads to an inadequate blood supply throughout the body and can lead to symptoms of heart failure. These symptoms include shortness of breath, dry cough, swelling of the legs, irregular heart beats, or fatigue.
A biventricular pacemaker sends a steady pattern of electrical impulses first to the right atrium then to both the right and left ventricles of the heart.
These small impulses help by keeping the contractions in sync and in a regular rhythm to provide adequate blood flow throughout the body.
This treatment resets the ventricular pumping system and is referred to as Cardiac Resynchronization Therapy (CRT).

What are the parts of a biventricular pacemaker?
A biventricular pacemaker consists of:
- Battery: the pacemaker battery supplies the power to the pacemaker. This is a small, sealed, lithium battery that can lasts for years. Battery life usually lasts 5-10 years depending on how active your pacemaker is.

- Computerized generator: this is a miniature computer inside the pacemaker. The battery transmits electrical impulses that go through this generator to stimulate the heart to beat.
- Case: the battery and computer generator are sealed inside a metal casing.
- Leads: 3 flexible wires connect the generator to the walls of the heart.
Biventricular Pacemakers not only monitor your heart beat, but also your body motion and breathing pattern. This can signal the pacemaker to increase during times of exercise.
What are the risks?
Although risks are very rare, as with any surgical procedure it does carry a small risk of complication.
These complications include:
- Allergy: may produce an allergic reaction to the dye.
- Swelling, bruising, or bleeding at or around the implantation site.
- Formation of a clot (hematoma).
- Infection: can occur where there pacemaker was implanted.
- Damage to blood vessels or nerves around pacemaker site
- Collapse lung
- Device may move out of place from the heart.
- Device might not detect or appropriately treat your heart rhythm.
How is this diagnosed?

Dr. Jamnadas or Dr. Kelly will discuss with you about your history and symptoms.
Following this a physical examination will be performed to get a better idea of your condition.
A number of different tests can be done including:
- Electrocardiogram (EKG) – a simple, non-invasive test that records the electrical activity of the heart and its beating pattern.
- Holter monitor- records the electrical events of the heart during your normal daily activities. It is important to accurately record your activities and symptoms so Dr. Jamnadas or Dr. Kelly can compare them to the Holter monitor findings. If symptoms do not occur within 24 hours, an event recorder may be ordered for you for up to 30 days.
- Echocardiogram (ECHO) - shows images of the heart determining the size and shape of the heart, and shows whether the heart walls and pumping activity are normal or performing weakly.
- Stress test- helps access the blood flow to the heart at rest and during stress. Will detect if any areas receiving less blood flow.
What to expect:
Before the procedure:
Today pacemaker devices are amazingly small and average about 5 x 5cms, weighing only 1 to 3 ounces.
A biventricular pacemaker is usually implanted just beneath the skin of your chest, below the collarbone.
Biventricular pacemakers are typically placed on the left side of the chest, but this may not be suitable for everyone.
The procedure will take place within a hospital and does not require general anesthesia.
You may or may not stay over night in the hospital. Pack an overnight bag just in case.
You will need to arrange for someone to pick you up and drop you off.
During the procedure:
The entire procedure usually takes about one to two hours.
A sedative will be given to help you relax through an I.V. in your hand or arm that may make you sleepy.
Under sterile precautions, Dr. Jamnadas will inject a local anesthetic until you are fully numb in the area where the pacemaker will be inserted.
A needle will be inserted into a large vein to get access to the heart.
A dye will be given to view the heart under x-ray guidance.
To enter the heart, the pacemaker leads will be threaded through the vein and fixed to the wall of the right atrium and both right and left ventricles. The other
end of the leads will be secured to the generator box.
The pacemaker settings are programmed to help your heart beat at a rate that is right for you.
Once the leads are fixed, Dr. Jamnadas will place a single incision just below the collar bone to insert the pacemaker underneath the skin of the chest.
After the pacemaker is placed within the chest, it will be tested to make sure it is working properly.
The incision will then be closed and a dressing will be applied.
After the procedure:
After the procedure you may feel some mild pain, tenderness, or swelling in the area of implantation. Full recovery from surgery can take several days to weeks.
On the side of incision, DO NOT raise your arm above your shoulder for a week. This will give the lead time to attach to the heart wall properly. Raising your arm can make the lead separate from the site of attachment.
Do not drive until you know how your pacemaker affects you.
Do not be afraid to exercise. Ask Dr. Jamnadas which exercise plan is best for you and when it is best to start.
Make sure to:
- Change your dressing as often as instructed and avoid getting the area wet for about a week.
- Take your temperature and check the incision everyday for signs of infection like redness, discharge, or a local rise in temperature.
- Learn how to take your own pulse and keep record of it.
- Avoid putting direct pressure on your pacemaker. For example, do not lie on your chest while sleeping at night.
- Let your primary health care provider or any other healthcare providers as well as dentist know about your pacemaker before receiving any treatment.
- Carry an ID card that contains information about your pacemaker. Your pacemaker may set off a metal detector while going through security. You may need to show this card to security personnel.
- Keep your cellular phone away from your pacemaker. Do not carry your phone in your shirt pocket, even when it is turned off.

- Avoid strong magnets including MRI scans and hand held security wands.
- Avoid strong electrical fields such as those made by radio transmitting towers, ham radios, and heavy duty electrical equipment.
- Avoid leaning over the open hood of a running car. A running engine creates am electrical field.
Keep all follow up appointments to check your pacemaker and leads. Following the surgery, an x-ray will be required to make sure the position of the lead is still intact.
What can I use?
The following items are all ok to use:
Microwave ovens
- Computers
- Hair dryers
- Power tools

- Radios
- Televisions
- Stereos
- Electric blankets
- Vacuum cleaners

- Heating pads
- Cars
Call Dr. Jamnadas if you experience any symptoms of:
- Dizziness
- Palpitations
- Shortness of breath
- Fatigue
- Fainting spells
- Twitching chest muscles
- Chest pain or pain at pacemaker site
- Fever
- Hiccups that won’t stop
Updated February 6, 2012-JS
For further information please contact Cardiovascular Interventions, PA at 407-894-4880
Implantable Cardioverter Defibrillators (ICDs)
What is an ICD?
An ICD is a small device that monitors the electrical activity of the heart. This not only evaluates the hearts activity, but can also store the electrical events so Dr. Jamnadas can assess them.
In the event a dangerously fast heart rhythm is detected, the ICD quickly delivers therapy in the form of electrical energy.
This helps to regulate a normal heart rate and rhythm.
![teligin-hand_large[1].JPG](erc_clip_image002_0013.jpg)
Who needs it?
An ICD is used to treat patients with:
-
Ventricular Fibrillation
- Ventricular Tachycardia
- Congestive Heart Failure
- Cardiomyopathy
- Long QT syndrome
How a normal heart beats:
In a normal heart, the muscles and chambers within the heart synchronize in a regulated type of harmony together. This occurs only when there is a normal rate and rhythm of the heart.
If the heart is beating too slow or too fast, sufficient amount of blood is unable to flow throughout the body. When this happens patients may get symptoms of arrhythmia. These symptoms include fatigue, shortness of breath, fainting, or loss of consciousness.
The electrical signals of the heart follow through a conduction pathway. The origin of this pathway is from the Sinoatrial node that is known as the natural pacemaker of the heart. The impulses are transmitted throughout the heart keeping it beating in sync in rate and rhythm.
What does an ICD do?
An ICD is needed when the heart has the potential to develop a dangerously fast heart rhythm due to the improper activity of the hearts electrical system.
As soon as this occurs, the ICD will send a shock to the heart muscle to defibrillate it or stop the cycle of rapid twitching to normalize the hearts rhythm.
The shock sensation varies amongst people. Some may not even be aware of the shock, where as others may be uncomfortable after responding to such a dangerous rhythm.
An ICD consists of:
- Battery: the ICD battery supplies the power to the ICD. This is a small, sealed, lithium battery that can lasts for years. Battery life usually lasts 5-10 years depending on how active your ICD is.
- Computerized generator: this is a miniature computer to evaluate the heart rate, store it in memory, and help the device know when to deliver therapy. The battery transmits electrical impulses that go through this generator to stimulate the heart to beat.
- Case: the battery and computer generator are sealed inside a metal casing.
- Leads: are flexible wires connecting the generator to the walls of the heart.
What are the different types of ICDs?
Depending on the condition of the heart, Dr. Jamnadas or Dr. Kelly will determine the right ICD for you.
The different types of ICDs are categorized by the type of pacemaker they contain:
- ICD with a single chamber pacemaker: this paces in one chamber of the heart. This uses one lead which connects either to the right atrium or ventricle.
- Dual chamber ICD with dual chamber pacemaker: this uses two leads with one connecting to the right atrium and the second connecting to the right ventricle to make the heart beat more efficiently.
- Bi ventricular ICDs: pace in three chambers of the heart including the right atrium and the left and right ventricles.
What are the risks?
Although risks are very rare, as with any surgical procedure it does carry a small risk of complication.
These complications include:
- Allergy: may produce an allergic reaction to the dye.
- Swelling, bruising, or bleeding at or around the implantation site.
- Formation of a clot (hematoma).
- Infection: can occur where there ICD was implanted.
- Damage to blood vessels or nerves around ICD site.
- Collapse lung
How is this diagnosed?

Dr. Jamnadas or Dr. Kelly will discuss with you about your history and symptoms.
Following this a physical examination will be performed to get a better idea of your condition.
A number of different tests can be done to find out the cause of irregular heartbeats including:
- Electrocardiogram (EKG) – a simple, non-invasive test that records the electrical activity of the heart and its beating pattern.
- Holter monitor- records the electrical events of the heart during your normal daily activities. It is important to accurately record your activities and symptoms so Dr. Jamnadas or Dr. Kelly can compare them to the Holter monitor findings. If symptoms do not occur within 24 hours, an event recorder may be ordered for you for up to 30 days.
- Echocardiogram (ECHO) - shows images of the heart determining the size and shape of the heart, and shows whether the heart walls and pumping activity are normal or performing weakly.
- Stress test- helps access the blood flow to the heart at rest and during stress. Will detect if any areas are receiving less blood flow.
What to expect:
Before the procedure:
Today ICDs devices are amazingly small and average about 5 x 5cms, weighing only 1 to 3 ounces.
An ICD is usually implanted just beneath the skin of your chest, below the collarbone.
ICDs are typically placed on the left side of the chest, but this may not be suitable for everyone.
The procedure will take place within a hospital and does not require general anesthesia.
You may or may not stay over night in the hospital.
You will need to arrange for someone to pick you up and drop you off.
During the procedure:
The entire procedure usually takes about one to two hours.
A sedative will be given to help you relax through an i.v. in your hand or arm that may make you sleepy.
Under sterile precautions, Dr. Jamnadas will inject a local anesthetic until you are fully numb in the area where the ICD will be inserted.
A needle will be inserted into a large vein to get access to the heart.
A dye will be given to view the heart under x-ray guidance.
To enter the heart, the ICD leads will be threaded through the vein and fixed to the wall of the right atrium or ventricle or both, depending on your ICD. The other end of the lead will be secured to the generator box. 
The ICD settings are programmed to help your heart beat at a rate that is right for you.
Once the leads are fixed, Dr. Jamnadas will place a single incision just below the collar bone to insert the ICD underneath the skin of the chest.
After the ICD is placed within the chest, it will be tested to make sure it is working properly.
The incision will then be closed and a dressing will be applied.
After the procedure:
After the procedure you may feel some mild pain, tenderness, or swelling in the area of implantation. Full recovery from surgery can take several days to weeks.
On the side of incision, Do NOT raise your arm above your shoulder for a week. This will give the lead time to attach to the heart wall properly. Raising your arm can make the lead separate from the site of attachment.
Do not drive until you know how your ICD affects you.
Do not be afraid to exercise. Ask Dr. Jamnadas which exercise plan is best for you.
Make sure to:
- Change your dressing as often as instructed and avoid getting the area wet for about a week.
- Take your temperature and check the incision everyday for signs of infection like redness, discharge, or a local rise in temperature.
- Learn how to take your own pulse and keep record of it.
- Avoid putting direct pressure on your ICD. For example, do not lie on your chest while sleeping at night.
- Let your primary health care provider or any other healthcare providers as well as dentist know about your ICD before receiving any treatment.
- Carry an ID card that contains information about your ICD. Your ICD may set off a metal detector while going through security. You may need to show this card to security personnel.
- Keep your cellular phone away from your ICD. Do not carry your phone in your shirt pocket, even when it is turned off.

- Avoid strong magnets including MRI scans and hand held security wands.
- Avoid strong electrical fields such as those made by radio transmitting towers, ham radios, and heavy duty electrical equipment.
- Avoid leaning over the open hood of a running car. A running engine creates am electrical field.
Keep all follow up appointments to check your ICD and lead. Following the surgery, an x-ray will be required to make sure the position of the lead is still intact.
Follow up appointments will be scheduled for you in the initial one month, and then the following 2 months, then every 3 months to get your device checked.
What can I use?
The following items are all ok to use:
- Microwave ovens

- Computers
- Hair dryers

- Power tools
- Radios
- Televisions
- Stereo
- Electric blankets

- Vacuum cleaners
- Heating pads
- Cars
Call Dr. Jamnadas if you experience any symptoms of:
-
Dizziness
-
Palpitations
-
Shortness of breath
-
Fatigue
-
Fainting spells
-
Twitching chest muscles
-
Chest pain or pain at ICD site
-
Fever
-
Hiccups that won’t stop
Updated January 27th, 2012- JS
For further information please contact Cardiovascular Interventions, PA at 407-894-4880
Biventricular Implantable Cardioverter Defibrillators
(ICDs)
What is it?
A biventricular ICD a small device used for cardiac resynchronization therapy for patients with heart failure. This constantly monitors your heart rate and rhythm and delivers small electrical impulses to both the left and right ventricles to help them contract at the same time.
This helps the heart to pump blood more efficiently with each beat. If any abnormal rhythm is detected, the ICD will deliver a shock to the heart back into a normal rhythm again.
This helps in treating dangerously fast rhythms that can lead to sudden cardiac arrest.
Who needs it?
A biventricular ICD is used to treat patients with:
- Moderate to severe heart failure with high risk of sudden cardiac death.
- Ejection fraction <35% (the amount of blood pumped out of the heart with each beat)
- Ventricular fibrillation
- Sustained ventricular tachycardia
- Interventricular conduction delay or bundle branch block
- Cardiomyopathy
How a normal heart beats:
In a normal heart, the muscles and chambers of the heart contract and relax in a synchronized regulated type of harmony together.
This timed synchronization or rhythm allows the ventricles to relax and fill with sufficient amount of blood then contract, pumping out a sufficient amount of blood throughout the body.
How does it work?
In a patient with heart failure, the chambers of the heart are not in sync with each other. This leads to an inadequate blood supply throughout the body and can lead to symptoms of heart failure.
Symptoms include shortness of breath, dry cough, swelling of the legs, irregular heart beats, or fatigue.
A biventricular ICD not only monitors the heart rate and rhythm but also senses dangerously fast heartbeats that can lead to cardiac arrest. If the device senses a life threatening heart beat, it will deliver a shock to the heart. This shock or defibrillation stops the abnormal rhythm and sets the heart into a regular rhythm again. This shock helps keep the contractions of the heart in sync and in a regular rhythm to provide adequate blood flow throughout the body.
This treatment resets the ventricular pumping system and is referred to as Cardiac Resynchronization Therapy (CRT).
What are the parts of a biventricular ICD?
A biventricular ICD consists of:
- Battery: the ICD battery supplies the power to the ICD. This is a small, sealed, lithium battery that can lasts for years. Battery life usually lasts 5-10 years depending on how active your ICD is.
- Computerized generator: this is a miniature computer inside the ICD. The battery transmits electrical impulses that go through this generator to stimulate the heart to beat.

- Case: the battery and computer generator are sealed inside a metal casing.
- Leads: 3 flexible wires connect the generator to the walls of the heart.
Biventricular ICDs not only monitor your heart beat, but also your body motion and breathing pattern. This can signal the ICD to increase during times of exercise.
What are the risks?
Although risks are very rare, as with any surgical procedure it does carry a small risk of complication.
These complications include:
- Allergy: may produce an allergic reaction to the dye.
- Swelling, bruising, or bleeding at or around the implantation site.
- Formation of a clot (hematoma).
- Infection: can occur where there ICD was implanted.
- Damage to blood vessels or nerves around ICD site
- Collapse lung
- Device may move out of place from the heart.
- Device might not detect or appropriately treat your heart rhythm.
How is this diagnosed?

Dr. Jamnadas or Dr. Kelly will discuss with you about your history and symptoms.
Following this a physical examination will be performed to get a better idea of your condition.
A number of different tests can be done including:
- Electrocardiogram (EKG) – a simple, non-invasive test that records the electrical activity of the heart and its beating pattern.
- Holter monitor- records the electrical events of the heart during your normal daily activities. It is important to accurately record your activities and symptoms so Dr. Jamnadas or Dr. Kelly can compare them to the Holter monitor findings. If symptoms do not occur within 24 hours, an event recorder may be ordered for you for up to 30 days.
- Echocardiogram (ECHO) - shows images of the heart determining the size and shape of the heart, and shows whether the heart walls and pumping activity are normal or performing weakly.
- Stress test- helps access the blood flow to the heart at rest and during stress. Will detect if any areas receiving less blood flow.
What to expect:
Before the procedure:
Today ICD devices are amazingly small and average about 5 x 5cms, weighing only 1 to 3 ounces.
A biventricular ICD is usually implanted just beneath the skin of your chest, below the collarbone.
Biventricular ICDs are typically placed on the left side of the chest, but this may not be suitable for everyone.
The procedure will take place within a hospital and does not require general anesthesia.
You may or may not stay over night in the hospital. Pack an overnight bag just in case.
You will need to arrange for someone to pick you up and drop you off.
During the procedure:
The entire procedure usually takes about one to two hours.
A sedative will be given to help you relax through an I.V. in your hand or arm that may make you sleepy.
Under sterile precautions, Dr. Jamnadas will inject a local anesthetic until you are fully numb in the area where the ICD will be inserted.
A needle will be inserted into a large vein to get access to the heart.
A dye will be given to view the heart under x-ray guidance.
To enter the heart, the ICD leads will be threaded through the vein and fixed to the wall of the right atr
ium and both right and left ventricles. The other end of the leads will be secured to the generator box.
The ICD settings are programmed to help your heart beat at a rate that is right for you.
Once the leads are fixed, Dr. Jamnadas will place a single incision just below the collar bone to insert the ICD underneath the skin of the chest.
After the ICD is placed within the chest, it will be tested to make sure it is working properly.
The incision will then be closed and a dressing will be applied.
After the procedure:
After the procedure you may feel some mild pain, tenderness, or swelling in the area of implantation. Full recovery from surgery can take several days to weeks.
On the side of incision, DO NOT raise your arm above your shoulder for a week. This will give the lead time to attach to the heart wall properly. Raising your arm can make the lead separate from the site of attachment.
Do not drive until you know how your ICD affects you.
Do not be afraid to exercise. Ask Dr. Jamnadas which exercise plan is best for you and when it is best to start.
Make sure to:
- Change your dressing as often as instructed and avoid getting the area wet for about a week.
- Take your temperature and check the incision everyday for signs of infection like redness, discharge, or a local rise in temperature.
- Learn how to take your own pulse and keep record of it.
- Avoid putting direct pressure on your ICD. For example, do not lie on your chest while sleeping at night.
- Let your primary health care provider or any other healthcare providers as well as dentist know about your ICD before receiving any treatment.
- Carry an ID card that contains information about your ICD. Your ICD may set off a metal detector while going through security. You may need to show this card to security personnel.
- Keep your cellular phone away from your ICD. Do not carry your phone in your shirt pocket, even when it is turned off.

- Avoid strong magnets including MRI scans and hand held security wands.
- Avoid strong electrical fields such as those made by radio transmitting towers, ham radios, and heavy duty electrical equipment.
- Avoid leaning over the open hood of a running car. A running engine creates am electrical field.
Keep all follow up appointments to check your ICD and leads. Following the surgery, an x-ray will be required to make sure the position of the lead is still intact.
What can I use?
The following items are all ok to use:
- Microwave ovens

- Computers
- Hair dryers

- Power tools
- Radios
- Televisions
- Stereos

- Electric blankets
- Vacuum cleaners
- Heating pads
- Cars
Call Dr. Jamnadas if you experience any symptoms of:
- Dizziness
- Palpitations
- Shortness of breath
- Fatigue
- Fainting spells
- Twitching chest muscles
- Chest pain or pain at ICD site
- Fever
- Hiccups that won’t stop
Updated February 6, 2012-JS
For further information please contact Cardiovascular Interventions, PA at 407-894-4880
Holter Monitor
What is it?
A Holter monitor is a small portable device used to record the electrical activity of the heart. A Holter monitor is typically worn for 24 hours to record each heart beat during your normal daily activity. This is performed to detect any irregular heart rhythms that may not be picked up on a routine electrocardiogram (EKG) during your office visit.

Who needs it?
A Holter monitor may be ordered in a patient for a number of reasons. Some indications for a Holter monitor include:
- Arrhythmia (irregular heartbeat)
- Atrial Fibrillation/ Flutter
- Atrial tachycardia
- Palpitations
- Supraventricular tachycardia
- Syncope
- Bradycardia (slow heart rate)
- Ventricular Tachycardia
- After a heart attack
- New cardiac medication usage
How do I prepare for the test?
There is no specific preparation before having a Holter monitor put on. Make sure to shower before the test as you will not be permitted to while wearing the monitor.
The test is a painless test. Most people do not notice they even have the monitor on.
There is no risk associated with this test. It is important to avoid getting the device wet, as it is an electrical device. Some things to avoid during the 24 hours include electric blankets, high-voltage areas, magnets, and metal detectors.
How is it performed?
Several small sticky electrodes will be placed on the chest and attached to the Holter monitor. These can easily be hidden under your clothes. The Holter monitor is small enough to attach to your belt or may be worn around the neck or waist. The Holter monitor will be worn for 24 hours throughout your normal daily activities including exercise, eating, sleeping, and even when you take your daily medications. It is important to make a dairy of your activities. Make sure to include the time of day of each activity. Any symptoms you might feel should be accurately recorded. After 24 hours, the monitor and diary must be returned to our clinic.

How are results interpreted?
The events recorded from the Holter monitor will be compared to your daily activity dairy as well as any symptoms you may have noted. The results of the test may reveal normal cardiac activity.

If your symptoms are so infrequent that a 24 hour monitor does not reveal any abnormal activity, an event monitor may be issued for the next 30 days. The events recorded in a 24 hour monitor may help reveal a present heart condition. Further management can be done following this.
Call our office:
If any of the following occur, be sure to call our office:
- Holter monitor makes a noise
- If an electrode comes off
Event Monitor
What is it?
A cardiac event monitor is a small portable device worn by patients during their normal activity for up to 30 days. This device is capable of recording the electrical activity of the heart in the form of an electrocardiogram (EKG) for up to several minutes. This is intended to record arrhythmias (abnormal heart rhythm) that would not be found on a routine EKG or to record an arrhythmia that is so infrequent, it is not detected on a 24 hour Holter monitor.
Who needs it?
- Patients with palpitations with or without arrhythmias
- Patients with symptoms of:
- Intermittent palpitations
- New syncope
- Dizziness
- Intermittent lightheadedness
- Patients requiring monitoring of the effects of drugs given to them to control atrial fibrillation
- Patients recovering from cardiac surgery who need arrhythmia monitoring
- Patients with obstructive sleep apnea who may have possible arrhythmias
Can anyone use it?
An event monitor is not intended for patients with life-threatening arrhythmias.
How does it work?
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This will be given to you up to a period of 30 days. If you feel any symptoms, push the button on the monitor to begin recording. This will allow Dr. Jamnadas to know what was happening with your heart at that point of time.
What are the benefits?
- This is capable of detecting asymptomatic events and transmitting them immediately to Dr. Jamnadas.
- Useful in recording arrhythmias that would not be found on a routine EKG or a 24 hour Holter monitor.
- The extended memory allows Dr. Jamnadas to examine the hearts electrical activity over an entire day.
- Useful in diagnosis of previously unrecognized arrhythmias and cause of symptoms.
- Allows confirmation or exclusion of a probable arrhythmic cause of symptoms.
- Useful in monitoring anti-arrhythmic medication.
Diagnostic Imaging
- Stress Test
- Nuclear Stress Test
- Echocardiogram
- Transesophageal echocardiogram
- Arterial Doppler
- Venous Doppler
Stress Test
What is it?
A stress test records the electrical activity of the heart during the stress of exercise. This test may show areas of low blood flow and damaged tissue.
Why do I need a stress test?
If the doctor suspects you have coronary artery disease or if you are experiencing symptoms of chest pain or sudden loss of breath.
To asses the amount of a possible blockage within the vessels of the heart.
A stress test may be advised to you even if you have already been diagnosed with coronary artery disease to asses the effectiveness of the treatment you are on.
An accurate stress test Decreases the use of unnecessary stenting.
How do I prepare for the test?
Wear sneakers and comfortable clothes (preferably a button down shirt with no metal buttons or zipper).
Avoid wearing long chains and necklaces.
Continue your medications up to the day of the procedure.
Stop this medication on the day of the test-
- Beta blockers including: Atenolol, Toprol, Metoprolol, Lopressor, Coreg, Zebeta, Betapace, or Corgard
If you have asthma and use an inhaler, bring it with you to your test.
Do Not smoke on the day of the test… or ever!!!
Do not apply body lotion on the day of the test
If you need to cancel your appointment, please do so at least 24 hours prior to your test or you may be charged.
What should I expect during the test?
A trained technician will place electrodes on your chest, arms, and legs. These electrodes connect to wires on an electrocardiogram (EKG) machine. The EKG records the electrical activity of the heart.
Next, you will slowly begin walking on the treadmill. As the test progresses, the speed and incline on the treadmill increases. Your blood pressure and EKG will be monitored throughout the test.
The length of the test depends on your physical fitness and symptoms. The goal is to have your heart working hard for about 5- 15 minutes in order to thoroughly monitor its function. Exercise is continued until you reach your target heart rate. 
During the test you may develop symptoms or signs of:
- Chest pain
- Shortness of breath
- High or low blood pressure
- Abnormal heart rhythm
- Dizziness
You may stop the test at any time if uncomfortable. Symptoms typically disappear once you rest.
The test lasts about 30 minutes.
What are the risks?
Stress tests are generally safe and complications are very rare. Although with any medical procedure, it does carry a small risk of complication.
These complications include:
- Hypotension: blood pressure may fall during or after exercise that can cause dizziness. This will usually ease off once you stop exercising
- Abnormal heart rhythms may occur during exercise. This usually returns to normal once exercise is ceased.
- Chest pain or flushing feeling may occur
The result:
Once the test is completed, the results of your test will be assessed to determine the condition of your heart.
This is useful in planning for your treatment and also helps in determining whether further testing is needed.
A stress test may reveal:
- The results may be beneficial in confirming or ruling out the diagnosis of heart disease
- Confirm whether you are in a stable state
- Assess the development of new blockage
Updated February 29th, 2012 -JS
For further information please contact Cardiovascular Interventions, PA at 407-894-4880
Nuclear Stress Test
What is it?
A nuclear stress test measures the blood flow to your heart both at rest and during stress on the heart during exercise.
This test provides images of the amount of blood flow in all sections of the heart, which can show areas of low blood flow and damaged tissue.
Why do I need it?
-
If the doctor suspects you have coronary artery disease
-
Experiencing symptoms of chest pain or sudden loss of breath
-
To asses the amount of possible blockage within the vessels of the heart
-
To asses the effectiveness of the treatment you are on.
-
To assess the size and shape of your heart. It may be enlarged in patients whose hearts are working harder than normal.
-
To measure the Ejection Fraction this shows how well your heart pumps with each beat.
-
An accurate stress test Decreases the use of unnecessary stenting
How do I prepare for a test?
-
Do not eat or drink 4 hours prior to the test. Small sips of water are permitted.
-
Avoid all caffeinated products for 12 hours before the test. This includes decaffeinated coffee, coffee, tea, soda. Avoid fatty foods as well as dairy products including butter, milk, and cheese. Fatty foods can alter the imaging results.
-
Bring a light breakfast or lunch sandwich and non-caffeinated drink with you.
-
Wear sneakers and comfortable clothes (preferably a button down shirt with no metal buttons or zipper). Avoid wearing long chains and necklaces.
-
If you are taking heart medications:
Continue your medications up to the day of the procedure. On the day of the test,
Stop these drugs:
Beta blockers (Atenolol, Toprol, Metoprolol, Lopressor, Coreg, Zebeta, Betapace, or Corgard)
Persantine (Dipyridamole)
Theophylline
Primatene/ Theo-dur
-
If you have diabetes and are taking insulin to control your blood sugar:
Ask your doctor how much insulin you should take the day of the test. Your doctor may recommend you to only take half of the usual morning dose and to eat a light meal 4 hours before the test.
Do NOT take your diabetes medication and skip a meal before the test.
- If you have asthma and use an inhaler, bring it with you to your test
- Do Not smoke on the day of the test… or ever!!!
- Do not apply body lotion on the day of the test
- If you need to cancel your appointment please do so at least 24 hours prior to your test or you may be charged.
What should I expect during the test?
First, a nuclear medicine technologist will place an I.V. into your arm or hand.
You may be injected with an isotope called Cardiolite that allows us to pick up your blood flow on camera.
After this is injected, you will wait about 30 minutes before the first set of “Resting” images are taken.
You will be asked to lie under a gamma camera with both arms above your head for 15-20 minutes. This camera will capture the images of the blood flow through your heart at rest.
Next, the technician will place electrodes on your chest, arms, and legs. These electrodes connect to wires on an electrocardiogram (EKG) machine. The EKG records the electrical activity of the heart.
If a pharmacological stress test has been ordered for you, you will then be injected with a pharmacological medication known as Lexiscan. This medication increases the blood flow to the heart to simulate exercise. This does NOT increase your heart rate, only dilates the vessels as if you were exercising.
Your blood pressure and EKG will be monitored throughout the test.
During the test you may develop symptoms or signs of:
- Chest pain
- Shortness of breath
- High or low blood pressure
- Abnormal heart rhythm
- Dizziness
Finally, after a 30-60 minute breakfast or lunch break you will be asked to again lie under the camera with both arms above your head for 15-20 minutes. The camera will capture the “exercise” images of the blood flow through your heart.
This set of images will be compared to the resting images.
The entire test will take about 3-4 hours.
Once your nuclear stress test is complete, you may return to your normal activities, eating habits, and medications.
What are the risks?
Nuclear stress tests are generally safe and complications are very rare. Although with any medical procedure, it does carry a small risk of complication.
These complications include:
- Infection at the site of intravenous puncture.
- Allergic reaction although uncommon, may be due to the radioisotope injected.
- Hypotension: blood pressure may fall during or after exercise that can cause dizziness.
- Abnormal heart rhythms may occur during exercise. This returns to normal once exercise is ceased.
- Chest pain or flushing feeling may occur from the medication or if you have coronary artery disease.
- As with all radiologic procedures, it is important to inform your physician and technologist if you are pregnant. Radiation exposure during pregnancy should be kept to a minimum.
The Result:
Dr. Jamnadas or Dr. Kelly will review the results of your nuclear stress test before your next office visit, not the same day as the test.
The results of the test will compare the images of the heart at rest and during exercise.

Interpretations of the results:
- Normal blood flow both at rest and exercise- it is unlikely you have coronary artery disease. No further work up is needed.
- Normal blood flow at rest and an abnormal blood flow during exercise- means part of your heart muscle is Not receiving adequate blood when you are exercising or during strenuous activity. You may have coronary artery disease; a blocked artery. Alteration of medication may be needed.
- Low blood flow at rest and exercise – means a significant amount of your heart is not getting enough blood flow. You may have had a previous heart attack or severe coronary artery disease. Further cardiac evaluation may be needed using a cardiac catheterization and coronary angiography.
In patients with mild to moderate blockage blood flow is not restricted. Usually only coronary blockages of >70% restrict blood flow and will usually show up on a nuclear stress test.
A nuclear stress test showing mild to moderate blockages usually do not show up on a nuclear stress test and do Not need stenting or bypass. This can be cured by lifestyle and preventative changes.
Updated March 2nd, 2012 -JS
For further information please contact Cardiovascular Interventions, PA at 407-894-4880
Echocardiogram
What is it?
An echocardiogram (ECHO) is a simple diagnostic study used to assess the condition and function of the heart using high frequency waves.
This study is performed here in our office.

When is it used?
An ECHO may show signs of:
- Congenital heart defects
- Previous heart attacks
- Blood clots in the heart
- Infection of the heart valves
- May explain chest discomfort or shortness of breath
- May explain the cause on an irregular heartbeat or heart sound
- Determines the overall strength of contractions of the heart
- Pericardial effusion (fluid around the heart)
What is the risk?
There is no risk associated with this study.
No radiation exposure is used during this study.
How do I prepare for the study?
No specific preparation is required. You may follow your daily eating habits before and after the study.
Clothing and jewelry need to be removed from the waist up to perform the study.
What happens during the study?
You will lie on a table while a trained ultrasound technician applies a clear jelly over the desired area. A probe is used to glide over this jelly which helps in transmission of sound waves through the skin.
The probe is pressed firmly over the chest to view the heart. Sound waves produced by the heart, create an echo that can be formed into an image. These images are recorded on to a monitor to be interpreted.
You may be asked to momentarily hold your breath or turn into a different position to enhance the images of the heart.
Sound waves given off the blood help to assess the direction of blood flow and blood pressures in the arteries of the heart. This also detects any abnormal sounds that may be produced by the heart.
The study may last up to 30 minutes.
What happens after the study?
After the study is over, the jelly is wiped off and you may return to your daily activities.
The result:
Dr. Jamnadas or Dr. Kelly will review the results of your study to look for healthy heart valves and chambers, as well as normal heart functioning.
This is useful in planning for your treatment and also helps in determining whether further testing is needed.
An ECHO may reveal:
- The size of the heart and the chambers of the heart
- The thickness and motion of the ventricular walls
- The movement and function of the of the heart valves
- The cardiac output (volume of blood pumped out of the heart per minute)
- The pressures within various chambers of the heart and major blood vessels in the heart
- The blood flow within the heart
- The heart function changes over time
- The presence of blood clots inside the heart
The results of your ECHO will be discussed with you during your office visit.
Updated February 28th, 2012 -JS
For further information please contact Cardiovascular Interventions, PA at 407-894-4880
Transesophageal Echocardiogram
What is it?
A transesophageal echocardiogram (TEE) is a diagnostic procedure used to asses the condition and function of the heart using high frequency waves.
This is done in a hospital setting where images of your heart are recorded by introducing an ultrasound transducer into the esophagus.

Why do I need this?
A TEE may have been ordered for you to obtain a more detailed image of your heart compared to a transthoracic echocardiogram done over the chest.
The transmission of beams to the heart may be difficult to reach in patients with over-crowding of the ribs, obesity, or emphysema.
In a transesophageal approach, the esophagus sits directly behind the heart without having to pass through any barriers.
When is it used?
A transesophageal echocardiogram may show signs of:
- Congenital heart defects
- Previous heart attacks
- Blood clots in the heart
- Infection of the heart valves
- May explain chest pain discomfort or shortness of breath
- May explain the cause on an irregular heartbeat or heart sound
- Determines the overall strength of contractions of the heart
- Pericardial effusion (fluid around the heart)
- Presence of a cardiac mass
- Determines whether cardioversion is required
- Detects the presence of an ascending aorta dissection or aneurysm
What are the risks?
TEE is a common diagnostic procedure, although with any medical procedure it carries risks.
These include:
- Allergic reaction: to the medication or latex
- Infection
- Dental injury
- Pharyngeal/ laryngeal trauma
- Esophageal trauma or bleeding
- Arrhythmia (irregular heart beat)
- Respiratory distress (breathing difficulty)
Notify the physician if you have known esophageal problems (i.e. difficulty in swallowing) or have undergone any radiation to the esophagus.
Let your physician know if you have any bleeding disorders.
If you are pregnant or suspect you may be pregnant, let the physician know.
If you are allergic to any medications or to latex notify the physician.
How do I prepare for this procedure?
The possible risks and benefits will be explained to you and any questions you or your family may have will be answered before your procedure.
You will be asked to have blood work done some time before the procedure.
You will be asked to sign a consent form to perform the diagnostic procedure.
Remove nail polish, necklaces, rings, bracelets, and any other jewelry before the procedure.
Do not eat or drink anything from midnight the night before your procedure. You may have small sips of water to take your medicine. The procedure is usually done in the morning. This is done to reduce the risk of vomiting and aspiration during the procedure.
A sedative will be given to you prior to the procedure to help you relax.
Make arrangements to have someone drive you to and from the hospital. You will not be permitted to drive after the procedure.
You will be asked to empty your bladder before the procedure.
The procedure is done on an outpatient basis so you will leave the same day as the procedure.
What happens during the procedure?
The procedure will take place in the hospital in the ECHO lab.
You will be asked to change into a hospital gown before entering the lab.
An I.V. line will be started in your hand or arm for the administration of a sedative.
You will lie on your back on a table and a pillow may be wedged underneath your back.
A local anesthetic spray will be applied to the back of your throat. This will make the back of your throat numb so you will feel more comfortable while the probe is being passed down your throat.
If required, oxygen may be administered through nasal tubes.
Then, you will be asked to lie on your left side.
The physician gently inserts a well lubricated probe into your mouth. You may be asked to swallow to help pass the probe down the esophagus.
Once the probe is in position, images of the heart can be obtained. This does not hurt or interfere with your breathing.
Throughout the procedure your heart rate, blood pressure, and oxygen will be closely monitored.
The procedure usually lasts 30 minutes, but the prep time may take longer.
What happens after the procedure?
Once the procedure is completed, you will be moved into a recovery area where your heart rate, blood pressure, and oxygen levels will be monitored.
Wait at least one hour after the procedure or until your throat is no longer numb to eat and drink again.
You may resume your daily activities after the procedure is completed.
No driving permitted for 24 hours.
Be sure to keep to your follow up appointment.
The result:
The results of your study will be reviewed to look for healthy heart valves and chambers, as well as normal heart functioning.
This is useful in planning for your treatment and also helps in determining whether further testing is needed.
A transesophageal echocardiogram may reveal:
- The size of the heart and the chambers of the heart
- The thickness and motion of the ventricular walls
- The movement and function of the of the heart valves
- The cardiac output (volume of blood pumped out of the heart per minute)
- The pressures within various chambers of the heart and major blood vessels in the heart
- The blood flow within the heart
- The heart function changes over time
- The presence of blood clots inside the heart
The results of your ECHO will be discussed with you during your office visit.
Updated February 29th, 2012 -JS
For further information please contact Cardiovascular Interventions, PA at 407-894-4880
Arterial Doppler
What is it?
An arterial Doppler study is a noninvasive, painless study used to assess the blood flow and pressure within the arteries using high frequency sound waves.
Why do I need it?
An arterial Doppler may be used to diagnose a variety of conditions including:
- Detecting the presence of blood clots
- Arterial occlusion
- Decreased blood flow to the arms or legs due to peripheral vascular disease
- Aneurysm
- Vascular tumor
What is the risk?
No risks are associated with this study.
No radiation exposure is used during this study.
How do I prepare for the study?
No specific preparation is required. You may follow your daily eating habits before and after the study.
Clothing will need to be removed over the area being examined.
What happens during the study?
You will lie on a table while a trained ultrasound technician applies a clear jelly over the desired area. A probe is used to glide over this jelly which helps in transmission of sound waves through the skin.
Two blood pressure cuffs are applied over the limb being studied. Each cuff is inflated one at a time. Inflation is done to determine the pressure within the artery and to detect the moment blood flow is returned to the vessel.
Blood pressures can be measured at different levels to assess arterial narrowing or occlusion. These readings are used to determine the site and size of arterial narrowing.
The study lasts up to 1 hour.
What happens after the study?
After the study is over, the jelly is wiped off and you may return to your daily activities.

The result:
The results of the test will show either a normal or abnormal study.
A normal study shows normal blood flow and pressure.
An abnormal study shows reduced blood flow and high pressure.
This will help diagnose atherosclerotic (narrowing due to plaque) changes.
Updated February 28th, 2012 -JS
For further information please contact Cardiovascular Interventions, PA at 407-894-4880
Venous Doppler
What is it?
A venous Doppler study is a noninvasive study used to measure the direction of blood flow through a vein using high frequency waves.
This study is useful in detecting any abnormalities which may present within a vein.
Why do I need it?
A venous Doppler study may be used for the following reasons:
- To search for deep vein thrombosis (DVT)- a blood clot within the vein
- To determine narrowing within the vein
- To detect any congenital malformations or presence of any vascular tumor
- To determine the cause of long standing leg swelling
- As an guidance tool for the exact placement of a catheter into a vein
What is the risk?
There is no risk associated with this study.
No radiation exposure is used during this study.
How do I prepare for the study?
No specific preparation is required. You may follow your daily eating habits before and after the study.
Clothing will need to be removed over the area being examined. You may consider wearing shorts for the study, if not a gown will be provided.
What happens during the study?
You will lie on a table while a trained ultrasound technician applies a clear jelly over the desired area. A probe is used to glide over this jelly which helps in transmission of sound waves through the skin.
The probe is pressed firmly over the skin in various angles and locations tracing over the area of interest. The limb may be squeezed at various times to check for the flow of blood through the vein. These sound waves show up as images on a monitor to be recorded and interpreted.
The study may last up to 30 minutes.

What happens after the study?
After the study is over, the jelly is wiped off and you may return to your daily activities.
The result:
The results of the study will show as either normal or abnormal.
A normal study shows normal blood flow with no evidence of deep vein thrombosis.
An abnormal study shows reduced blood flow with or without deep vein thrombosis.
This study helps in coming to a conclusion to some of the symptoms you may be having, assess the patency of the vein, as well as detecting any other abnormal findings.
Updated February 28th, 2012 -JS
For further information please contact Cardiovascular Interventions, PA at 407-894-4880
Blood Pressure
- Home Blood Pressure Monitoring
- Hypertension
- White Coat Syndrome
- Resperate
Home Blood Pressure Monitoring
Home blood pressure monitoring is important in the diagnosis and management of high blood pressure (hypertension). Self monitoring helps monitor your blood pressure within a comfortable and familiar setting.
This allows your physician and you to know whether your blood pressure is under control or how well your blood pressure medication is working.
Home blood pressure monitoring helps in the early prevention of potential health complications.
Who needs to be doing this?
Home blood pressure monitoring is particularly useful in clinical situations including:
- Borderline hypertension
- Newly diagnosed hypertension
- Variable blood pressure readings in the office or wide blood pressure discrepancy between home and doctors office (white coat hypertension)
- Apparent drug resistance
- Hypotensive symptoms from medications
- Suspected autonomic dysfunction
- Episodic hypertension
- Prognostic data
Why do I need it?
Since blood pressure fluctuates throughout the day, multiple recordings need to be taken. Readings can vary throughout the day and can be triggered by emotions, diet, or medications. Multiple blood pressure readings give a more accurate range of what your actual blood pressure is oppose to your clinical office visits.
Blood pressure recording in a comfortable and familiar surrounding helps to eliminate emotional stress or anxiety some patients may have after walking into a doctor’s office.
Blood pressure monitoring can help in a personal self improvement for controlling blood pressure. Most patients become encouraged and better motivated about their own health with a home monitoring system.
Who should not be doing this?
For patients with an irregular heartbeat (arrhythmia), home blood pressure monitoring is not advised.
This is because home monitoring does not give accurate readings in these patients.
What is the risk?
There are no known risks associated with home blood pressure monitoring.
A temporary and brief squeezing or sensation of tingling and numbness may be felt during the inflation of the cuff.
How is it done?
For the diagnosis of hypertension, multiple readings should be taken at various times throughout a patient’s day in addition to regular monitoring at the physicians office.
Blood pressure should be measured twice daily, at the same time everyday. Blood pressure should be measured once in the morning about one hour after waking and before taking your medications or foods. An evening blood pressure can be taken before dinner and before your evening medications.
Avoid caffeine, tobacco, and food 30 minutes before recording your blood pressure. Make sure to be seated, comfortable, and relaxed before taking your blood pressure. Avoid speaking while taking your blood pressure.
The correct method to check blood pressure is to place the cuff over your Left arm about 2cms above the elbow. Place the lined marking on the cuff against the artery of the inner arm, on the inside area of the biceps muscle. The cuff should not be too tight or too loose. Two fingers should be able to pass beneath the cuff. Turn the machine on and the cuff will automatically inflate.

It is important to make a diary of your activities. Normal daily activities including exercise, eating, sleeping, and even when you take your daily medications must be noted. Record the time of day or any symptoms you may feel accurately in your diary.
Bring your diary with you to your next appointment so your physician can assess how well your blood pressure is controlled.

Interpretation:
Interpreting your blood pressure readings may be confusing. The top number, Systolic pressure is the pressure in the arteries during contraction of the heart when arteries are being filled with blood. The bottom number is the Diastolic pressure which measures the pressure within the arteries during the relaxation of the heart resulting in a lower value than the top number.
Normal blood pressure ranges up to 120/80, but blood pressure can rise and fall with exercise, rest, or emotions.
|
Systolic Pressure |
Diastolic Pressure |
Normal |
<120 |
<80 |
Prehypertension |
>120-139 |
80-89 |
Essential (primary) Hypertension |
140-159 |
90-99 |
Secondary Hypertension |
>160 |
>100 |
If the presence of hypertension has been made, the following information should be assessed:
- The presence and extent of target organ damage
- The cardiovascular risk status
- Rule out other possible and reversible causes of hypertension
What are the benefits?
Several benefits of home blood pressure monitoring include:
- Establishment of true vs. false hypertension (i.e. White coat hypertension)
- Assess the response to blood pressure medications
- Patient compliance improvement
- Possible reduction in health care costs
Updated March 5th, 2012 -JS
For further information please contact Cardiovascular Interventions, PA at 407-894-4880
Hypertension (High Blood Pressure)
What is high blood pressure?
High blood pressure is also known as hypertension. In this condition, the force of the blood pressing against your artery walls is high. High blood pressure is important to control because it can lead to serious health problems including heart attack, stroke, and heart failure.
Hypertension can be asymptomatic and therefore dangerous. High blood pressure is easily detected and should be routinely monitored.


What are the causes of high blood pressure?
There are 2 types of hypertension, each having different causes:
- Essential (primary) hypertension
Accounts for 85% of cases and influenced by diet, lifestyle, or family history of high blood pressure.
- Secondary hypertension
Accounts for 15% of cases as a sudden cause of high blood pressure due to involvement of other organs or medications including:
- Congenital defects
- Sleep Apnea
- Kidney diseases
- Adrenal gland tumors
- Thyroid disorders
- Birth control pills, pain relievers, and cold remedies.
What are the risk factors?
-
Sex- Men are more likely to develop hypertension. Women are more likely to develop high blood pressure after menopause.
-
Old age
-
Race- more common amongst African Americans than Caucasians.
-
Family history of high blood pressure

-
Smoking
-
Alcohol
-
Being overweight or obese
-
Lack of physical activity
-
Too much salt in your diet
-
Stress
-
Chronic conditions may increase the risk of high blood pressure including high cholesterol levels, diabetes, adrenal and thyroid disorders, and sleep apnea.
What are the symptoms?
Some people may have NO signs or symptoms and can be surprised when diagnosed.
Symptoms include:
- Headache
- Dizziness
- Blurring of vision
- Nausea and vomiting
- Chest pain
- Shortness of breath
How do I know if I have high blood pressure?
Blood pressure is recorded at your routine doctor’s visit. If you are not diagnosed with hypertension, check your blood pressure at least twice a year. For patients already diagnosed with high blood pressure should check their blood pressure more frequently. Blood pressure does not only have to be recorded at your doctor’s appointment. You can check at any local grocery store or at home if you have your own blood pressure monitor. Take your blood pressure when you are calm and relaxed. Home blood pressure monitors are a great way to get accurate readings while you are at rest. 24 hour blood pressure recording is better than office recordings.
The correct method to check blood pressure is to place the cuff over your Left arm about 2cms above the elbow. Not too tight or too loose, you should be able to pass 2 fingers in the cuff.
Once you have recorded your blood pressure you will be left with 2 numbers. The top number, Systolic pressure is the pressure in the arteries during contraction of the heart when arteries are being filled with blood. The bottom number is the Diastolic pressure which measures the pressure within the arteries during the relaxation of the heart resulting in a lower value than the top number. Normal blood pressure ranges up to 120/80, but blood pressure can rise and fall with exercise, rest, or emotions.
|
Systolic Pressure |
Diastolic Pressure |
Normal |
<120 |
<80 |
Prehypertension |
>120-139 |
80-89 |
Essential (primary) Hypertension |
140-159 |
90-99 |
Secondary Hypertension |
>160 |
>100 |
What happens in my blood pressure is not controlled?
You are at risk of having:
- Heart attack
- Atherosclerosis i.e.: hardening of the arteries
- Angina i.e.: chest pain
- Stroke
- Congestive Heart failure, weak heart muscle
- Peripheral vascular disease
- Kidney failure- need for renal dialysis
- Aortic aneurysm
- Vascular dementia
- Blindness
How can I prevent this?
-
Check your blood pressure routinely.

-
Diet modification: cut back on salty and buttery foods. Be sure to eat plenty of fruits, vegetables, and foods rich in potassium.
-
Certain supplements can help lower cholesterol including Cod liver oil, Coenzyme Q10, and Omega 3 fatty acids.
-
Follow a diet. Try using the “No Whites Diet”, “DASH diet”, or the “Paleolithic diet”. Ask Dr. Jamnadas about which diet plan is best for you.
-
Maintain a healthy weight with a body mass index <25. This will minimize the pressure in your arteries.
-
Get a comprehensive nutritional evaluation. Certain elements and vitamin deficiencies can cause high blood pressure. I.e. Vitamin D deficiency affects an enzyme produced by your kidneys that affects blood pressure.
-
A full endocrine evaluation may be recommended to rule out secondary causes of hypertension.
-
Be more physically active. Try going for a walk, jog, bike ride, or swim.
-
Quit smoking
-
Limit alcohol: if you choose to drink alcohol, limit it to no more than 2 drinks a day.
-
Keep stress levels to a minimum.
Treatment of high blood pressure:

Diet modification, weight loss, and active lifestyle all help in lowering blood pressure.
Medical treatment varies in each patient.
Dr. Jamnadas will prescribe a single low dose medication to lower the blood pressure first.
If not controlled with just one, a combination of 2 or more medications may be advised for best results.
The category of medication Dr. Jamnadas prescribes depends on the stage of high blood pressure and whether you have other medical problems.
Blood pressure lowing drugs include:
-
Diuretics- sometimes referred to as “water pills”. These reduce sodium and water to lower blood volume.
- Beta Blockers- act by lowering the heart rate to decrease the work load of the heart and help dilate the blood vessels.
- ACE Inhibitors- lower blood pressure by preventing some of your natural chemicals to be made that constrict blood vessels and promote dilatation.
- Angiotensin II Receptor Blockers (ARBs) - block the action of the natural chemicals that narrow blood vessels.
- Calcium Channel Blockers- relax the muscles within the blood vessels by selectively blocking excess calcium and slow the heart rate. This increases the oxygen supply to the heart, increases blood flow, lowers resistance within the vessel, and prevents coronary artery spasm.
- Vasodilators- directly act on the muscles within the walls of the blood vessels, promoting dilatation and decreases blood pressure.
Once your blood pressure is under control, Dr. Jamnadas may have you take a daily aspirin to reduce your risk of cardiovascular disorders.
*RESPeRATE: Resperate is a portable FDA approved device that lowers your blood pressure by altering your breathing patterns. This uses chest sensors to measure breathing to synchronize you to a breathing pattern with longer expirations. This is recommended to be used for 15 minutes a day, 3- 4 days a week. A reduction in stress levels will be noticed. This device is available for purchase in our office.
Don’t Forget!
Schedule routine visits to Dr. Jamnadas to monitor your blood pressure.
Take your medications regularly, exactly as prescribed.
If you are having any side effects or the costs of medications are posing problems please do NOT discontinue your medications on your own. Ask Dr. Jamnadas about other options.
Ensure good nutrition and get regular exercise.
Quit smoking
Manage stress: stay positive, optimistic, and most of all calm.
*The single most important parameter to treat in your life is your blood pressure*
Updated January 31st, 2012 -JS
For further information please contact Cardiovascular Interventions, PA at 407-894-4880
White Coat Syndrome
What is it?
White coat syndrome (WCS) is a temporary elevation of blood pressure caused by excitement or anxiety that does not necessarily constitute hypertensive disease, but may indicate an inclination towards its development.
White coat syndrome occurs in a patient whose blood pressure is consistently higher at a physician’s office than when measured at home. Patients may or may not have an existing diagnosis of hypertension.
Patients with white coat syndrome have an increased risk of cardiovascular disease, although the risk is greater in a true hypertensive patient.
Ambulatory 24-hour blood pressure monitoring is useful in the evaluation of these patients.

What are the causes?
The causes of white coat syndrome are usually always caused by:
- Fear
- Dread
- Worry
- Excitement
- Anxiety
These emotions stimulate the fright or flight response.
This stimulates hormones (i.e. adrenaline) and the nervous system to constrict the blood vessels causing a temporary increase in blood pressure.
What are the symptoms?
There are no symptoms of white coat syndrome.
Patients are generally asymptomatic.
How is it diagnosed?
The goal in diagnosis is to avoid unnecessary drug therapy, provide a better diagnostic and prognostic assessment, and reduce costs.
A good way to overcome white coat syndrome is by multiple office blood pressure readings done in a routine fashion, to minimize anxiety.
Blood pressure may also be recorded outside the physician’s office at home or at a local grocery store. This is referred to as blood pressure self-monitoring.

The best way to diagnose white coat syndrome is with a 24 hour ambulatory blood pressure monitor, which is provided here at CVI. This is a small portable monitor that takes regular readings of your blood pressure over the day and night. A 24 hour ambulatory blood pressure monitor allows the physician to assess blood pressure under the conditions of a typical day. Blood pressure will also be monitored during sleep. Obtaining recordings during sleep is beneficial as normal blood pressure decreases or “dips” during sleep. With the lack of this “dip” during sleep, the diagnosis of true hypertension can be clarified.
If you have been diagnosed with white coat syndrome, the risk of developing heart disease is greater and many patients go on to develop hypertension (high blood pressure) in the future.
It is essential to have your blood pressure recorded on a regular basis to ensure that if blood pressure escalates, action can be taken promptly to bring blood pressure down and minimize the risk of heart disease.
Lifestyle modifications are reinforced in patients with white coat syndrome.
How is it prevented?
Lifestyle modifications are highly recommended in patients with white coat syndrome.
This includes:
- Quit smoking
- Limiting alcohol
- Controlling cholesterol and diabetes
- Eating a healthy diet
- Reducing your salt intake
- Keep physically active with the right kind of exercise
- Maintain a healthy weight with a BMI <25
- Manage stress: stay positive, optimistic, and most of all calm
Updated April 20th, 2012 -JS
For further information please contact Cardiovascular Interventions, PA at 407-894-4880
RESPeRATE
What is it?
RESPeRATE is a portable FDA approved drug free device used to lower blood pressure by altering breathing patterns. RESPeRATE uses chest sensors to measure breathing to help synchronize you to a breathing pattern with longer expirations. RESPeRATE has been shown to reduce blood pressure, stress, and depression.
What are the indications?
RESPeRATE is used as a relaxation treatment for the reduction of stress using guided and monitored breathing exercises. The device is indicated for use only as an adjunctive treatment for high blood pressure, together with other pharmacological and/or non-pharmacological interventions.
Who benefits from RESPeRATE?
RESPeRATE has been shown to be particularly helpful in the following clinical situations:
- White coat syndrome or labile hypertensive patients who might benefit from reducing stress and sympathetic activity
- Patients with isolated systolic hypertension
- Resistant hypertensive patients (uncontrolled BP despite the use of a diuretic and at least 2 other medications at maximum dosage).
How do I use it?
- Place respiration sensor belt around your abdomen or upper chest.
- Resperate analyses your breathing pattern to create a personalized guiding melody.
- Synchronize your breathing with the melody tones. Inhale when tone goes up, exhale when tone goes down.
- Resperate guides you to slow breathing and reach the ‘therapeutic zone’ which is just under 10 breaths per minute.
How does it work?
Interactive slow breathing affects the neural system by relaxing the small blood vessels which results in improved blood circulation.
When do I use RESPeRATE?
Patients are instructed to use the RESPeRATE device routinely at rest, in 15 minute daily sessions, aiming to accumulate at least 45 minutes of slow breathing per week (10 breaths per minute or less). The weekly accumulated effective time (minutes of slow breathing) appears on the device display momentarily each time the device is turned on. Patients should be made aware that results, just like physical conditioning, may take a few weeks to become fully manifested and that without continued device use, any achieved benefits would likely be diminished. Studies show that the bulk of the reduction in BP occurs within 4 to 6 weeks of routine use.
Procedures
- NIPS
- Tab 2
Content
Prevention
- Glycemic Index
- Reading Food Labels
- Diabetes and Eating Healthy
- Nutrition and Heart Disease
- Paleolithic Diet
- Vitamin D
- "No Whites" Diet
- The TV Method for Exercise
- The Importance of Physical Fitness
- Nature's Superfoods
- Fish Oil
- SpectraCell Analysis Test
- Alcohol
- Smoking
Glycemic Index
What is it?
The glycemic index (GI) is measure of the ability of foods (specifically carbohydrates in food) to raise your blood sugar (glucose) levels after being eaten.
The GI scale ranks carbohydrates from 0 to 100, with 0 as the slowest absorption and 100 as the fastest absorption into the bloodstream.
Foods with a high GI (above 70) are quickly digested and absorbed, resulting in a rapid increase in blood sugar and, consequently, insulin levels.
Foods with low GI (less than 55) are slowly digested and absorbed, meaning they gradually increase blood sugar and insulin levels.

How is it measured?
Glycemic index measures the amount of carbohydrates in food and the glucose levels after eating.
Different carbohydrate containing foods affect blood glucose levels differently.
Food items are charted as high, medium, and low according to the GI value measured.
-
High glycemic index is above 70 (i.e. white bread)
- Medium glycemic index is 50 to 70 (i.e. brown bread)
-
Low glycemic index is below 50 (i.e. apple)
-
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Who should be using this?
By watching what you eat, your hunger and energy levels will have a beneficial affect.
Your total blood glucose, cholesterol, and triglyceride levels will significantly improve.
Those who may benefit from using the glycemic index include:
- Those who want to lose weight
- Control high glucose levels (i.e. diabetics)
- Control low glucose levels
- Control cholesterol and triglyceride levels
- Patients who want to decrease the risk of cardiovascular disease
- Patients who want to decrease the risk of diabetes
- Patients who want to decrease the risk of kidney disease
- Patients who want to decrease the risk of age-related macular degeneration
What guidelines should be followed?
Glycemic index depends on a variety of factors such as the presence of starch, protein, fat, fiber, and organic acids in food.
Elevated glucose in the blood stream produces elevated levels of insulin in the body. Constant elevation of glucose may exhaust insulin levels that can lead to diabetes, weight gain, and inflammation.
Some ways to avoid this include:
- Eat more low GI foods like beans, whole grains, and pasta regularly but in moderate quantities. Limit high GI foods such as bagels or white bread to small quantities.
- Substitute snacks like chips for vegetables and side dishes like rice for beans.
- Eat smaller and more frequent meals. Have a small snack prepared between breakfast and lunch as well as a small snack between lunch and dinner.
- Have a moderate sized lunch and a smaller portion for dinner.
- Cook pasta until till they are crisp or al dente style.
- Limit high GI foods like sweets to very small quantities and rare occasion as these have large amounts of sugar.
- Include plenty of fruits and vegetables in your meals, while limiting potatoes.
- Add a small amount of vinegar to rice to lower the GI.
- Eat foods high in fiber as they slow gastric emptying and have a low GI.
- If you choose to have an alcoholic beverage, limit drinking to one glass prior to a meal to reduce the GI.

- Look for the glycemic index symbol while shopping for groceries.
Low Glycemic Index food (GI < 50)
FOOD ITEM |
GI |
FOOD ITEM |
GI |
|
Yogurt low-fat (sweetened) |
14 |
Rye |
34 |
|
Peanuts |
15 |
Milk, semi-skimmed |
34 |
|
Artichoke |
15 |
Vermicelli |
35 |
|
Asparagus |
15 |
Spaghetti, whole wheat |
37 |
|
Broccoli |
15 |
Apples |
38 |
|
Cauliflower |
15 |
Pears |
38 |
|
Celery |
15 |
Haricot beans, boiled |
38 |
|
Cucumber |
15 |
Plums |
39 |
|
Eggplant |
15 |
Ravioli, meat filled |
39 |
|
Green beans |
15 |
Carrots, cooked |
39 |
|
Lettuce, all varieties |
15 |
Snickers bar |
40 |
|
Low-fat yogurt, artificially sweetened |
15 |
Apple juice |
41 |
|
Peppers, all varieties |
15 |
Wheat kernels |
41 |
|
Snow peas |
15 |
Spaghetti, white |
41 |
|
Spinach |
15 |
Black-eyed beans |
41 |
|
Young summer squash |
15 |
All-Bran |
42 |
|
Tomatoes |
15 |
Peaches |
42 |
|
Zucchini |
15 |
Chickpeas, tinned |
42 |
|
Soya beans, boiled |
16 |
Oranges |
44 |
|
Cherries |
22 |
Lentil soup, tinned |
44 |
|
Peas, dried |
22 |
Carrot juice |
45 |
|
Milk, chocolate |
24 |
Macaroni |
45 |
|
Pearl barley |
25 |
Pineapple juice |
46 |
|
Grapefruit |
25 |
Rice, instant |
46 |
|
Milk, whole |
27 |
Grapes |
46 |
|
Spaghetti, protein enriched |
27 |
Grapefruit juice |
48 |
|
Kidney beans, boiled |
29 |
Multi grain bread |
48 |
|
Lentils green, boiled |
29 |
Rice, parboiled |
48 |
|
Soya milk |
30 |
Baked beans, tinned |
48 |
|
Apricots (dried) |
31 |
Porridge, non-instant |
49 |
|
Milk, Fat-free |
32 |
Chocolate bar; 30g |
49 |
|
Milk ,skimmed |
32 |
Jams and marmalades |
49 |
|
Fettuccine |
32 |
Whole grain |
50 |
|
M&Ms (peanut) |
32 |
Barley, cracked |
50 |
|
Chickpeas |
33 |
Ice-cream (low- fat) |
50 |
|
Moderate Glycemic Index Foods List (50 to 70)
FOOD ITEM |
GI |
FOOD ITEM |
GI |
Yam |
51 |
Hamburger bun |
61 |
Orange juice |
52 |
Ice-cream |
61 |
Kidney beans |
52 |
Potato, tinned |
61 |
Lentils beans |
52 |
Muffin (unsweetened) |
62 |
Kiwi fruit |
53 |
Shortbread |
64 |
Pound cake |
54 |
Rye-flour bread |
64 |
Banana |
54 |
Apricots (tinned in syrup) |
64 |
Sweet potato |
54 |
Raisins |
64 |
Crisps |
54 |
Macaroni cheese |
64 |
Oat bran |
55 |
Beetroot |
64 |
Rice, brown |
55 |
Mars bar |
64 |
Fruit cocktail |
55 |
Black bean soup, tinned |
64 |
Spaghetti, durum wheat |
55 |
Cake , tart |
65 |
Popcorn |
55 |
Water biscuits |
65 |
Muesli |
56 |
Potato, steamed |
65 |
Mangoes |
56 |
Table sugar (sucrose) |
65 |
Potato, boiled |
56 |
Barley, flakes |
66 |
Pita bread, white |
57 |
Pineapple |
66 |
Mini Wheats (whole meal) |
57 |
Green pea soup, tinned |
66 |
Rice, wild |
57 |
Cake, angel |
67 |
Apricots |
57 |
Croissant |
67 |
Potato, new |
57 |
Taco Shell |
68 |
Digestives |
58 |
Whole meal bread |
69 |
Rice, white |
58 |
Shredded Wheat |
69 |
Danish pastry |
59 |
Potato, mashed |
70 |
Pizza, cheese |
60 |
|
|
High Glycemic Index Foods List (GI > 70)
FOOD ITEM |
GI |
FOOD ITEM |
GI |
White bread |
71 |
Broad beans |
79 |
Golden Grahams |
71 |
Jelly beans |
80 |
Millet |
71 |
Pretzels |
81 |
Watermelon |
72 |
Rice Krispies |
82 |
White rolls |
73 |
Potato, micro waved |
82 |
Puffed wheat |
74 |
Cornflakes |
83 |
Corn chips |
74 |
Potato, instant |
83 |
Chips |
75 |
Potato, baked |
85 |
Waffles |
76 |
Rice pasta, brown |
92 |
Doughnut |
76 |
Baguette |
95 |
Wafer biscuits |
77 |
Parsnips |
97 |
Rice cakes |
77 |
Dates |
103 |
Weetabix |
77 |
|
|
Updated March 12th, 2012 -JS
For further information please contact Cardiovascular Interventions, PA at 407-894-4880
Definitions of Claims on Food Labels
The following definitions of nutrition labels will help you to understand exactly what is in the foods you buy:
Free - the product contains no or only a trivial amount of one or more of the following:
- fat
- saturated fat
- cholesterol
- sodium
- sugars
- calories
Low - this means a large quantity of the food can be eaten without exceeding the Recommended
Dietary Value for the nutrient
- Calorie free - fewer than 5 calories per serving
- Cholesterol free - less than 2 mg of cholesterol & 2 g or less of saturated fat per serving
- Fat free - less than 0.5 g per serving
- Sodium free - less than 5 mg of sodium per serving. Sugar free - less than 0.5 g per serving
- Low calorie - contains 40 calories or less in a serving
- Low cholesterol - 20 mg or less and 2 g or less of saturated fat per serving
- Low fat - contains 3 g or less per serving
- Low saturated fat - contains 1 g or less per serving
- Low sodium - contains 140 mg or less per serving
- Very low sodium - contains 35 mg or less per serving
- Lean - used in the description of the fat content of meat, poultry, seafood, and game meats. Less than 10 g fat, 4.5g or less saturated fat, and less than 95 mg cholesterol per serving for every 100 g of product
- Extra Lean - less than 5 g fat, less than 2 g saturated fat, and less than 95 mg cholesterol per serving for every 100 g of product
- Good source - this means that one serving has 10 to 19 percent of the RDA for a particular nutrient.
- High - used when food contains 20% or more of the RDA for a nutrient in one serving
- Light - this term can mean two things:
- A nutritionally altered product has 1/3 fewer calories or 1/2 the fat of the referenced food
- The sodium content of a low calorie, low-fat food has been decreased by 50%
- Less - a food, altered or not, contains 25% less of a nutrient or of calories than the referenced food.
- More - a serving contains a nutrient that is at least 10% of the Daily Value more than the referenced food. This term also applies to "fortified," "enriched," and "added," when the food has been altered.
- Percent fat free - the product with this claim must be low-fat or fat-free. It reflects the amount of fat in 100 grams of the product. For example: 95% fat free would have 5 g fat for every 100 g of product.
- Reduced - The nutritionally altered product has 25% less of a nutrient or of calories than the regular, or referenced, product.
Updated January 29, 2009 - MJ
Diabetes and Eating Healthy
If you have Diabetes, you do not need a special diet. You simply need to follow the same healthy eating plan that is recommended for everyone. The following information can help you plan delicious and nutritious meals to keep you healthy.
Eat small meals and snacks throughout the day
Consume regular meals and snacks so that your food intake is spread throughout the day; this will help control your blood sugars. It is important to start each day with a healthy breakfast. Do not skip meals. If you take tablets or insulin, regular meals and snacks can help you to avoid symptoms of low blood sugar.
Reduce your sugar intake by choosing low sugar foods and drinks
It is not necessary to avoid sugar completely. Aim to eat less sugary foods and drinks, such as candy bars, ice cream, cookies, sweet sodas, and fruit juices. These foods can make your sugar levels rise quickly, especially if eaten in large quantities. Try to choose low sugar foods and sugar free drinks when possible.
Eliminate fast foods & junk foods
Fast foods and junk foods are full of fat and calories and usually offer little or no nutritional value. A simple change in habits can help you eliminate these foods from your diet. If you are in the habit of driving through a fast food restaurant pick up window to grab a quick burger and fries, start taking your lunch to work or school. For snacks, avoid vending machines, which usually only offer foods high in fat and calories. Instead, have sliced carrots, an apple or low fat yogurt. Unfortunately, convenience often means sacrificing nutrition.
Include a helping of starchy food at each meal
Starchy foods are breads, cereals, rice, potatoes and pasta. Try to base your meals around these foods, as they will help to keep your blood sugar levels steady. Include high fiber varieties where possible.
Eat more fruits and vegetables
They are an important part of a healthy diet because of their vitamin and mineral content. Additionally, they are important in regulating bowel function because they increase the fiber content of your diet. For a snack or dessert, start having a piece of fruit and enjoy all varieties of vegetables, salads and fruits. They can be fresh, frozen, tinned, or present in natural juice. Unsweetened fruit juice should be kept to one small glass per day before or with a meal.
Reduce fried and fatty foods
Following a low fat diet makes it easier to keep a healthy weight and a healthy heart. Cut down on butters and avoid frying foods altogether. Adopt other cooking methods such as grilling or baking. Choose lower fat varieties of milk and other dairy products such as cheese and yogurts. Consume only small portions of meats, poultry, and fish. Always cut off any visible fat.
Alcohol
Moderate consumption of alcohol is normally safe. But remember, alcohol can lower your blood sugar. Never drink alcohol on an empty stomach. Be sure to check with your doctor if you have any concerns or if you are unsure whether alcohol is safe for you.
Updated January 29, 2009 - MJ
Nutrition and Heart Disease
The Basics
Vitamin and mineral supplements combined with an unrefined diet high in fruits and vegetables, some omega-3 oils, no hydrogenated fats, and a good lifestyle are the basics for a healthy heart. Include weight control, not smoking, some exercise, and the ability to manage stress, and you will improve your general health and prevent or help control heart disease.
A Healthy Diet
Science shows that a varied diet of relatively unrefined foods with many fruits, vegetables, brown rice and whole grains is the basics of a healthy diet. Unfortunately, these foods are becoming scarce in many Western diets. When the food industry processes these foods, at least 75% of the minerals, vitamins, fiber, and antioxidant nutrients is lost, and this contributes to the development of chronic diseases.
Most of the world's heart and cardiology organizations suggest reducing saturated (solid) fat and cholesterol intake while increasing polyunsaturated fat consumption. Although less fat is generally good, the only clear benefits of fat substitutions are those with omega-3. Polyunsaturates without omega-3 (all the high omega-6 linoleic oils: corn, sunflower, safflower or cottonseed) can in fact cause harm, especially if they are partially hydrogenated (shortening and 90% of margarines). Virgin olive oil has no omega-3 but it's the healthy unprocessed oil for daily use. Butter is better than margarine except possibly unhydrogenated canola and soy based types.
Reduce sugar, white flour, white rice, ordinary white noodles and foods that are deep fried, have shortening and anything "hydrogenated".
Potassium (found in bananas, celery, potatoes, fruits & veggies) and magnesium (whole grains, nuts, greens) can help prevent heart attacks. Sweating and most diuretics flush out these spark plugs for the heart. It is recommended that you drink at least eight 8oz. glasses of water every day.
Vitamin & Mineral Supplements
Foods low in these "minor" nutrients can cause initially hidden illness, like heart disease, cancer, Alzheimer's, and arthritis. These are the slow-building diseases that drug-based medicine has not been able to prevent or effectively control.
Even when in great health, there's excellent reason for taking a good multi-vitamin plus foods and supplements so that your total daily intake reaches about 1 gram of vitamin C, 1.5 g calcium, 3/4 g magnesium, 200-400 IU vitamin E and 200 mcg selenium. It is also the easiest life-style change you can make.
Us a single multivitamin with most B's near the 25 mg level, B3 at 100 mg, folic acid at 400 mcg and B12 at 100 mcg. Separately take 200 IU natural (type d) or 400 IU synthetic vitamin E (type dl), about 1 gram vitamin C (not Ester-C) and take 200 mcg selenium.
It is important to get about 1½ g calcium, ¾ g magnesium (most people will need to supplement) and 4-800 IU vitamin D to make the calcium build bone.
Only take iron if you are in your child bearing years or have another reason. Go easy on the copper (1 - 2 mg max.) but do include 15 mg zinc. These plus the selenium and the D can be found in the same single multi. If you have a cholesterol problem, try to avoid the statin drugs by taking the niacin route (vitamin B3, about 3x1 g/day) and always consult a doctor. Generic niacin is 10-20 times cheaper, effective and unique in its action on blood fats, lowering all that's bad, raising all that's good, and more.
If you have a (congestive) heart condition or take a statin drug, consider at least 60 mg coenzyme Q10 (CoQ10).
Exercise
Everyone knows that regular physical activity has a direct link to weight loss, lowering cholesterol levels, fighting obesity, controlling heart disease and helps maintain general good health. Make it a part of your daily routine.
There is an excellent chance that with long-term use of these foods, supplements, omega-3's along with regular exercise, you will significantly reduce your risk of sudden heart death.
The Paleolithic Diet
INTRODUCTION:
Although we are living in the 21st century, our human genome is greater than 10,000 years old. The way we metabolize our food is largely determined by our genetic machinery. Yet today, we live in a mechanized urban setting, lead sedentary lifestyles, and eat highly processed and synthetic diets that our genome is not accustomed to. It therefore comes as no surprise, that cardiovascular disease is the number-one cause of death and accounts for 41% of all fatalities. It should also come as no surprise that metabolic syndrome is present in over 40% of middle-aged American adults and that the lifetime probability of hypertension is greater than 90%. This cardiovascular disease pandemic will continue until we realize that our bodies were not designed for this lifestyle and diet. The genes we are born with will remain unchanged; therefore, our diet and lifestyle need to change.
When our forefathers transitioned to an agricultural grain based diet, a gradual deterioration in health care began. Our ancestors consumed lean meats, fruits, and vegetables as opposed to cereals. The grain-based diet resulted in shorter life spans, higher childhood mortality, and a higher incidence of osteoporosis, rickets, and various other mineral and vitamin deficiency diseases. Further adaptation to Western lifestyles have lead to obesity, diabetes, atherosclerosis, and other degenerative diseases.
DIET:
There is so much controversy and confusion in recent times about the type of diet that we should follow. The Ornish diet consists of 80% carbohydrates with minimal consumption of animal fat or protein. The Atkins diet is high in protein and high in saturated fats and avoids almost all carbohydrates. Each diet has its benefits and disadvantages.
Over 150 studies about diet and cardiovascular health have demonstrated that the following may reduce cardiovascular events:
- Increasing consumption of omega-3 fats from fish or plant sources such as nuts
- Eating a diet high in fruits, vegetables, nuts, and whole grains, while avoiding food of high glycemic index
- Replacing saturated and trans-fats with more unsaturated and polyunsaturated fats. There is very little evidence suggesting a link between cardiovascular disease in the studies and the intake of total fat, cholesterol, or meat.
Our ancestors consumed only natural and unprocessed food from the environment that provided them with a diet of protein, fiber, vitamins, minerals, antioxidants, and other beneficial phytochemicals. Our ancestors’ diet had three times more fiber, twice as much polyunsaturated and monounsaturated fats, four times more omega-3 fatty acids, and 70% less saturated fat. Furthermore, their sodium intake was 1/5 of what it is today, and their diet did not contain refined grains and sugar. It is evident that the epidemic of cardiovascular diseases is at least in part due to the striking discrepancies between the diet we are designed to eat and what we eat today.
CALORIES:
In this modern world, calorie-dense foods are abundant and inexpensive; therefore, most people are dying of caloric excess, which manifests itself as hypertension, cardiovascular disease, metabolic syndrome, and obesity. We also expend much less energy than our ancestors did. A diet that is high in carbohydrates and low in fat will have a much lower satiety index than the diet that has adequate quantities of proteins and fats. Therefore, diets containing moderate amounts of beneficial fats and proteins, low glycemic index carbohydrates, and a regular exercise regimen are the most effective ways to maintain an ideal body weight and reduce cardiovascular disease risk.
GLYCEMIC INDEX OF CARBOHYDRATE CONCENTRATIONS PER 100g
|
Carbohydrate Concentrn |
Glycemic Index |
Aubergines |
4 |
10 |
Brocolli |
4 |
10 |
Cabbage |
4 |
10 |
Garlic |
28 |
10 |
Green Vegetables |
4 |
10 |
Lettuce |
4 |
10 |
Mushrooms |
4 |
10 |
Onions |
5 |
10 |
Red Peppers |
4 |
10 |
Tomatoes |
4 |
10 |
Walnuts |
5 |
15 |
Apricots (fresh) |
10 |
20 |
Fructose |
100 |
20 |
Grapefruit |
10 |
20 |
Peanuts |
9 |
20 |
Soya (cooked) |
15 |
20 |
Cherries |
17 |
22 |
Dark Chocolate (<70% cocoa solids) |
32 |
22 |
Lentils – Green |
17 |
22 |
Peas – Split |
22 |
22 |
Plums |
10 |
22 |
All Bran |
46 |
30 |
Apple |
12 |
30 |
Beans – French |
3 |
30 |
Beans – Haricot |
17 |
30 |
Chick Peas (cooked) |
22 |
30 |
Fruit Preserve (w/out sugar or grape juice) |
37 |
30 |
Lentils – Brown |
17 |
30 |
Milk (semi-skimmed0 |
5 |
30 |
Peach |
9 |
30 |
Apricots (dried) |
63 |
35 |
Carrots (raw) |
7 |
35 |
Chinese Vermicelli (mungo bean) |
15 |
35 |
Fig (fresh) |
12 |
35 |
Ice Cream (made w/ alginates) |
25 |
35 |
Maize/Corn on the cob (traditional variety) |
21 |
35 |
Orange |
9 |
35 |
Pear |
12 |
35 |
Peas - Dried (cooked) |
7 |
35 |
Quinoa (cooked) |
7 |
35 |
Yogurt (full-milk) |
4.5 |
35 |
Yogurt (skimmed) |
5.3 |
35 |
Apple Juice (fresh) |
17 |
40 |
Black Bread (German) |
45 |
40 |
Flour T200 (unrefined) - Bread |
45 |
40 |
Flour T200 (unrefined) - Pasta |
17 |
40 |
Grapes |
16 |
40 |
Kidney Beans |
11 |
40 |
Orange Juice (freshly pressed) |
10 |
40 |
Peas (Fresh Petis Pois) |
10 |
40 |
Rye (wholegrain, cooked) |
49 |
40 |
Boulgour (wholegrain, cooked) |
25 |
45 |
Bran Bread |
40 |
45 |
Flour T1550 (unrefined) - Pasta |
19 |
45 |
Spaghetti (hardgrain, cook al dente) |
25 |
45 |
Buckwheat (black wheat flour0 |
65 |
50 |
Crepe/Pancake (made w/ buckwheat) |
25 |
50 |
Flour T150 (unrefined) - wholemeal bread |
47 |
50 |
Kiwi |
12 |
50 |
Rice (Basmati) |
23 |
50 |
Rice (Brown) |
23 |
50 |
Sorbet |
30 |
50 |
Sweet Potato |
20 |
50 |
Petite Beurre Biscuit |
75 |
55 |
Shortbread Biscuit (Flour B) |
68 |
55 |
White Pasta (normal cooking) |
23 |
55 |
Rice (long grain, white) |
23 |
60 |
Banana |
20 |
65 |
Brown Flour T85 (brown bread) |
50 |
65 |
Jam (traditional) |
70 |
65 |
Melon |
6 |
65 |
Orange Juice (industrial) |
11 |
65 |
Potatoes (boiled in their skins) |
14 |
65 |
Raisins |
66 |
65 |
Semolina (refined) |
25 |
65 |
Cereals (sugared) |
80 |
70 |
Chocolate Bars (ex. Mars Bar) |
60 |
70 |
Cola Drinks |
11 |
70 |
Cornflour |
88 |
70 |
Flour T65 - country style bread |
53 |
70 |
Maize/Corn on the cob (modern variety) |
22 |
70 |
Noodles, Ravioli |
23 |
70 |
Potato (peeled and boiled) |
20 |
70 |
Rice (pre-cooked and non-stick) |
24 |
70 |
Sugar |
100 |
70 |
Turnip |
3 |
70 |
Pumpkin |
7 |
75 |
Watermelon |
7 |
75 |
Broad Beans (cooked) |
7 |
80 |
Crackers |
60 |
80 |
Potato Crisps |
49 |
80 |
Tapioca |
94 |
80 |
Carrots (cooked) |
6 |
85 |
Corn Flakes |
85 |
85 |
Flour T55 – Baguettes |
58 |
85 |
Popcorn (no sugar) |
63 |
85 |
Rice Cake |
24 |
85 |
Honey |
80 |
90 |
Mashed potato |
14 |
90 |
Rice (pre-cooked) |
24 |
90 |
Potato (chips) |
33 |
95 |
Puffed Rice |
85 |
95 |
Beer |
5 |
110 |
Glycemic Index and Average Carbohydrate Concentration per 100g
|
Carbohydrate Concentration |
Glycemic Index |
All Bran |
46 |
30 |
Apple |
12 |
30 |
Apple Juice (fresh) |
17 |
40 |
Apricots (dried) |
63 |
35 |
Apricots (fresh) |
10 |
20 |
Aubergines |
4 |
10 |
Banana |
20 |
65 |
Beans – French |
3 |
30 |
Beans – Haricot |
17 |
30 |
Beer |
5 |
110 |
Black Bread (German) |
45 |
40 |
Boulgour (wholegrain, cooked) |
25 |
45 |
Bran Bread |
40 |
45 |
Broad Beans (cooked) |
7 |
80 |
Brocolli |
4 |
10 |
Brown Flour T85 (Brown Bread) |
50 |
65 |
Buckwheat (black wheat flour) |
65 |
50 |
Cabbage |
4 |
10 |
Carrots (cooked) |
6 |
85 |
Carrots (raw) |
7 |
35 |
Cereals (sugared) |
80 |
70 |
Cherries |
17 |
22 |
Chick Peas (cooked) |
22 |
30 |
Chinese Vermicelli (mungo bean) |
15 |
35 |
Chocolate Bars (ex. Mars Bar) |
60 |
70 |
Cola Drinks |
11 |
70 |
Corn Flakes |
85 |
85 |
Cornflour |
88 |
70 |
Crackers |
60 |
80 |
Crepe/Pancake (made w/ buckwheat) |
25 |
50 |
Dark Chocolate (<70% cocoa solids) |
32 |
22 |
Fig (fresh) |
12 |
35 |
Flour T150 (unrefined) - Pasta |
19 |
45 |
Flour T150 (unrefined) - Wholemeal Bread |
47 |
50 |
Flour T200 (unrefined) - Bread |
45 |
40 |
Flour T200 (unrefined) - Pasta |
17 |
40 |
Flour T55 – Baguettes |
58 |
85 |
Flour T65 - country style bread |
53 |
70 |
Fructose |
100 |
20 |
Fruit Preserve (w/out sugar or grape juice) |
37 |
30 |
Garlic |
28 |
10 |
Grapefruit |
10 |
20 |
Grapes |
16 |
40 |
Green Vegetables |
4 |
10 |
Honey |
80 |
90 |
Ice Cream (made w/ alginates) |
25 |
35 |
Jam (traditional) |
70 |
65 |
Kidney Beans |
11 |
40 |
Kiwi |
12 |
50 |
Lentils – Brown |
17 |
30 |
Lentils – Green |
17 |
22 |
Lettuce |
4 |
10 |
Maize/Corn on the cob (modern variety) |
22 |
70 |
Maize/Corn on the cob (traditional variety) |
21 |
35 |
Mashed Potatoes |
14 |
90 |
Melon |
6 |
65 |
Milk (semi-skimmed) |
5 |
30 |
Mushrooms |
4 |
10 |
Noodles, Ravioli |
23 |
70 |
Onions |
5 |
10 |
Orange |
9 |
35 |
Orange Juice (freshly pressed) |
10 |
40 |
Orange Juice (industrial) |
11 |
65 |
Peach |
9 |
30 |
Peanuts |
9 |
20 |
Pear |
12 |
35 |
Peas (Fresh Petis Pois) |
10 |
40 |
Peas - dried (cooked) |
7 |
35 |
Peas – split |
22 |
22 |
Petite Beurre Biscuit |
75 |
55 |
Plums |
10 |
22 |
Popcorn (no sugar) |
63 |
85 |
Potato (chips) |
33 |
95 |
Potato (peeled and boiled) |
20 |
70 |
Potato Crisps |
49 |
80 |
Potatoes (boiled in their skins) |
14 |
65 |
Puffed Rice |
85 |
95 |
Pumpkin |
7 |
75 |
Quinoa (cooked) |
18 |
35 |
Raisins |
66 |
65 |
Red Peppers |
4 |
10 |
Rice (Basmati) |
23 |
50 |
Rice (Brown) |
23 |
50 |
Rice (long grain, white) |
23 |
60 |
Rice (pre-cooked and non-stick) |
24 |
70 |
Rice (pre-cooked) |
24 |
90 |
Rice Cake |
24 |
85 |
Rye (wholemeal bread) |
49 |
40 |
Semolina (refined) |
25 |
65 |
Shortbread Biscuit (Flour B) |
68 |
55 |
Sorbet |
30 |
50 |
Soya (cooked) |
15 |
20 |
Spaghetti (hardgrain, cooked al dente) |
25 |
45 |
Sugar (saccharose) |
100 |
70 |
Sweet Potato |
20 |
50 |
Tapioca |
94 |
80 |
Tomatoes |
4 |
10 |
Turnip |
3 |
70 |
Walnuts |
5 |
15 |
Watermelon |
7 |
75 |
White Pasta (normal cooking) |
23 |
55 |
Yogurt (full-milk) |
4.5 |
35 |
Yogurt (skimmed) |
5.3 |
35 |
|
Glycemic Index (GI) by Glycemic Load (GL) |
||
|
|
|
|
|
Low GI |
Medium GI |
High GI |
|
All Bran Cereal |
Beats |
White Flour |
|
Apples |
Cantalope |
Whole Wheat Flour |
|
Carrots |
Pineapple |
Popcorn |
|
Chana Dal |
|
|
|
Chick Peas |
Succrose |
Watermelon |
|
Grapes |
Sugar |
|
|
Green Peas |
Banana |
|
Low GL |
Kidney Beans |
Mangos |
|
|
Oranges |
|
|
|
Peaches |
|
|
|
Peanuts |
|
|
|
Pears |
|
|
|
Pinto Beans |
|
|
|
Strawberries |
|
|
|
Red Lentils |
|
|
|
Sweet Corn |
|
|
|
Low GI |
Medium GI |
High GI |
|
Apple Juice |
Life Cereal |
Cheerios |
|
Buckwheat |
New Potatoes |
Shredded Wheat |
|
Fettucinni |
Sweet Potatoes |
|
Medium GL |
Navy Beans |
Fried Rice |
|
|
Orange Juice |
|
|
|
Parboiled Rice |
|
|
|
Sourdough Wheat Bread |
|
|
|
Low GI |
Medium GI |
High GI |
|
Linguini |
White Rice |
Baked Russet Potatoes |
High GL |
Maccaroni |
Couscous |
Corn Flakes |
|
Spaghetti |
|
|
|
|
|
|
|
|
|
|
|
GI: Low - 1-55 |
GL: Low - 1-10 |
|
|
Medium - 56-69 |
Medium - 11-19 |
|
|
High - >70 |
High - >20 |
|
TRANS-FATTY ACIDS:
These are found in commercially prepared foods and are synthesized when hydrogen is applied to edible oils under high pressures and temperatures in the presence of a catalyst. Hydrogenation of oils is done by the food industry to prolong the shelf life of commercially baked goods such as cookies, crackers, doughnuts, croissants, and snacks. It is also found in shortening, margarines, and deep-fried foods. These hydrogenated fats lower HDL levels, increase LDL levels, and increase the risk of cardiovascular disease. Some studies indicate that replacing trans-fatty acids with the same amount of natural, unsaturated fatty acids will result in a 50% reduction in risk of coronary heart disease.
OMEGA-3 FATTY ACIDS:
The sea algae and the grasses and leaves on land are rich in omega-3 fatty acids. Therefore, fish and larger grazing animals have a high content of omega-3 fatty acids. However, today, the meats from domesticated animals are very low in omega-3 fatty acids because they are generally grain- or corn-fed rather than grass-fed. Compared to the European Mediterranean diet, the American dietary intake of omega-3 is extremely low. Patients on a Mediterranean diet rich in omega-3 fats, unsaturated fats, fruits, vegetables, legumes, and nuts experienced 50 to 70% reductions in risk of cardiovascular events in long-term follow-up. In the GISSI prevention study, survivors of myocardial infarction were given 1g a of omega-3 supplements daily and experienced a 45% reduction in sudden cardiac death and a 20% decrease in all cause mortality during a 3 ½-year follow-up. Increased fats in the form of omega-3 fatty acids either from plant sources such as flaxseed oil or fish oils will reduce cardiovascular risks anywhere from 32% to 50%.
MONOUNSATURATED FATS:
Our ancestors had a diet where one half of the total fat is composed of monounsaturated fats. Monounsaturated fats reduce cardiovascular risks when substituted for the high glycemic index carbohydrates and sugars. Nuts are a valuable source of monounsaturated fats and have been shown to be cardio-protective in multiple studies. The calories in nuts are typically 80% from fat, but most of the fat is healthy monounsaturated and polyunsaturated fatty acids. Epidemiological studies showed that nut consumption, five times a week at least, is associated with a 50% reduction in the risk of myocardial infarction, compared to the risk for people who rarely or never eat nuts. Nut consumption also reduces the risk of developing diabetes, lowers LDL, and provides a plant based protein together with vitamin E, folic acid, magnesium, copper, zinc, and selenium. Nuts have a high satiety index and therefore, often prevent over-eating. Oleic acid is the major monounsaturated fat in our diets and is found in meats, nuts, avocados, dark chocolate, and olive oil. Replacing saturated fat with monounsaturated fats results in a 30% reduction in risk.
VEGETARIANISM:
Our ancestors were omnivorous. Modern vegetarian diets rely heavily on processed carbohydrates such as white rice, potatoes, white flour, and sugars. Therefore, they are not recommended. The South Asian paradox refers to the high prevalence of coronary artery disease despite low levels of LDL cholesterol and low prevalence of obesity in urban vegetarians from India who consume a diet that is high in refined carbohydrates. High glycemic index (GI) diets lower HDL cholesterol and predispose people to metabolic syndrome and cardiovascular disease. In fact, high-GI diets are one of the most atherogenic features of our modern eating pattern. Overall, a vegetarian diet composed of low GI foods is cardio-protective. However, our current vegetarian diets are very rich in refined flour and grains; as a result; their GI values are extremely high. Consuming foods in the natural state retains more nutritional value and keeps the GI of the food low.
Our ancestors derived 45% of their calories from animal foods. However, the meats had less than 4% fat by weight and contained relatively high levels of monounsaturated and omega-3 fatty acids, compared to the grain-fed domestic meats of today that contain 20% to 35% fat - much of it in the form of saturated fats. It is highly likely that it is the high levels of saturated fats typically found in the meat of modern domesticated animals which is most atherogenic. There are many other compelling reasons for not consuming large amounts of currently available meats in the United States. The leading source of saturated fats and cholesterol in the American diet is meat, poultry, and dairy products. These are directly linked with atherogenesis, or accumulation of plaque on the innermost layer of the artery walls. There is no cholesterol found in grains, legumes, fruits, vegetables, nuts, and seeds.
BEVERAGES:
Our ancestors only drank water. Soda drinks are the predominant beverage consumed in America today. Over 50 million cans of soda are sold every day in the United States of America. These are calorie dense, nutritionally empty drinks which increase the risk of obesity and insulin resistance. Between 1990 and 1995, the consumption of soda among children and adolescents increased by 41%. The average consumption of soda among teenage males between 13 and 18 years is 3 or more cans of soda pop a day. Ten percent of teenage males drink 7 or more cans a day. The average teenage girl drinks two cans of soda pop a day, and 10% of all teenagers drink more than 5 cans a day. The Center for Science in the Public Interest is asking for more water fountains, soda-free schools, and health education campaigns in schools. Soda consumption is linked to obesity, tooth decay, caffeine dependence, and bone weakening. A team of Harvard researchers presented the first evidence linking soft drink consumption to childhood obesity in The Lancet. Recent human studies also demonstrate that girls consuming soda pop are more prone to have brittle bones. Recent animal studies using rats also demonstrated the same findings. Phosphorus encourages calcium loss and weakening of the bones. Soda consumption increases the likelihood of bone fractures in female teenagers fivefold.
Tea is high in natural antioxidants, which are beneficial. An inverse relationship between tea consumption and cardiovascular disease has been observed.
SUMMARY:
The most practical solution for reducing the incidence of chronic degenerative diseases such as atherosclerosis is to realign our current maladaptive diet and lifestyle to simulate what our bodies were initially made to consume. Our food sources today are different from the food sources that were available to our ancestors; for example, today’s meat is totally different from the meat that was available to our ancestors. Our vegetarian diets have become so refined and have such high glycemic indices that it is essential that we make radical changes to our eating habits and follow a more natural, healthy diet.
It is therefore my strong recommendation that the ideal diet currently available to the public is predominantly a vegetarian diet, closely related to the Mediterranean diet. It should be rich in fruits, vegetables, and whole grains, and very low in meats and dairy products. The ideal diet should also be sugar-free. The best drink is water, and the next-best would be tea.
ARGUMENTS AGAINST MEAT CONSUMPTION
Cholesterol: The leading sources of saturated fats and cholesterol in the American diet is meat, poultry, and dairy products. Vegetables do not contain cholesterol. The body is able to make its own cholesterol, so it is important to limit your dietary cholesterol intake so that your cholesterol levels remain in the normal range. Cholesterol is directly responsible for heart disease, which is the most common cause of death in the United States, with $135 billion spent annually to treat it.
Cancer: Among men who consume meats, dairy products, and eggs on a daily basis, the risk of fatal prostate cancer 3.5 times greater than the risk for men who consume meat sparingly. The risk of breast cancer among women who eat meat daily is 3.5 times higher than the risk for women who eat meat once a week. There is a 3.2 times greater risk of breast cancer among women who eat butter and cheese two to four times a week compared to women who eat butter and cheese once a week. Nearly 40% of all cancers in the United States are related to diet.
Health: Approximately 68% of all diseases are diet-related. The following conditions can be commonly prevented or improved with a low-fat diet that is free from animal products: arthritis, breast cancer, asthma, colon cancer, diabetes, constipation, gallstones, diverticulosis, heart disease, hypertension, impotence, hypoglycemia, renal disease, obesity, peptic ulcers, osteoporosis, prostate cancer, food poisoning, and stroke.
Protein: Many feel that a vegetarian diet will not provide enough protein in their diets. According to the World Health Organization, only 4.5% of the calories need to come from protein. According to the food and nutrition Board of the United States Department of Agriculture, only 6% of the protein calories are needed. The national research Council states that only 8% of the calories need to be derived from protein. The following vegetables have high percentages of calories as protein: Broccoli 47%; zucchini 28%; wheat: 17%; brown rice 8%; lettuce is 34%; tomatoes 18%; potatoes 11%.
Antibiotic: About 55% of all the total antibiotic production in the United States is fed to livestock and finds its way into our foods. Staphylococcal resistance to penicillin increased from 13% in 1960 to 291% in 1988. A major contributor to the rise of antibiotic resistance is the breeding of antibiotic resistant bacteria in farms due to routine feeding of antibiotics to livestock.
Food Safety: One-third of all inspected chickens are infected with salmonella. Surprisingly, 75% of federal poultry inspectors said that they would not eat chicken.
The Environment: One-third of US cropland is permanently removed from production due to excess soil erosion directly related to animal husbandry. One pound of feedlot steak requires the loss of 55 pounds of topsoil. Every second, US livestock produces 250,000 pounds of excrement. Water pollution created by US Agriculture with the runoff of topsoil, pesticides, and manure is greater than all municipal and industrial sources combined. Fifty percent of all the wells and surface streams in the United States are contaminated by agricultural pollutants. Approximately 200 years ago, America’s top soil was 21 inches deep; now, it is only 6 inches deep. It takes 500 years to the paste one inch of topsoil. Nearly 85% of the topsoil loss is directly related to cattle farming. One acre of land can produce 20,000 pounds of potatoes or 165 pounds of beef. A startling arithmetic is as follows: the livestock population in the United States consumes enough grain and soybeans to feed the entire human population of the United States five times over. Cycling our grain through livestock, we receive only 10% of the available calories.
Over one-half of the water in our country is used to grow feed and fodder for livestock. If cattle farmers in California reduced their water consumption by just 6% it would be equivalent to a 75% reduction in domestic use.
The Natural Resources: One-third of all the raw materials, including base products, for farming, forestry, and mining (including fossil fuels) consumed in the United States is devoted solely to the production of livestock. It requires 78 calories of fossil fuel to produce one calorie of protein from beef. It requires only two calories of fossil fuel to produce one calorie of protein from soybeans. The livestock production accounts for more than half of all water consumption for all purposes in the United States. Approximately 64% of all United States cropland is used for producing livestock feed. Only 2% of the United States cropland is used to produce fruits and vegetables.
One acre of prime land can produce 5000 pounds of cherries, 20000 pounds of apples, 40000 pounds of potatoes, 60000 pounds of celery, 10000 pounds of green beans, 30000 pounds of carrots, and 50000 pounds of tomatoes. Yet, one acre of prime land only produces 250 pounds of beef.
World Hunger: It is ironic that 70% of the grain production in the United States is actually consumed by livestock. Yet, 38,000 children die every day because of malnutrition and starvation. Livestock consumes 50% of the world grain harvest. If we could reduce our intake of meat by a fraction, there would be so much land, water, and energy freed up from growing livestock that we could probably feed millions of people.
For more information, read John Robbins: “Diet for a New America David Pimentel,” Energy and Land Constrains in Food Protein production,” Science
Newsweek, The Browning of America Feb 22, 1981
WHAT ABOUT MILK?
Milk contains a sugar called lactose. At least 20% of the population does not produce the enzyme, lactase, needed to digest lactose. Furthermore, milk products are very fatty and create tremendous amounts of mucus, which can line the intestinal tract and decrease absorption of other nutrients. To test for lactose intolerance, one can do a simple experiment and consume lots of milk. If you notice postnasal drip and excess of phlegm in your throat, it is a clear indication of the effects of milk. The same applies to cheese as well. Milk also contains growth hormones that are designed to increase the weight of the calf from 90 pounds and birth to 1000 pounds within two years. Large quantities of growth hormones have been detected in milk. The principal protein in cow’s milk is casein. However, casein is not the protein that humans need. Much of the casein remains undigested in humans, and may be responsible for much of the allergies known today.
Americans consume more milk products than any other country in the world; yet, there is a very high incidence of osteopenia among Americans. Consuming milk products can actually cause osteoporosis. The incidence of osteoporosis is lowest in countries with smallest amounts of milk products are consumed.
The adult cow does not drink milk, then why should humans? We are the only adult mammals that drink the milk of another species.
WHAT WE SHOULD EAT TODAY
- Avoid all highly processed foods. Eat foods in their natural state. Food should be fresh, so eat organic food, free of hormones and pesticides.
- Avoid foods of high glycemic index. Remember, processed foods increase the glycemic index. Avoid all sugars. Do not eat any foods with more than 5g sugar. Your body was not designed for high-sugar diet.
- Increase your intake of omega-3 fatty acids. Eat more nuts, use flaxseed oil, eat flaxseeds, or use mustard seed oil. Eat two fish dishes a week, or take pharmaceutical grade fish oil supplements.
- Significantly increase your intake of berries especially blueberries, nuts, vegetables, and fruits: Citrus, berries, apples, spinach, tomatoes, broccoli, cauliflower, and avocado.
- Eliminate all trans-fats entirely from the diet by eliminating fried foods and margarines. Avoid all baked goods or processed foods. Hydrogenated or partially hydrogenated fats should be avoided completely. Eliminate all saturated fats. Instead, substitute with monounsaturated fats such olive oil.
- Eliminate dairy products as much as possible. The high fats in dairy products should be avoided completely.
- Avoid processed meats such as bacon, sausage, and deli meats.
- If you are going to eat any animal protein, consume only organic and lean meats such as skinless poultry, fish, and game meats.
- Exercise daily. Your aerobic activity heart rate should be 180 minus age.
- Drink water. Avoid all sodas. Restrict alcohol to one to two drinks a day only. Restrict coffee to one to two small drinks a day. Green tea is good.
Albert CM, Gaziano JM, Willett WC, Manson JE, Nut consumption and decreased risk of sudden cardiac death in the Physicians’ Health Study. Arch Intern Med. 2002;162:1382-1387.
Atkins RC. Dr. Atkins; The New Diet Revolution. New York, NY: Avon Books; 1998.
Cohen MN. Health and the Rise of Civilization. New Haven, Conn; Yale University Press; 1989:118-119.
Cordain L. The nutritional characteristics of a contemporary diet based upon Paleolithic food groups. J Am Neutraceut Assoc. 2002;5:15-24.
De Lorgeril M, Salen P, Martin JL, Monjaud I, Delaye J, Mamelle N. Mediterranean diet, traditional risk factos, and the rate of cardiovascular complications of myocardial infarction: final report of the Lyon Diet Heart Study. Circulation. 1999;99:779-785.
Foot DK, Lewis RP, Pearson TA, Beller GA. Demographics and cardiology, 1950-2050. J Am Coll Cardiol. 2000;35(5, suppl B):66B-80B.
Ford ES, Giles WH, Dietz WH. Prevalence of the metabolic syndrome among US adults: finding from the third National Health and Nutrition Examination Survey. JAMA. 2002;287:356-359.
Ford ER, Liu S. Glycemic index and serum high-density lipoprotein cholesterol concentration among US adults. Arch Intern Med. 2001;161:572-576.
GISSI-Prevenzione Investigators (Guppo Italiano per lo Studio della Sopravvivenza nell’Infarto miocarico). Dietary supplementation with n-3 polyunsaturated fatty acids and vitamin E after myocardial infarction; results of the GISSI-Prevenzione trial [published correction appears in Lancet. 2001;357:642]. Lancet. 1999;354:447-455.
Hu FB, Willett WC. Optimal diets for prevention of coronary heart disease. JAMA.2002;288:2569-2578.
Joshipura KJ, Hu FB, Mason JE, et al. The effect of fruit and vegetable intake on risk for coronary heart disease. Ann Intern Med.. 2001;134:1106-1114.
Kris-Etherton PM, Harris WS, Appel LJ, American Heart Association, Nutrition Committee. Fish consumption, fish oil, omega-3 fatty acids, and cardiovascular disease [published correction appears in Circulation. 2003;107:512]. Circulation.2002;106:2747-2757.
Lemaitre RN, King IB, Mozaffarian D, Kuller LH, Tracy RP, Siscovick DS. N-3 Polyunsaturated fatty acids, fatal ischemic heart disease, and nonfatal myocardial infarction in older adults: the Cardiovascular Health Study. Am J Clin Nutr. 2003;77:319-325.
Lugwig DS. The glycemic index: physiological mechanisms relating to obesity, diabetes, and cardiovascular disease. JAMA. 2002;287:2414-2423.
Marchiloi R, Barzi F, Bomba E, et al, GISSI-Prevenzione Investigators. Early protection against sudden dealth by n-3 polyunsaturated fatty acids after myocardial infarction: time-course analysis of the results of the Guppo Italiano per lo Sudio della Sopravvivenza nell’Infarto Miocardico (GISSI)-Prevenzione. Circulation. 2002;105:1987-1903.
Ornish D. Dr. Dean Ornish;s Program for Reversing Heart Disease: The OnlySystem Scientfically Proven to Reverse Heart Disease Without Drugs or Surgery. New York, NY: Random House; 1990.
Reddy ST, Wang CY, Sakhaee K, Brinkley L, Pak CY. Effect of low-carbohydrate high-protein diets on acid-base balance, stone-forming propensity, and calcium metabolism. Am J Kidney Dis. 2002;40:265-274.
Sacks FM, Katan M. Randomized clinical trials on the effects of dietary fat and carbohydrate on plasma lipoproteins and cardiovascular disease. Am J Med. 2002;113(suppl 9B):13S-24S.
Singh RB, Dubnov G, Niaz MA, et al. Effect of an Indo-Mediterranean diet on progression of coronary artery disease in high risk patients (Indo-Mediterranean Diet Heart Study): a randomized single-blind trial. Lancet. 2002;360:1455-1461.
Vasan RS, Beiser A, Seshadri S, et al. Residual lifetime risk for developing hypertension in middle-aged women and men; the Framingham Heart Study. JAMA. 2002;287:1003-1010.
Updated February 4, 2009 - MJ
Vitamin D and Cardiovascular Disease
What does Vitamin D do?
- prevent chronic diseases
- keep your bones and teeth strong and healthy
- reduce inflammation
- regulate growth and activity of your cells
What is the Major Source of Vitamin D?
Most Vitamin D is produced in your body through the sun’s ultraviolet light, which reacts with the 7-dehydrocholesterol precursors on your skin. This reaction converts the precursors to Vitamin D3. In the liver, Vitamin D3 undergoes 25-hydroxylation to form 25(OH)D.1 The best level of 25(OH)D is suggested to be 30ng/ml or above.1
Few foods contain Vitamin D, but some that do include the following: fish (salmon, mackerel, tuna), and fish liver oils are among the best sources of Vitamin D. Beef liver, cheese, and egg yolks contain small amounts of Vitamin D.2
In the American diet, fortified foods provide the most Vitamin D; this includes the U.S. milk supply, fortified with 100 IU/cup of vitamin D.2
Symptoms of Vitamin D Deficiency
- rickets – a condition characterized by bone deformities and growth retardation
- bone pain
- osteomalacia (soft bone)
- frequent bone fractures
- osteoporosis
How Can I Test for Vitamin D Deficiency?
The SpectraCell Analysis Test is an evidence based test that measures the levels of select vitamins (including Vitamin D), minerals (including calcium), antioxidants, and other micronutrients in your white blood cells. This test is important because nutrient status is a very important component of your health. Every micronutrient plays an important role in optimal functioning of your cells. The SpectraCell Analysis Test will identify any micronutrient deficiencies you may have; identifying and correcting deficiencies is essential in maintaining optimal health.
The SpectraCell Analysis Test is available Monday through Friday from 8:30am-3:30pm
Vitamin D and Cardiovascular Disease
Studies have shown that low 25(OH)D levels are associated with cardiovascular heart disease and congestive heart failure.1
Vitamin D and Hypertension
Results from cross-sectional studies show an inverse relationship between Vitamin D levels and blood pressure – the higher the Vitamin D levels, the lower the blood pressure.1 Other studies show that high blood pressures were associated with higher levels of parathyroid hormone (PTH), a hormone that increases blood calcium concentration by removing calcium from your bones.1
Vitamin D and Diabetes
Insulin is secreted by cells in the pancreas called beta cells. According to a meta-analysis, Vitamin D appears to influence the response of insulin to elevated glucose levels; Vitamin D may also regulate extracellular calcium to ensure that there is normal calcium influx through the cell membranes and intracellularly. This is important because insulin release by beta cells is dependent on calcium.3 Furthermore, calcium is needed in insulin-mediated intracellular processes in tissues that are insulin-responsive, such as adipose tissue and skeletal muscle.3 Therefore, changes in the concentration of calcium in the cell may lead to insulin resistance because of impaired signaling in the cell. Insulin resistance, in turn, can lead to diabetes.
Vitamin D and Obesity
Vitamin D deficiency has been linked to obesity.1 A recent study found that obese individuals have lower levels of 25(OH)D and higher PTH levels than do non-obese individuals. Vitamin D is fat soluble and therefore is stored in adipose tissue.4 Results from the study found identical concentrations of Vitamin D among obese and non-obese patients after exposure to UV-B irradiation.4 However, the increase in blood Vitamin D concentration was less in obese patients than in non-obese patients.4 This suggests that obesity does not affect the capacity of the skin to produce Vitamin D, but obesity may alter the release of Vitamin D from the skin to the bloodstream because the excess adipose tissue hinders Vitamin D’s release into the bloodstream.4
Calcium and Metabolic Syndrome
A research study using data from the Women’s Health Study found an inverse relationship between calcium intake and prevalence of metabolic syndrome. This study did not find a relationship between Vitamin D intake and metabolic syndrome, but the effects of Vitamin D are dependent on the presence/absence of other nutrients such as calcium. Therefore, low serum calcium levels influence Vitamin D action, and this affects your health.
George Mateljan Foundation (2009). Vitamin D. Retrieved February 16, 2009, from http://ods.od.nih.gov/factsheets/vitamind.asp#en23.
Pittas, A.G. et al. (2007). The role of vitamin D and calcium in type 2 diabetes. A systematic review and meta-analysis. The Journal of Clinical Endocrinology & Metabolism, 92(6), 2017-2029.
Updated February 16, 2009 - MJ
"No Whites" Diet
The purpose of Dr. Jamnadas’ “No Whites” Diet is to lose weight. This is a fairly simple diet and is easy to follow as well.
The plan consists of:
- No pastas
- No breads, including all grains
- No potatoes
- No milk products, except for skim milk and low fat mozzarella
- No oils; use olive oil if needed
- No margarine (because of trans-fatty acids)
- No white salad dressings
- No sugars
If you are still hungry, have more of the main entrée (the proteins):
- Chicken
- Turkey
- Fish
- Lean meats
- Beans
- Lentils
Make sure you include:
- Vegetables (eat a salad!)
- Fruits (including peaches, plums, apples, apricots)
- Avoid cantaloupe, watermelon, bananas, kiwi fruit, and pineapple
- Nuts (serving size is fistful)
Following this plan will provide you with a healthy and safe way to lose weight. You are on your way to a healthier new you!
Updated February 26, 2009 - MJ
The TV Method: Exercise Made Fun and Easy
Exercise is an important component in maintaining optimal cardiovascular health; yet many people feel that time conflicts prevent them from exercising. However, the TV Method, an innovative and fun exercise regimen, will allow you to gain the health benefits of exercise while you watch your favorite television programs.
In a Nutshell:
If you have a treadmill, elliptical, or cycling machine at home, place it near the television. Find a show to watch on television and exercise while the program is running.
Workout Schedule:
Week 1: Pick a 30-minute television program to watch. Exercise during the program. Take a break during the commercials. This is a 22 minute workout.
Week 2: Pick another 30-minute television program. Exercise throughout the duration of the program and also during the first commercial break. Take a break during the second set of commercials. This is a 25 minute workout.
Week 3: Pick another 30-minute television program. Exercise throughout the duration of the program, including both commercial breaks. This is a 30 minute workout.
Week 4: Pick a 1-hour television program to watch. Exercise during the program and take a break during the commercials.
Week 5: Pick a 1-hour television program to watch. Exercise during the program and every other commercial break.
Week 6: Pick a 1-hour television program to watch. Exercise throughout the entire duration of the program.
|
Week 1 |
Week 2 |
Week 3 |
Week 4 |
Week 5 |
Week 6 |
Exercise |
Only during program |
During program + first commercial break |
During program + both commercial breaks |
Only during the program |
During program + every other commercial break |
During program + all commercial breaks |
Rest |
Every commercial break |
Second commercial break |
None |
Every commercial break |
Alternating commercial breaks |
None |
The Importance of Physical Fitness
Over the past 25 years, the United States has experienced a steady decline in the number of deaths from cardiovascular disease (CVD), primarily in mortality caused by coronary heart disease and stroke. Still, coronary heart disease is the leading cause of death while stroke is third. Lifestyle improvements and better control of the risk factors for heart disease and stroke have been major contributors to this decline.
There are many risk factors associated with coronary heart disease and stroke. Modifiable risk factors include smoking, high blood pressure, blood lipid levels, obesity, diabetes, and physical inactivity. National trends observed with smoking, high blood pressure, and high blood cholesterol have improved, but obesity and physical inactivity have not. Advanced technologies have taken the place of physical activity in many daily tasks. As a result, many Americans are physical inactive.
Physical activity protects against the development of CVD and also improves other CVD risk factors, including high blood pressure, blood lipid levels, insulin resistance, and obesity. Physical activity is also important in the treatment and management of those with hypertension, stable angina, prior myocardial infarction, peripheral vascular disease, or heart failure. Physical activity, therefore, is important for cardiac rehabilitation.
Advantages to an Active Lifestyle
Physical inactivity among the U.S. population is now widespread. About one in four adults (more women than men) currently lead sedentary lifestyles with no leisure time physical activity. An additional one-third of adults are insufficiently active to achieve health benefits. The prevalence of inactivity varies by gender, age, ethnicity, health status, and geographic region, but it is common to all demographic groups.
Girls become less active than boys as they grow older. Children become far less active as they move through adolescence, and as a result, obesity is increasing among children. It is related to an energy imbalance (i.e., calories consumed in excess of calorie expended.) Data indicate that obese children and adolescents have an increased risk of becoming obese adults, and obesity in adulthood is related to coronary artery disease, hypertension, and diabetes. Thus, the prevention of childhood obesity has the potential of preventing CVD in adults.
There is evidence that increased physical activity leads to weight loss and that combining physical activity with reducing food intake can help maintain a healthy body weight.
Middle-aged and older men and women who engage in regular physical activity have significantly higher high-density lipoprotein (HDL) cholesterol levels than do those who are sedentary. When exercise training has extended to at least 12 weeks, the HDL cholesterol levels are even more improved.
What Type, Intensity, and Quantity of Exercise Are Best?
You don't need a structured or vigorous exercise program to reduce CVD risk factors and gain many other health benefits. Most benefits of physical activity can be gained by performing moderately intense activities.
Everyone should maintain regular physical activity at a level appropriate to his or her abilities and interests. Both children and adults should have at least 30 minutes or more of moderate-intensity physical activity most days of the week. However, physical activity must be performed regularly to maintain positive effects. Intermittent or shorter bouts of activity (at least 10 minutes), including tasks of daily living, also have similar cardiovascular and health benefits if performed at a level of moderate intensity (such as brisk walking, cycling, vacuuming, and yard work) for a total of 30 minutes daily. People who currently meet the recommended minimal standards may gain additional health and fitness benefits from increasing their activity. Higher intensity or longer duration activity could be performed approximately three times weekly and achieve cardiovascular benefits, but low-intensity or shorter duration activities should be performed more often to achieve cardiovascular benefits.
People who are not currently active should gradually build up to the recommended goal of 30 minutes of moderate activity daily by adding a few minutes each day until reaching their personal goal to reduce the risk of injury.
Developing muscular strength and joint flexibility is also important for an overall activity program to improve one's ability to perform tasks and to reduce the potential for injury. Resistance training may contribute to better balance, coordination, and agility that may help prevent falls in the elderly.
Physical activity carries risks as well as benefits. The most common adverse effects of activity relate to musculoskeletal injury and are usually mild and self-limited. The risk of injury increases with increased intensity, frequency, and duration of activity and also depends on the type of activity. Exercising in moderation can reduce these injuries.
Because the risks of physical activity are very low compared with the health benefits, most adults do not need medical consultation before starting a moderate-intensity physical activity program. However, those with known CVD and men over age 40 and women over age 50 with multiple cardiovascular risk factors should have a medical evaluation prior to initiating such a program.
Updated January 29, 2009 MJ
Nature's Superfoods
Food’s Influence on Your Health:
-
Nearly half of all cardiovascular disease and hypertension cases can be attributed to diet
-
An estimated 300,000 to 800,000 preventable deaths per year in the United States are nutrition related. This number includes deaths from atherosclerotic diseases, diabetes, and some cancers
Fruits, vegetables, and other natural foods are loaded with phytonutrients
What are Phytonutrients?
- Phytonutrients are non-vitamin, non-mineral compounds found in foods that have significant benefits for human health. Benefits include:
- Improved cell-cell communication in our bodies
- Prevention of genetic mutations in cells
- Prevention of cancer cell proliferation
3 Types of Phytonutrients:
- Polyphenols: are antioxidants, have anti-inflammatory properties, and are anti-allergenic.
- Carotenoids: are the pigments that give yellow and red vegetables their color. This class of phytonutrients includes beta-carotene, lutein, and lycopene, which are all antioxidants and protect us from cancer and signs of aging.
- Phytoestrogens: are natural chemicals that are found in soy foods, whole wheat, seeds, grains, and some vegetables and fruits. Phytoestrogens may serve as a protective factor against breast and prostate cancers.
Micronutrients in Fruits/Vegetables are Age-Defying
- Micronutrients are very powerful antioxidants, which keep your body healthy and prevent oxidation.
- Our bodies need oxygen to conduct many metabolic processes. As a result of these metabolic activities, oxygen is consumed and transformed into unstable free radicals - oxygen atoms with a missing electron.
- The oxygen free radicals want to find a molecule to replace its missing electron, and so the free radicals seek out molecules from any cells they can attack, causing damage to the cell.
- This includes DNA, proteins, and enzymes.
- Antioxidants fight free radicals in your body and neutralize them by giving them an extra electron
- Battling free radicals may lead to improved long-term health
BEANS
Nutrients:
- low-fat protein
- Beans contain the amino acid Lysine, which is deficient in many forms of plant proteins.
- phytonutrients
- Usually, as you increase protein intake, you increase bone calcium loss
- Eating animal protein leads to increased acidity in the body, contributing to increased calcium loss from the body
- Eating plant protein leads to reduced loss of bone calcium compared to when you eat animal protein
- Fiber, folate, potassium, magnesium, iron, and B Vitamins
Nutritional Benefits of Beans
Lower cholesterol levels:
Frequent consumption of beans is associated with lower cholesterol levels. Cholesterol is found only in animal foods, so if you substitute animal protein with plant protein, and if you limit your intake of saturated fat and partially hydrogenated oils, you will be on track to lowering your cholesterol
Combat cardiovascular disease:
Beans are just as effective at keeping low blood cholesterol levels are oat bran is. Combine the two with a regular exercise regimen, and you have a great remedy for reducing your cardiovascular risk.
Reduce obesity:
Beans have a low Glycemic Index (GI), meaning that when you eat beans, they are slowly absorbed through the blood stream. Consequently, you feel satiated after eating, and this prevents overeating.
Reduce blood sugar:
Beans are high in fiber and have a low GI, so they help maintain a steady blood sugar level and provide slow-burning energy for your body.
Reduce cancer risk:
Beans contain phytoestrogens called lignins that have estrogen-like properties that help reduce estrogen levels and reduce the risk of cancers (mainly breast cancer and prostate cancer) that are related to estrogen levels.
How Do I Incorporate Beans Into My Diet?
- Make hummus – puree garbanzo beans with garlic, olive oil, and tahini sauce
- Bean salads – mix different cooked beans together with some olive oil and herbs
- Beans and pasta
- Boil them and add spices
BLUEBERRIES
Nutrients:
- Phytonutrients
- Low Calories
- Polyphenols
- Carotenoids
- Fiber, Folate, Potassium, Magnesium, Manganese, Iron,
- Riboflavin, Niacin
Nutritional Benefits of Blueberries:
Antioxidant Boost:
Blueberries contain more antioxidants than do any other fruit/vegetable. One serving of blueberries (1/2 cup) contains as many antioxidants as do five servings of carrots, apples, broccoli, and squash. Antioxidants are important because they can reduce your risk of cancer, cardiovascular disease, and diabetes.
Anthocyanin:
This is an antioxidant in the flavonoid family that gives blueberries their strong blue color and their powerful antioxidant properties. Anthocyanin helps to neutralize cellular free-radical damage.
Reduce Effects of Age-Related Conditions:
Animal studies have shown that blueberries help to reduce effects of Alzheimer’s disease and dementia.
Quercetin:
This is another antioxidant in the flavonoid family that has anti-inflammatory properties.
The Brain:
The nutrients in blueberries have an affinity for the areas in the brain that control movement. Studies assessing motor skills showed that people who ate 1 cup of blueberries daily performed 5-6% better on motor skills tests, compared with the control group.
Digestive Health:
Blueberries are rich in pectin, a soluble fiber that works to relieve diarrhea and constipation. The tannins in blueberries reduce inflammation in the digestive system. Blueberries also reduce the ability of the E.coli bacterium to adhere to the mucosal linings of the bladder and urethra. This reduces the risk of urinary tract infections.
How Do I Incorporate Blueberries Into My Diet?
- dried fruit
- sprinkle some in yogurt, cereal, or oatmeal
- make a blueberry smoothie
- muffins, cakes, and pancakes
BROCCOLI
Nutrients:
- low calories
- iron, folate, fiber, calcium
- Vitamin C, Vitamin K, beta-carotene
- Indoles, sulforaphane
- These are phytochemicals that combat cancer by blocking estrogen receptors on breast cancer cells, preventing the growth of estrogen-sensitive breast cancer cells.
- Indole-3-carbinol (I3C) is a powerful indole that is a strong breast cancer preventive agent
Nutritional Benefits of Broccoli
Prevention of Birth Defects:
Broccoli is loaded with folate and Vitamin B, which helps prevent birth defects, such as spina bifida.
Cardiovascular Disease Prevention:
The folate in broccoli helps remove homocysteine from the circulatory system. This is important because high levels of homocysteine are linked with cardiovascular disease. Broccoli is also one of the few vegetables that have a high level of coenzyme Q10, which is a fat-soluble antioxidant linked to energy production in the body. In patients with cardiovascular disease, coenzyme Q10 serves a cardio-protective function.
Cancer Prevention:
Researchers at Johns Hopkins University discovered a compound in broccoli that prevented tumor development by 60% and reduced the size of developed tumors by 75%. Another study showed that eating two servings of cruciferous vegetables (mainly cabbage, broccoli, and Brussels sprouts) a day results in as much as a 50% reduction in the risk for certain types of cancers. Furthermore, ½ cup of broccoli daily protects from numerous cancers, including colon, lung, stomach, and rectal cancers.
Bone Health:
Broccoli, along with other cruciferous vegetables, help build your bones because it is loaded with calcium and Vitamin C, which increases calcium absorption to your bones.
How Do I Incorporate Broccoli Into My Diet?
- Steam or microwave it
- Stir-fry with other vegetables, garlic, chopped toasted walnuts or pine nuts
- Toss some in a salad with light vinaigrette dressing
- Puree with sautéed onion and mix with low-fat milk/soymilk for a quick soup
- Cut and toss with olive oil and salt. Roast for 20-30 minutes at 425°F
- Dip raw broccoli florets in hummus for a fresh and healthy snack
OATS
Nutrients:
- low calories, high protein
- high fiber, magnesium, potassium, zinc, copper, manganese, selenium, thiamine
Nutritional Benefits of Oats:
Lowers Cholesterol Levels:
Oats contain beta glucan, a soluble fiber that is responsible for lowered cholesterol levels. Since oats are low on the GI, they help maintain a steady blood glucose level; this is especially important for diabetics.
Improved Cardiovascular Health:
Consumption of whole grains is associated with lowered overall mortality rates, lowered cholesterol levels, and stabilized blood sugar levels. Additionally, whole grain consumption is linked to a reduction in stroke risk.
Phytochemicals:
The germ and bran of oats have lots of phytonutrients, including ferulic acid and caffeic acid. Ferulic acid may be beneficial in preventing colon cancer; additionally, it has been found to be a strong antioxidant to protect against oxidative damage to the cell. Lastly, it inhibits the formation of cancer-promoting compounds in the body.
Flaxseeds:
Flaxseeds are the best plant source of omega-3 fatty acids, as well as fiber, protein, iron, and magnesium. Flaxseeds also contain lignins, which help to protect against breast cancer. Flaxseeds are usually found in stores as ground (flaxseed meal) or in whole seed form. They must be ground before eating because the nutrients are more easily absorbed when ground.
Wheat Germ:
This is the embryo of the wheat berry, and it is loaded with nutrition, and it is a good source of omega-3 fatty acids.
Whole Grains:
This relates to the Glycemic Index (GI), a ranking scale of carbohydrates and how quickly they are absorbed into the body. The lower the GI, the slower the absorption into the bloodstream is. As a result, you feel satiated (full) when you eat low-GI foods, and this prevents overeating and helps to maintain your ideal weight. Whole grains decrease the risk of coronary heart disease, stroke, diabetes, hypertension, obesity, and some cancers.
There are 3 parts to a whole grain:
- Bran: This the outer layer of the grain, and it contains B vitamins, protein, and phytochemicals.
- Endosperm: The middle layer, containing carbohydrates, proteins, and small amount of Vitamin B
- Germ: This is the inner-most layer of the grain, containing B Vitamins, Vitamin E, and phytochemicals
How Do I Incorporate Whole Grains Into My Diet?
- Eat whole grain bread, tortillas, crackers, cereals
- Eat brown rice instead of white rice
- Toss some oats in stuffing, meats, and meat loaf
- Add oats to homemade muffins, cakes, pancakes, yogurt
ORANGES
Nutrients:
- Vitamin C
- Fiber, folate, potassium
- Limonene, polyphenols, pectin
Nutritional Benefits of Oranges:
Citrus Flavonoids:
These are a class of polyphenols found in fruits’ tissue, juice, pulp, and skin and contain the health-promoting power of citrus foods. Citrus flavonoids (especially hesperidin in oranges) are antioxidant and antimutagenic, meaning that they prevent mutations in cells.
Improved Cardiovascular Health:
Hesperidin is a flavonoid that enhances Vitamin C’s effect of neutralizing free radicals in the body. In fact, orange pulp contains ten times the amount of Vitamin C that is found in the juice. Oranges also contain folate, which is one of the B Vitamins. Dietary folate is also important in maintaining normal DNA in the cell and preventing colon and cervical cancers. Folate is important in maintaining low levels of homocysteine in the blood, and this is important because high homocysteine levels increase the risk of cardiovascular disease. The risk of heart attack and stroke is inversely related to the amount of folate consumed.
Reduced Cancer Risk:
Limonene is a phytonutrient found in the oil of the peel of citrus foods, and this stimulates antioxidant detoxification, meaning limonene stops cancer even before it develops. Additionally, limonene reduces protein activity that may trigger abnormal cell growth. Citrus also contains Vitamin C, and this protects against nitrosamines, which may trigger cancers of the colon, mouth, and stomach
Stabilized Blood Sugar Levels:
Citrus contains pectin, which slows the absorption of glucose, helping to maintain steady glucose and insulin levels. This is important for diabetic individuals, because regular citrus consumption helps stabilize blood sugar levels.
How Do I Incorporate Oranges Into My Diet?
- Eat oranges, tangerines, or clementines
- Add mandarin orange to salads
- Use orange or lemon zest in cakes, muffins
- Use citrus zest in tea and other drinks
- Because Vitamin C is rapidly excreted from the body, regular consumption of citrus is essential.
PUMPKIN
Nutrients:
- Low calories, high fiber
- alpha- and beta-carotene
- Vitamins C and E
- Potassium, magnesium, and pantothenic acid
Nutritional Benefit:
Prevent Progression of Atherosclerosis:
Alpha- and Beta-carotene are major fighters against chronic disease because they have strong antioxidant and anti-inflammatory properties. Beta-carotene prevents oxidation of cholesterol, and this is important because it is oxidized cholesterol that builds up in blood vessels and contributes to the risk of heart attacks. Beta-carotene consumption may also reduce colon cancer risk because it protects cells of the colon from cancer-causing chemicals.
Protection From Cardiovascular Disease:
The cumulative effects of carotenoids, potassium, magnesium, and folate offer protection against cardiovascular disease and decrease the risk of cardiovascular events.
Slowed Biological Aging:
Alpha-carotene consumption is inversely related to biological aging. Alpha-carotene also protects against cataracts and various cancers.
How Do I Incorporate Pumpkin Into My Diet?
- Pumpkin is usually only available fresh in the fall and early winter.
- There are many winter squashes that are available in the market for most of the year that come close to pumpkin in terms of nutrition.
- Look for squash with its stem still on. Without the stem, bacteria can enter it.
- A deep and rich color indicates a ripe squash
- Pumpkin seeds are an excellent source of plant-based omega-6 and omega-3 fats
- Roast pumpkin seeds on a cookie sheet for 15-20 minutes at 350°F.
WILD SALMON
Nutrients:
- Omega-3 fatty acids
- B Vitamins and Vitamin D
- Selenium, Potassium, and Protein
Nutritional Benefits of Wild Salmon
Excellent Source of Omega-3 Fatty Acids:
There are four types of dietary fats: saturated, trans, monounsaturated, and polyunsaturated.
- Saturated fats increase your risk of diabetes, stroke, cardiovascular disease, and obesity.
- Trans fats may even be worse than saturated fats and are usually found on food labels as partially hydrogenated vegetable oil.
- Wild Salmon contains “good” fats, which are monounsaturated and polyunsaturated fats. Omega-3 and omega-6 are essential polyunsaturated fatty acids, and since our bodies do not make these, we must obtain them through out diets. Omega-6 fatty acids are very abundant in the Western diet in the form of corn, safflower, and sunflower oils. Omega-3 fatty acids come from plants and marine life. Salmon is an excellent source of marine-derived omega-3.
Omega-3 Fatty Acids:
- Reduce coronary artery disease risk by increasing HDL (“good”) cholesterol levels, reducing your blood pressure, and stabilizing your heart beat
- Prevent cancer – research suggests that omega-3 may prevent breast and colon cancers
- Mitigate autoimmune diseases such as lupus, and rheumatoid arthritis because omega-3’s anti-inflammatory capabilities help reduce symptoms of autoimmune diseases and prolong the lives of individuals who have them
SOY
Nutrients:
- Phytoestrogens
- Plant-derived omega-3 fatty acids
- Vitamin E
- Potassium, magnesium, selenium
- Folate
- Protein
Nutritional Benefits of Soy
Non-Meat Protein:
Soy is loaded with protein – a half-cup of tofu has 18 to 20 grams of protein, 258mg calcium, and 13mg of iron. Furthermore, soy has a good balance of various fats and does not contain any cholesterol, making it ideal an ideal source of protein and fiber. Additionally, soy provides the highest-quality protein of any plant food, provides all nine essential amino acids, and is a great source of omega-3 fatty acids. In comparison with other plant-based sources of protein, tofu is low in calories and high in protein.
Cancer Prevention:
Soy contains isoflavones, two of which are genistein and daidzein, which act as antioxidants and weak estrogens in the body that compete with natural estrogens to prevent hormone-dependent cancers such as breast cancer and prostate cancers.
Lignins:
These bind with carcinogens in the colon and facilitate their swift exit from the body, reducing negative effects
Protease Inhibitors:
These block the activity of cancer-causing enzymes called proteases. This reduces the risk of cancers.
Oil:
Soy provides oil that is cholesterol free and offers a good ratio of fatty acids, including omega-3 fatty acids.
How Do I Incorporate Soy Into My Diet?
- Mix tofu with vegetables and stir-fry
- Use tofu as a substitute for meat products when cooking
- Eat soybeans or toss them in a salad
- Ferment soy beans to make tempeh, miso or soy sauce
- Drink soymilk
- Eat edamame (green soybeans still in their pods)
- Soy protein powder
- Soy flour
SPINACH
Nutrients:
- Low calories
- Lutein/zeaxanthin
- Beta-carotene
- Plant-derived omega-3 fatty acids
- Vitamins C and E,
- B Vitamins
- Polyphenols, Minereals, alpha lipoic acid
Nutritional Benefits of Spinach:
Lowered homocysteine levels:
Betaine is a derivative of choline, which is an essential fat. Betaine lowers homocysteine levels in the blood, and this is important because high levels of homocysteine increase cardiovascular risk.
Protection From Age-related Macular Degeneration (AMD)
Spinach helps elevate macular pigment levels, and this lowers the risk of AMD. Free-radical damage from long-term exposure to light and UV radiation may play a role in causing macular degeneration. Lutein and zeaxanthin are two powerful carotenoids that can reduce AMD risk. Lutein and zeaxanthin also prevent other eye problems, such as cataract problems. Orange bell peppers are also very rich in lutein and zeaxanthin.
Vitamin K
Spinach is a rich source of Vitamin K, which is needed for production of six of the proteins needed for proper blood coagulation. Additionally, just one cup of spinach daily helps to reduce the risk of hip fracture in women.
Combat Cardiovascular Disease
The carotenoids in spinach protect the artery walls from damage. Spinach contains Vitamin C and beta-carotene, and these two nutrients work to prevent oxidized cholesterol from accumulating in the walls of your blood vessels. Spinach also has a lot of folate, which helps repair damaged DNA in cells, thereby aiding in cancer prevention.
Cancer Prevention:
Various flavonoid compounds in spinach work together to prevent cancer development. Glutathione and alpha lipoic acid are both found in spinach, and glutathione protects our DNA by repairing damaged DNA and promoting healthy cell replication. Alpha lipoic acid boosts glutathione levels and stabilizes blood sugar levels.
Improved Immune System:
Lutein is another antioxidant in spinach and enhances the immune system, warding off many cancers. Usually, the darker the greens, the more bioactive phytonutrients they contain and the more powerful they are against cancer and other diseases.
How Do I Incorporate Spinach Into My Diet?
- Put spinach leaves in salads
- Layer spinach and other green vegetables in lasagna
- Toss some spinach in pasta, soup, omelet
- Shed greens onto tacos and burritos
TEA
Nutrients:
- No calories
- Flavonoids, fluoride
Nutritional Benefits of Tea
Cancer Protection:
Caffeine from tea has anti-mutagenic properties, which may offer protection against cancer. Caffeine may also offer protection against the development of Parkinson’s disease. Other evidence suggests that tea consumption decreases the risk of bladder, breast, colorectal, esophageal, lung, prostate, and stomach cancers. Researchers demonstrated that catechins in tea prevent cell mutations and deactivate carcinogens. They also inhibit the growth of blood vessels that tumors need to grow. Although one cup of tea may offer health benefits, it may take up to four cups a day to achieve a significant decrease in cancer risk.
Cardiovascular Protection:
A study from Harvard showed a 44% reduced risk of heart attack and a 40% decrease in death from coronary artery disease in people who drank at least one cup of tea daily. Other evidence shows that tea consumption is associated with a decreased risk of heart disease and stroke. Tea consumption is inversely related to homocysteine levels, which is important because high homocysteine levels are correlated with increased cardiovascular risk. Tea also maintains plaque-free blood vessels, which reduces coronary artery disease.
Dental Health:
Tea consumption reduces the risk of developing cavities and gum disease; one study found that tea consumption may reduce cavity formation by up to 75% because the fluoride content of tea inhibits cavity formation. Additionally, tea inhibits bacteria from sticking to tooth surfaces, and this inhibits the rate of acid production of oral bacteria.
Improved Bone-Mineral Density:
Studies focusing on hip fracture risk discovered that tea consumption for 10+ years has benefits to bone-mineral density. This may be due to the flavonoids in tea, which have phytoestrogen activity that benefits bone health.
How Do I Incorporate Tea Into My Diet?
- Brewed tea yields more health benefits than does instant tea. Brew for at least 3 minutes
- Because flavonoids degrade with time, drink freshly brewed tea that is hot
- Squeeze the brewed tea bag to double the polyphenol content
- Add a wedge of lemon or lime with the rind for polyphenol boost
- Avoid drinking extremely hot tea
TOMATOES
Nutrients:
- Low calories
- Lycopene
- Alpha- and beta-carotene, lutein/zeaxanthin, biotin, fiber
- B vitamins, Vitamin C, potassium, chromium
- Phytuene and phytofluene possess antioxidant and anti-carcinogenic capabilities
Nutritional Benefits of Tomatoes:
Very Powerful Antioxidants:
Lycopene is a pigment that contributes to the red color of tomatoes and is a very strong antioxidant that is very efficient at quenching free radicals. Lycopene is also involved in the antioxidant defense network and helps raise the SPF of the skin, thereby protecting your skin from the sun’s damaging rays. Lycopene absorption depends on the presence of a bit of dietary fat, so tomatoes are often served with olive oil or cheese.
Cancer Protection:
Tomatoes have been shown to protect against cancer, specifically prostate cancer. Lycopene blocks the destructive effects of free radicals in the body and interferes with the growth factors that stimulate cancer cells to grow and proliferate.
One study found that tomato sauce consumption is the most reliable indicator of reduced risk for prostate cancer. This suggests that tomato sauce and paste may be more effective than raw tomatoes at reducing cancer risk; processed tomato products and cooked tomatoes contain 2-8 times the available lycopene of raw tomatoes. Processing tomatoes does reduce the Vitamin C levels, but it elevates total antioxidant activity, providing enhanced benefits.
Cardiovascular Protection:
Lycopene and other vitamins such as Vitamin C and beta-carotene, work to neutralize free radicals in the body that damage cells. This reduces the progression of atherosclerosis because it reduces the potential for inflammation. Tomatoes have high levels of potassium, niacin, vitamin B6, and folate, all of which are very heart-healthy nutrients. Potassium-rich foods help to achieve optimal blood pressure, and niacin lowers elevated blood cholesterol levels. The combination of folate and Vitamin B6 reduce homocysteine levels in the blood; this is important because high homocysteine levels increase cardiovascular disease risk.
How Do I Incorporate Tomatoes Into My Diet?
- Sauté tomatoes in olive oil and herbs and toss over pasta or serve as a side dish
- Add sun-dried tomatoes to sandwiches and salads
- Make homemade pizza with extra tomato sauce
- Add diced tomatoes to soups and stews
TURKEY (SKINLESS BREAST)
Nutrients:
- Low-fat protein
- Niacin, Vitamin B6, Vitamin B12
- Iron, Selenium, Zinc
Nutritional Benefits of Turkey
Low-Fat Protein:
Turkey breast is very low in saturated fat, so it closely approximates the lean sources of animal protein during Paleolithic times. The problem with commonly available meats, specifically red meat, in the United States is that they are very high in saturated fat and omega-6 fatty acids, which is not the type of fat we do need. Free-range and free-roam cattle are a better alternative to meat raised in feedlots because free-roaming cattle consume more omega-3 fats. Grass-fed meats contain omega-3 fats and Vitamin E.
Cardiovascular Health:
Turkey is a good source of niacin, Vitamin B6, and Vitamin B12. These vitamins are important for energy production because niacin is associated with lowered risk of heart attack and heart attack-associated mortality. Vitamins B6 and B12 help to keep homocysteine levels low, which is important because high homocysteine levels are associated with cardiovascular risk.
Immune Health:
Turkey is rich in zinc, and the zinc found in turkey is more bio-available than zinc found in non-meat sources. Zinc promotes wound healing and normal cell division. Turkey also has a lot of selenium, which is involved in a myriad of body functions, including thyroid hormone metabolism, antioxidant defense systems, and immune function. Evidence suggests that an inverse relationship exists between selenium intake and cancer risk.
How Do I Incorporate Turkey Into My Diet?
- Make a turkey dinner with a roasted whole fresh turkey breast
- Make a turkey sandwich for lunch on toasted whole grain bread
- Turkey tacos or burritos
- Turkey soup
- Turkey slices and BBQ sauce
WALNUTS
Nutrients:
- Plant-derived omega-3 fatty acids
- Vitamin E, Vitamin B6
- Magnesium, Potassium
- Polyphenols, plant sterols, protein, arginine
Nutritional Benefits of Walnuts:
Low Glycemic Index (GI)
Nuts have a low GI, so when you eat them, you feel satiated (full). This prevents overeating and helps you maintain a healthy weight. People who eat nuts in a balanced diet tend to be thinner than those who do not.
Cardiovascular Health:
Nut consumption is associated with a decreased risk of coronary artery disease. People who eat nuts five or more times a week had a 15-51% reduction in coronary heart disease risk, according to recent studies. This is mainly attributable to omega-3 fatty acids found in nuts; omega-3 fats thin the blood and help blood to freely flow throughout the body, preventing clot formation and preventing blood cells from sticking to vessel walls. Omega-3 fats also reduce hypertension, decrease cardiovascular disease risk and macular degeneration as well.
Blood Vessel Flexibility:
Arginine is an essential amino acid that is found in walnuts, and this helps keep the interior of blood vessels smooth and flexible to increase blood flow. This reduces blood pressure to relieve hypertension.
Decreased Diabetes Risk:
Harvard researchers studied 83,000 women and found that those who ate a handful of nuts or two tablespoons of peanut butter at least five times a week were more than 20% less likely to develop type II diabetes. The fiber and magnesium found in nuts maintain balanced insulin and glucose levels.
Fiber and Vitamin E:
Nuts are a rich source of dietary fiber and Vitamin E, which have strong anti-inflammatory properties, contributing to heart health
How Do I Incorporate Nuts Into My Diet?
- Add nuts to yogurt, ice cream, or frozen yogurt
- Add chopped nuts or pine nuts to salads
- Use finely chopped nuts to coat fish or poultry cutlets
- Sauté chopped nuts in olive oil along with bread crumbs and chopped garlic and toss with freshly cooked pasta
YOGURT
Nutrients:
- Live active culture
- Protein, calcium, potassium, magnesium, zinc
- Vitamin B2, Vitamin B12
Nutritional Benefits of Yogurt:
Prebiotics and Probiotics:
Prebiotics are non-digestible food ingredients that affect the gut by stimulating the growth or activity of beneficial bacteria in the colon. Probiotics are live microorganisms that can benefit your health when taken in small amounts.
Gastrointestinal Health:
A healthy digestive system is essential to good health, and probiotics help to preserve intestinal health. Probiotics absorb mutagens that cause cancer, and evidence suggests that they are effective in fighting colon and breast cancer. They stimulate the immune system by promoting immunoglobulin production and lower cancer risk by decreasing inflammation and preventing growth of cancer-causing intestinal microflora. Probiotics regulate the body’s inflammatory response, which relieves symptoms of inflammatory bowel disease (IBD).
Fish Oil
What is it?
Fish oil is the oil derived from the tissue of oily fish. Fish oil can be obtained from eating fish or by taking fish oil supplements.
Fish oil contains omega-3 fatty acid, docosahexaenoic acid (DHA) and eicosapentaenoic acid (EPA). Omega-3 may also be derived from a plant source containing alpha-linolenic acid (ALA).
This decreases the morbidity and mortality of heart disease by reducing the risk of coronary artery disease, high blood pressure, and fatal heart attacks.
Omega-3 fatty acids are not produced within the body, therefore are obtained from your diet or supplements.
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Why do I need it?
Multiple studies have shown that dietary fish or fish oil:
- Lowers triglycerides
- Lowers LDL (Lousy Cholesterol) levels
- Increases HDL (Healthy Cholesterol) levels
- Lowers blood pressure
- Reduces the risk of arrhythmias (dangerous abnormal heart rhythms)
- Reduces the progression of atherosclerosis (plaque build up in arteries)
- Reduces the risk of heart attacks
- Reduces inflammation
- Reduces the risk of stroke
- Reduces the risk of death
These are all beneficial in reducing the morbidity and mortality of heart disease which is the leading cause of death in the United States.
What are the sources?
Fish oil can be obtained by eating oily fish or fish oil supplements.
Recommended fish include:
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Plant derived sources of alpha linolenic acid are also recommended.
Plant sources of ALA include:
- Canola oil
- Flaxseed oil
- Soybeans
- Tofu
- Walnuts
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How much should I take?
The recommended dietary allowance is:
EPA + DHA: 0.3- 0.5 grams
Alpha linolenic acid: 0.8- 1.1 grams
Healthy adults with no cardiovascular disease are recommended to eat fish twice a week.
Please note that fish preparation is to be broiled or baked. Fried fish or fish sandwiches negate the benefits of fish oil and can actually increase the risk of heart disease.
Fish oil capsules are to be taken once daily.
Consult your physician before starting fish oil supplements.
What are the risks?
People with fish allergy should avoid fish oil and omega-3 products derived from fish.
People with allergy to nuts should avoid ALA and omega- 3 products derived from nuts.
Some predatory fish like sharks, cod, and tuna are high in omega- 3 fatty acids, but also have toxic substances. These toxic substances include mercury, dioxin, PCBs, and chlordane which are mostly found in cod liver and shark liver oils. The liver is a major detoxifying and filtering organ, therefore high levels of these toxins are found here. Liver based oils have a higher PCB content than fish oil derived from an entire fish.
Very large amounts of fish oil and omega-3 may increase the risk of hemorrhagic stroke.
Omega- 3 fatty acids may have the potential to slightly increase blood sugar levels. This should be taken with caution in diabetic patients, although no significant long term effects have been noted.
Patients with low blood pressure and those taking blood pressure medications are advised to take fish oils with caution.
Some patients may present with diarrhea at very high doses.
Increased burping, acid reflux, heartburn, abdominal pain, or bloating may occur.
Fishy aftertaste is common.
Updated March 12th, 2012 -JS
For further information please contact Cardiovascular Interventions, PA at 407-894-4880
SpectraCell Analysis Test
What Does the SpectraCell Analysis Test Measure?
The SpectraCell Analysis Test assesses the function of a range of vitamins, minerals, antioxidants, and other micronutrients in your blood cells. The SpectraCell test is currently the gold standard in evaluating nutritional status, and this technology is known as Functional Intracellular Analysis, or FIA™.
Why is the SpectraCell Test Important?
This preventive, diagnostic test is important because nutrient status is a very important component of your health. Every micronutrient plays an important role in optimal functioning of your cells. The SpectraCell Analysis Test will identify any micronutrient deficiencies you may have; correcting such deficiencies is essential in maintaining optimal health. This test, therefore, allows clinicians to develop appropriate interventions for patients wanting to maintain optimal health.
What Influences My Micronutrient Requirements?
There are many things influencing each individual’s micronutrient requirements. These include the following: biochemical individuality, absorption, chronic conditions, age, and lifestyle. Even people with healthy habits can have micronutrient deficiencies.
Who Benefits from This Test?
Everybody. You may be deficient in a micronutrient and not even know about it. Nearly 50% of patients taking a multivitamin are functionally deficiency in at least one essential nutrient that is vital to long-term health.
How Does the Test Work?
You will have your blood drawn (no need for fasting) and sent to the SpectraCell laboratory in California. The lymphocyte cells are isolated from the blood sample, grown in patented culture media, and stimulated to grow in a control media that has optimal amounts of specific micronutrients. As each micronutrient is removed from the culture media, your lymphocytes must use their own internal mechanisms (storage reserves or metabolic processes) to grow. If the cells grow in the absence of the micronutrient in the culture media, you are not deficient in that micronutrient. However, if the cells do not grow optimally, you are deficient in that micronutrient.
What Specific Micronutrients Does This Test Measure?
Vitamins: D, E, B1, B2, B3, B6, B12, Biotin, Folate, Pantothenate
Minerals: Calcium, Magnesium, Selenium, Zinc
Antioxidants: Coenzyme Q10, Glutathione, Cysteine, Total Antioxidant Function
Amino Acids: Asparagine, Carnitine, Glutamine, Serine
Does My Insurance Plan Pay For This Test?
Most insurance carriers cover the SpectraCell tests.
Call 407-894-4880 to make your appointment. Space is limited, so sign up today!
Updated February 16, 2009 - MJ
Alcohol and Heart Disease
Research has shown that the consumption of alcohol in moderation reduces the risk factors associated with coronary artery disease. A moderate amount of alcohol refers to an average of one to two drinks per day for men and one drink per day for women. Drinking alcohol in excess or consuming three of more drinks per day can lead to a variety of adverse effects including liver disease, heart failure, arrhythmias, stroke, increases the risk of cancer, neurological complications, and unintentional injuries. Excessive alcohol consumption and binge drinking attribute to the third leading lifestyle-related cause of death in the United States.
What are the benefits of alcohol on heart disease?

Drinking alcohol in moderation has been shown to improve cardiovascular health, particularly in regard to coronary artery disease and reducing the risk of a heart attack. Multiple studies have described a J-shaped curve relating the benefits of moderate alcohol intake to a reduction in the risk of morbidity and mortality of coronary artery disease. The lowest amount being about two drinks (20 grams) per day reduces the risk of coronary artery disease, while the consumption of three of more drinks (70 grams) is associated with a greater risk.
The benefits of moderate alcohol consumption include:
- Improvement of lipid profile:
- Increases High Density Lipoprotein Cholesterol (HDL-C) - this is the most notable benefit as this healthy cholesterol, HDL-C, removes cholesterol from the arterial wall and transports it back to the liver.
- Decreases Low Density Lipoprotein Cholesterol (LDL-C) - sometimes referred to as your Lousy cholesterol because LDL-C builds up in the arterial wall making them narrow.
- Improves Cholesterol (both HDL and LDL) Particle Size- improves HDL-P that reduces the risk of cardiovascular disease and reduces cardiovascular morbidity and mortality.
- Reduces thrombosis (clotting formation)
- Reduces platelet aggregation
- Reduces fibrinogen- this promotes blood clotting
- Increases fibrinolysis – this is a process by which clots dissolve
- Additional effects
- Reduces coronary artery spasm in response to stress- by relaxing the vessel
- Increases coronary blood flow
- Reduces blood pressure
- Reduces insulin level
- Increases estrogen level- this increases HDL levels and reduces LDL levels and reduces platelet aggregation
- Decreases C- Reactive Protein- marker of inflammation
- Antioxidant- flavonoids found in grapes (wine) reduce free radical damage in the heart and vessel
The effects of moderate alcohol consumption in the average study population has shown reduced risks of a new diagnosis of coronary artery disease by 29% and a risk reduction of deaths from cardiovascular disease by 25%. The risk of death due to a heart attack or coronary artery disease was reduced by 25%.
Should I consume alcohol with meals?
Drinking with meals has been found to reduce blood pressure and have a positive affect on lipids and the dissolution of thrombosis; although the relationship of moderate alcohol consumption has not been proven to reduce coronary artery disease.
Also, the presence of food in the gastrointestinal tract may reduce alcohol absorption or increase the rate of alcohol excretion from the body.
Does the type of beverage matter?
The type of beverage either wine, beer, or liquor does not matter. It is the alcohol which reduces the morbidity and mortality of cardiovascular disease. Although, red wine has an added affect due to the antioxidant properties.
Who should not consider alcohol consumption?
Excessive alcohol intake can lead to liver disease, heart failure, increased risk of cancer, neurological complications, and unintentional injuries.
Excessive alcohol consumption shows an inverse relation between alcohol and coronary artery disease that appears to increase LDL-C and lower HDL-C.
People should NOT start drinking if they already do not drink.
In addition, even moderate alcohol consumption should be avoided whenever consumption would put an individual People with medical and social conditions that are made worse by alcohol. These people should not consume any alcohol whatsoever including those with prior diagnosis of:
- Hypertriglyceridemia
- Pancreatitis
- Liver disease
- Porphyria
- Uncontrolled hypertension
- Congestive heart failure
Pregnant women and persons on medications that interact with alcohol should refrain from alcohol consumption. Also those with personal or strong family history of alcoholism should avoid all alcoholic beverages.
What are the recommendations?
Consult a physician about the benefits and risks of alcohol consumption personal to your health.
Those with prior diagnosis of hypertriglyceridemia, pancreatitis, liver disease, porphyria, uncontrolled hypertension, congestive heart failure or pregnant females or persons on certain medications that interact with alcohol should avoid alcohol consumption.
If your physician approves and there is no contraindication for alcohol consumption present, moderate consumption of alcohol (one or two drinks per day) may be considered safe.
Alcohol should never be consumed when operating heavy machinery or motor vehicles.
The risks and benefits of alcohol consumption should be reviewed periodically with your physician.
Adolescents and young persons are not recommended for alcohol consumption and should be targeted for assessment and advice before potentially harmful habits of consumption become established.
Alcohol affects the entire body, but has limited benefits, mainly on the coronary arteries. A careful approach must be warranted for alcohol consumption.
Each person has a unique combination of factors such as age, sex, and family history that influence a personal risk for specific diseases potentially caused or prevented by alcohol use. The balance of the risks and benefits of alcohol consumption for each person will be unique.
Effects of Smoking
What happens?
Smoking can cause serious health problems and in many cases, lead to death. About 4,000 chemicals are present in cigarettes with hundreds being poisonous. This causes damage to just about every aspect of the human body ranging from the inner lining of blood vessels to the outer lining of the skin surface. Damage does not only involve vessels and organs, but also reduces the functioning of organs and reduces the entire body’s immune system.
Over 443,000 deaths per year or one death every 8 seconds or the death toll of two planes full of passengers involved in a head on collision per day. Anyway you want to look at it; people are constantly dying from smoking. Often tobacco users are in denial and believe, ‘that won’t happen to me’, but chances are that it is highly likely to occur. More deaths are due to tobacco than deaths due to HIV, alcohol, illegal drug usage, suicides, car accidents, and murders combined.
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Healthy Lung |
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What is in a cigarette?
Cigarettes contain hundreds of toxins that are harmful to the body. Some of the most dangerous toxins include:
- Nicotine (addictive substance)
- Tar (cancer causing substance)
- Carbon monoxide (occupies the binding site oxygen normally takes)
- Cyanide present in cigarette smoke (deadly poison and cancer causing substance)
- Ammonia (cancer causing substance)
- Benzene (causes bone marrow depression)
What are the health risks of smoking?
Cardiovascular System
The leading cause of death in the United States due to smoking is surprisingly not lung caner, its heart disease.
Smoking takes its toll on the heart by narrowing the vessels supplying and leaving the heart. Smoking promotes the acceleration of arterial hardening and narrowing and the risk of clot formation. Narrowing of the vessels supplying the heart can precipitate a heart attack.
Smoking can narrow the arteries supplying the brain. A decrease or obstruction in blood flow to the brain due to blood clot can cause stroke, paralysis, and possible dementia.
Smoking can weaken the lining of the aorta, which is the largest blood vessel in the body. High pressures are exerted from the large volume of blood within the aorta. With the effects of smoking weakening the aorta, it is more prone to the development of an aneurysm or bulge. An aneurysm due to vessel weakening, can lead to rupture and massive internal bleeding.
Narrowing of the peripheral arteries in the limbs cause pain while walking, cold extremities, ulcers, and in severe cases gangrene. Treatment of gangrene is by amputation of the affected part.
The kidneys control the body’s blood pressure system. Smoking can even cause a decrease in blood supply to the kidneys resulting in elevated blood pressure. High blood pressure further increases the risk of all the above.
Respiratory System
Smoking is the most common cause of lung cancer. Smoking represents 90% of all lung cancer cases. If smoking were to be eliminated, lung cancer would become a rare disease.
Smoking also causes COPD which is a collective term for a group of conditions that restrict airflow and make breathing difficult. This is because smoking affects the function of the immune system and increases the risk of respiratory infections. Patients who are unable to breathe due to the effects of smoking are severely restricted even at rest. The quality of life is significantly reduced; therefore airways should be maintained and breathing should not be taken for granted.
Smoking can worsen asthma and counteract medications used for asthma by worsening the inflammation in the airways.
Cancer
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Carcinogens (cancer causing agents) in tobacco smoke damages the important genes that control the growth of cells, causing them to grow abnormally or reproduce too rapidly, causing cancer.
Cigarette smoke does its damage by oxidative stress that mutates DNA, promoting the development of cancer and chronic inflammation.
Smoking not only causes cancer in the lung, but also causes cancer at many other sites.
Smoking causes the following cancers:
- Cancer of the oral cavity
- Cancer of the bladder
- Cancer of the esophagus
- Cancer of the kidneys
- Cancer of the pancreas
- Cancer of the cervix
- Cancer of the larynx (voice box)
- Cancer of the pharynx
- Cancer of the stomach
Cancer cells have the tendency to spread and don’t always stay in one place. They may enter the lymphatic system (a network of nodes and vessels that help your body fight disease).
Cancer can also enter the bloodstream and spread to other parts of the body. When cancer spreads, the process is called metastasis. Lung cancer that spreads (metastasizes) often goes to the other lung or the liver, brain, or bones.
Reproductive System and Childhood
Smoking has an affect in reproduction. Smoking increases the risk of infertility in couples and erectile dysfunction in men.
Mothers who smoke during pregnancy are more likely to have a premature birth, stillbirth, or low birth weight babies. The nicotine mother’s smoke during pregnancy crosses the placenta, exposing the baby to nicotine. At birth, the baby will undergo the stress of the withdrawal effects from nicotine is at risk of sudden infant death syndrome. They may also suffer from learning and behavioral problems.
Children and adolescents mimic the actions of their parents. A child with a parent smoker is more likely to smoke than those without parent smokers.
Other Health Effects
Smoking affects the eye and increases the risk of macular degeneration and cataracts. The blood vessels of the eye are sensitive to smoke and can result in red, itchy eyes.
Smoking stains the teeth and gums. The gums are more prone to inflammation and disease causing the teeth to fall out.
Smoking increases the body’s pH levels making the mucosa of the oral and digestive tract susceptible to ulceration.
Smoking causes the skin to lose its elasticity due a reduction of vitamin A levels resulting in wrinkles and pale skin.
Passive Smoking
The people around you whom are exposed to second hand smoke are at higher health risks than you. The smoke burned off from the cigarette is unfiltered and toxic. Smokers indirectly put a loved one or friend at risk.
Infants and children exposed to second hand smoke are more prone to respiratory infections, asthma, and death.
Environmental Effects
Smoking not only affects humans, but the environment as well. Cigarette butts are tossed everywhere. Most smokers toss their cigarette butts out the car window or on any public grounds. A single smoker will have multiple cigarettes a day or even packs a day. The number of cigarette butts combined from all smokers adds up. Cigarette butts are turning up everywhere. The poisonous substances from cigarettes are accumulating in the Earths soil and damaging the ecosystem. Animals are also being exposed to these toxins.
Why do people smoke?
Most smokers are aware of the health risks associated with smoking, yet they continue to smoke. Smoking is addictive just as cocaine or heroin is addictive.
When a smoker inhales smoke, nicotine reaches the brain within 10-20 seconds. Nicotine stimulates a chemical transmitter in the brain called dopamine which provides a feeling of pleasure. When the nicotine levels drop, cravings develop. This cycle can make a person addicted to nicotine
.
Techniques to stop smoking:
The more education you have on the effects of smoking, the easier it will be to quit. Your health will start to improve the same day you decide to quit smoking, allowing the body to start the healing process. The arrest of smoking can add about 7 years to your lifespan, leading to a better quality of life. The earlier you quit the better. The quality of life will be poor in those who prolong smoking; therefore adding 7 years to your lifespan should be 7 healthy years.
Stopping smoking is the best thing you can do for your health, but it is a difficult task. When you are ready to stop smoking, you need to make a firm commitment. Challenges lie ahead, but the best was to overcome this is to fully believe you can quit and stay smoke-free.
A number of ways are available to assist you through the challenge, including:
- Nicotine replacement therapy (NRT) - products including nicotine gum, lozenge, skin patches, or nasal spray are available. These act by delivering nicotine to the body, but without the poisonous and carcinogenic chemicals from cigarettes. Although these are used as an aid for the cessation of smoking, you must gradually wean yourself off of the nicotine.
- Bupropion (Zyban) - a medication used to help the cessation of smoking. This acts by inhibiting the reuptake of dopamine from the nerves.
- Varenicline (Champix) - a medication used to help in the cessation of smoking. It acts by blocking the effects of nicotine on the brain.
- Behavioral modification programs – these programs help people stop the habit of smoking by stopping the cycle that triggers the urge to smoke. This is achieved by substituting unpleasant associations with pleasant associations and removing smoking ‘triggers’. Common triggers include anger, frustration, stress, hunger, boredom, loneliness, drinking or socializing.
- Alternative therapies- this includes acupuncture or hypnosis. Acupuncture is done by using the application of needles on pressure points to balance the flow of vital energy. Hypnosis is done by putting a person in a relaxed state of mind and suggesting them to quit smoking.
- Yoga and meditation – this can strengthen the mind and body giving you the will and drive to stop smoking permanently. Keeping a positive attitude helps you conquer negative habits.
The truth is that most people require several attempts to quit, but you are not alone. Many support groups or partners are available to you. Getting help from others significantly increases the chances for successfully quitting. A partner can be a family member, friend, or other quitters. Whoever you decide should be fair, understanding, trustworthy, and firm. Having someone to call when a ‘craving’ occurs gives you a chance to calm down and let the craving pass while letting your focus change.

Smoking requires honesty to yourself and your partner. If you are not being honest with your partner and do not tell them of a slip up, you are not utilizing their help. Hiding your cravings and slip ups can lead to a complete relapse. Increase your odds of success by getting the support you need to quit smoking permanently.
Smoking policies are made to encourage you to quit. This is used to reduce cigarette usage, eliminate second hand smoke, and break social norms issuing the acceptability of smoking. Indoor smoking policies are made to enforce an inconvenience to smokers which further encourages you to quit. Help is provided for you to quit smoking in a number of ways. Besides, smoking costs the United States about $96 billion dollars per year in direct medical costs.
Take action by throwing away all cigarettes from your home, car, or any other place you store them. Throw away matches, lighters, and ash trays from your settings. Keep smoking aids such as gums or patches with you at all time. When a craving or trigger occurs, try and keep your hands busy with something like filing your nails or by going out and exercising.
Tobacco is the single largest preventable cause of death and disease in the United States.
Vascular
- Abdominal Aortic Aneurysm
- Aortic Dissection
- Aortic Stenosis
- Deep Vein Thrombosis
- EECP
- Pulmonary Hypertension
- Renal Artery Stenosis
- T Wave Alternans
- Stroke
- Cardioversion
- Peripheral Vascular Disease
- Venous Insufficiency
- Venous Ablation
- Syncope
Abdominal Aortic Aneurysm
What is it?
An abdominal aortic aneurysm (AAA) is a permanent local dilatation or bulge in the aorta up to 1.5 times of its normal diameter.
The aorta is the largest vessel carrying blood from the heart to the rest of the body. The aorta leaves the heart and descends through the chest and abdomen until it splits into two arteries to supply the legs.
The aorta is an elastic structure which is capable of loosing its elasticity as it gets further away from the heart. Therefore the most common site for an aneurysm is in the abdomen. Most aneurysms have a spindle shape, but the size, shape, and extent varies in patients.
When there is a weakening in the walls of the aorta, the high pressures within the vessel cause a bulge or sac. Further weakening of this sac and enlargement can lead to a rupture of this major blood vessel causing severe bleeding. A ruptured aneurysm requires emergency management.
Blood tends to collect within these sacs which may lead to clot (thrombus) formation. If this dislodges from the sac, it can travel to other areas of the body and obstruct circulation to a particular area.
What are the causes?
Abdominal aortic aneurysm is a degenerative process of the aorta that is often attributed to atherosclerosis; however, the exact cause remains unknown.
Other causes of an abdominal aortic aneurysm include:
- Congenital causes: born with a genetic predisposition to a weakness in the artery walls
- Family History: AAA may have a tendency to run in families
- Inflammatory causes: arteritis (inflammation of the arteries)
- Infective causes: syphilis or fungal infections
- Trauma
What are the risk factors?
Risk factors of AAA share many of the same risk factors of atherosclerosis including:
- Sex: men are more commonly affected than women
- Age: ages 60-65 and increasing age
- Race: Caucasians have a higher risk than African Americans
- Smoking
- Family history of AAA
- High blood pressure (hypertension)
- High cholesterol
- Diabetes
Who needs to be screened?
Screening is done with an abdominal ultrasound used to view images of the aorta to detect any abnormalities.
Screening is recommended for the following groups of people:
- Men between the ages of 65 to75 with a history of smoking
- Men aged 60 and older with family history of AAA
- Both men and women with risk factors of AAA
Screening reduces the incidence of emergency repair and significantly reduced the mortality rate.
What are the symptoms?
Most abdominal aortic aneurysms are small and have no symptoms. Most people are unaware of it and are often discovered as an incidental finding during work-ups for other problems.
Symptoms are produced as aneurysms grow. The most common symptom of an abdominal aortic aneurysm is pain that may be felt in the abdomen, back, or groin.A sudden and very severe abdominal, back, or groin pain may indicate a rupture of the aneurysm. A ruptured aneurysm requires emergency management.
How is it diagnosed?
Diagnosis is based around 3 parameters. These parameters include: Patient History, Physical Examination, and Imaging tests.
Patient History:
Symptoms of pain in the abdomen, back, or groin
- Symptoms of claudication (pain while walking) in both of the legs may indicate an occlusion in the abdominal aorta
- A patient may complain of a swelling located in the abdomen
Physical Examination:
During a physial examination, your vitals will be taken. Vitals include your pulse, blood pressure, and temperature. High blood pressure is an indication for further work up.
An aneurysm may be felt in the abdomen as a small soft pulsating mass around the navel.
Peripheral pulses will also be checked to determine if an associated aneurysm or occlusive disease is present.
A stethoscope is placed over the abdomen to listen for a whooshing sound called a bruit.
Imaging tests
Imaging tests are performed to determine the size, location, and extent of an aneurysm. This also helps assess the risk of rupture and management.
- Abdominal Ultrasound- is a simple, non-invasive office procedure used to view images of the abdominal aorta.
- Magnetic Resonance Angiography (MRA) - this is a test similar to an MRI, which involves a contrast dye to be injected into a vein, while pictures are taken of the aorta.
Computerized Tomographic (CT) Angiography: this test also involves the injection of a dye into the vein while pictures of the aorta are taken.
- Angiography: This is an invasive test done in a hospital setting, involving the insertion of a thin catheter into the groin which is then advanced up to the aorta. A contrast dye is given to view the aorta and seen under x-ray guidance. This shows the location and extent of an aneurysm.
How is it treated?
Treatment includes preventing the progression of growth and preventing complications.
Treatment involves three separate parameters including: lifestyle changes, medications, and procedures.
Life style changes
These lifestyle changes help maintain a normal blood pressure and also lower the risk of progression of growth of an aneurysm.
- First line of treatment is to reduce the risk factors by diet changes, exercise, and weight management.
- Eat foods high in fiber such as whole grain cereals, oatmeal, and figs. Eat plenty of fruits such as apples, bananas, prunes, oranges, and pears. Include fish and legumes such as beans or chickpeas.
- Lower your salt intake
- Avoid fatty foods including bad saturated fat that is found in some meats, dairy products, chocolates, baked goods, and deep-fried food.
- Maintain a healthy weight with a BMI below 25
- No smoking
- Be more physically active
- Relax and reduce stress
Medications:
Medications are used to prevent the progression and complications plus close monitoring. This does not treat an aneurysm.
Some medications used are:
- Beta Blockers- to lower the blood pressure within the vessels.
- Cholesterol lowering medications- these reduce cholesterol levels in patients with atherosclerosis.
- Aspirin – prevents platelets from clumping together. This reduces the chances of a blood clot from forming and narrowing or obstructing the arteries. This is not appropriate for all patients including patients with bleeding disorders or those already taking a blood thinner.
Procedures:
Aneurysm repair is the primary treatment for symptomatic aneurysms and with a high risk of rupture.
The goal of surgical management is to treat the aneurysm before it ruptures.
Two types of surgical procedures may be done for a large abdominal aneurysm.These involve:
- Open surgery
- Endovascular repair
Open surgery- is a definitive treatment for symptomatic and high risk patients. This operation is done under general anesthesia and can last 4-6 hours. The abdomen is opened and a section of the aorta is removed. This section is replaced with a graft and the graft is covered by the artery wall and sutured into place.
Following this, normal blood flow is ensured. All surgeries carry a risk, although this holds a definitive treatment for aneurysm repair.
Endovascular repair- is a less invasive procedure which involves the placement of a stent within the abdominal aorta. A catheter is inserted into the groin through a small incision and advanced up to the aorta. A contrast dye is given to view the aorta and seen under x-ray guidance. A metal stent is positioned in the aorta which acts as a scaffolding for the artery.Although this is less invasive, this is not a definitive treatment.
Who is not eligible?
The following people may be contraindicated for an open surgical repair of an abdominal aortic aneurysm:
- Severe COPD
- Severe heart disease
- Active infection
- Severe co-morbidities including cancer or other end stage diseases
What are the risks of surgery vs. the risk of rupturing?
The risk of rupture of a small (<4 cm) aneurysm is less compared to a large (>6 cm) aneurysm. A small aneurysm if asymptomatic must be closely monitored every 6 months. The risk of rupture also depends on the rate of growth of the aneurysm.The average rate of growth of an aneurysm is about 0.3-0.4cm per year. A large aneurysm has an increase risk of rupture and grows faster in smokers.
The decision of surgical intervention depends on the associated risks as well as the surgical risks.Most small, asymptomatic, aneurysms do not require surgical intervention and only need monitoring every 6 months. Whereas patients who have an asymptomatic aneurysm close to 5.5 cm along with a rapid growth, are advised for repair.
Factors that need to be considered include:
- The size and rate of growth of the aneurysm
- Symptoms
- Multiple aneurysms present
- Surgical risk
What are the complications?
Complications of AAA include:
- Inflammation
- Infection
- Aorto-venous fistula- an abnormal connection between the aorta and a vein
- Aortoenteric fistula- an abnormal connection between the aorta and bowel
- Thromboembolism- dislodged clot with the potential to obstruct or narrow a vessel
*Rupture of an abdominal aortic aneurysm is a true medical emergency and can cause the following:
- Excruciating pain
- Severe hemorrhage (bleeding)
- Low blood pressure
- Shock
- Death
What is the prognosis?
Most abdominal aortic aneurysms grow slowly. If the aneurysm is slow growing, the prognosis is good.
Increasing size on an aneurysm increases the chance of rupture. The prognosis is good for patients who undergo aneurysm repair.However, a ruptured abdominal aortic aneurysm holds a bad prognosis. Less than 80% of patients survive a ruptured aneurysm of such a major vessel.
How can I prevent this?
Risk factor modification slows the rate of progression as well as prevents complications.Some ways to prevent these risk factors include:
- NO Smoking
- Controlling high blood pressure, cholesterol, and diabetes
- Eating a healthy diet
- Keeping physically active regularly with the right kind of exercise
- Maintain a healthy weight with a BMI <25
- Reduce stress!
Updated February 24th, 2012 -JS
For further information please contact Cardiovascular Interventions, PA at 407-894-4880
Aortic Dissection
What is it?
Aortic dissection is a condition in which a tear develops in the inner layer of the aorta.
The aorta is the largest and main blood vessel branching off the heart. Blood flows through this tear into the middle layer of the aorta, causing the inner and middle layer to separate or dissect.
This weakening in the wall of the aorta is prone to rupture which is often fatal.

What are the causes?
The most common cause of aortic dissection is due to chronic or long standing hypertension (high blood pressure).
Chronic hypertension causes stress in the aortic vessel wall making it more susceptible to tearing.
Other causes include:
- Bicuspid aortic valve
- Marfan’s syndrome – connective tissue disease
- Cystic medial necrosis - connective tissue disease
- Ehlers- Danlos syndrome – collagen synthesis defect
- Turner syndrome – chromosomal abnormality
- Coarctation of the aorta – narrowing of the aorta
- Syphilis – infection
- Arteritis – inflammation of artery
- Pregnancy – III trimester
- Cocaine abuse
- Traumatic injury to the chest – motor vehicle accidents in which the chest hits the steering wheel
How does it occur?
Deterioration of the inner layer of the aorta is said to be a prerequisite in the development of non-traumatic aortic dissection. Following deterioration, tearing of the inner layer or aortic intima may occur.
Blood passes into the middle layer or aortic media through the tear, separating the intima from the media and dissects the blood vessel.
The dissection can spread both proximal (before) and distal (after) to the tear, involving major branching vessels of the aorta and aortic valve and filling blood around the heart.
Spread of this nature is responsible for many of the symptoms including ischemia (decrease in blood supply) to vital organs, aortic regurgitation, and cardiac tamponade.
Who is at risk?
Those at potential risk of developing aortic dissection include:
- Sex: men are at higher risk compared to woman
- Age: ages between 60s and 70s
- Uncontrolled hypertension
- Atherosclerosis
- Pre-existing aortic aneurysm
- Bicuspid aortic valve
- Aortic coarctation
- Turner’s syndrome
- Marfan syndrome
- Loeys-Dietz syndrome
What are the symptoms?
Symptoms if aortic dissection includes:
- Sudden severe chest pain in chest or upper back, described as tearing, ripping, or shearing
- Loss of consciousness
- Shortness of breath
- Profuse sweating
- Sudden difficulty in speech, vision, or paralysis of half of the body
- Confusion or disorientation
- Weak pulse compared to other arm
- Dizziness
- Nausea and vomiting
What are the types?
Aortic dissection is classified by the site of dissection.
The most widely used
classification is the Stanford system which classifies dissection into two types.
Type A – dissections involving the ascending aorta, regardless of the site of tear
Type B – all other dissections
What are the complications?
Complications of aortic dissection include:
- Death due to massive internal bleeding
- Organ damage
- Insufficient circulation past the area of dissection
- Thrombosis
- Heart attack
- Stroke
- Cardiac tamponade
- Aortic regurgitation
How is it diagnosed?
The diagnosis of aortic dissection is based on three parameters including patient history, physical examination, and tests.
1. Patient history
-
Sudden severe tearing or ripping pain in the chest or back
-
Disorientation or confusion
-
Profuse sweating
2. Physical Examination
-
Delay in pulse between arms and legs
-
Hypertension or hypotension
-
Blood pressure difference between right and left arms
-
Auscultation over the chest may reveal a “blowing” murmur
-
New stroke symptoms and signs like decreased sensation or movement
3. Tests
-
Chest X-ray: reveals widening of the mediastinum

-
Transesophageal echocardiogram (TEE) – a test that uses high pitched sounds to produce images of the heart by passing a probe down the esophagus to view the heart
-
Computerized tomography (CT) scan: injection of a contrast dye is used to visualize the aorta
-
Magnetic resonance angiogram (MRA) – uses magnetic fields to form images of the body
How is it treated?
Aortic dissections are treated according to the type of dissection presented.
Type A
Type A aortic dissections are considered surgical emergencies. This is the more common and dangerous type of dissection. Surgery is preferred in this type of dissection. 
During the surgical procedure, surgeons remove as much of the dissected aorta as possible and make a blockade so blood does not enter into the aortic wall any longer.
The aorta is then reconstructed with a synthetic tube using a graft.
Some patients may have their aortic valve replaced at the same time if a leaky valve is present.
Type B
Type B aortic dissections may be treated medically unless the patient demonstrates progressive dissection with ischemia (decrease blood supply) or continued bleeding into the lung or abdominal space.
Medical treatment is used to reduce symptoms and prevent progression of the disease.
Medications include beta blockers (i.e. esmolol; labetalol; metoprolol) to reduce the action of adrenaline on the heart and blood vessels, vasodilators (i.e. sodium nitroprusside) to reduce the heart rate and blood pressure and nitrates (i.e. nitroglycerin) to dilate the blood vessels to decrease blood pressure.
Surgical repair is curative and is similar to the surgical procedure for Type A.
Sometimes a small wire mesh stent is inserted which acts as a scaffolding for the aorta.
What is the prognosis?
Aortic dissection is life threatening. The condition can be managed with surgery if it is done before the aorta ruptures. Less than half of the patients with ruptured aorta survive.
For Type A aortic dissection, the mortality rate remains high, with up to 30% deaths after surgery.
For Type B, when treated medically, the mortality rate is less with a death rate of 10%.
In both types, the ten year survival rate is more than 60%.
How can I prevent this?
The most important way to prevent aortic dissection is to control your blood pressure.
Prevention is enforced in patients who are at risk of dissection.
Some ways to reduce your risk is by:
- Controlling blood pressure, diabetes, and cholesterol

- No smoking
- Maintain a healthy weight with a BMI <25
- Wear a seat belt
- Continue taking the medications prescribed to you
- Any chest pain should not be ignored and medical care should be given immediately by emergency medical services and calling 9-1-1
Updated April 23, 2012 -JS
For further information please contact Cardiovascular Interventions, PA at 407-894-4880
Aortic Stenosis
What is it?
Aortic stenosis is a condition in which the aortic valve narrows and prevents the valve from completely opening. This restricts blood flow out of the aorta and heart. Restriction of blood flow exiting the aorta diminishes the body’s potential to receive the maximum amount of oxygen rich blood.
Over time the pressure increases in the left ventricle forcing the left ventricular wall to work harder and eventually become thicker and weaker. Weakening of the heart muscle can lead to the development of symptoms including weakness and fatigue.

What are the causes?
Narrowing of the aortic valve may be caused by the following:
- Congenital heart defects: unicuspid or bicuspid aortic valve
- Calcification
- Rheumatic fever- a condition that may develop after strep throat with valvular disease developing 5-10 years after rheumatic fever occurs
- Type II hyperlipoproteinemia
Rare causes include:
- Fabry’s disease
- Systemic lupus erythematosis (SLE)
- Paget disease
- Alkaptonuria
How does a normal heart work?
The heart consists of four chambers. The top two chambers (atria) receive blood while the bottom two chambers (ventricles) pump blood out.
Deoxygenated venous blood returns to the right atrium of the heart. Blood flows from the right atrium to the right ventricle by the opening of the tricuspid valve. Blood is prevented from going backwards into the atrium by the closure of the tricuspid valve. The right ventricle then contracts and pumps blood out through the pulmonary valve into the pulmonary artery. The pulmonary valve is closed and the blood is sent to the lungs where it picks up oxygen.
Oxygenated blood returns from the lungs by the pulmonary veins and enters the left atrium. From the left atrium, blood enters the left ventricle by the opening of the mitral valve. Blood is prevented from flowing back into the left atrium by the closure of the mitral valve. Blood is then pumped out the left ventricle to the aorta by the opening of the aortic valve. Blood is prevented from flowing back into the left ventricle by the closure of the aortic valve. From the aorta, oxygenated blood is pumped out to the entire body.


What happens in aortic stenosis?
The aortic valve consists of three leaflets (cusps) which connect to the aorta by a ring called the annulus. Heart valves only open in one direction. Once all of the blood has emptied out of the left ventricle, the aortic valve tightly closes to ensure no backflow of blood.
In aortic stenosis, the leaflets become thickened or calcified preventing the valve to completely open. The thickening of the leaflets make the valve opening narrowed. The ejection of blood from the left ventricle struggles to get through the narrowed opening. The complete emptying of oxygenated blood from the left ventricle is limited, increasing pressure in the ventricle and eventually thickens the wall of the left ventricle.
The limiting of blood flow into the aorta results in symptoms of breathlessness, dizziness, and loss of consciousness.


What are the risk factors?
Aortic stenosis is not considered a preventable disease, yet some risk factors include:
- Congenital abnormalities: bicuspid aortic valve (born with two aortic leaflets)
- Age: increasing age along with calcium deposits on valves
- History of rheumatic fever: causes leaflets to thicken, stiffen, or fuse
- Hypertension (high blood pressure)
- Hypercholesterolemia (high cholesterol)
- Diabetes (high glucose)
What are the symptoms?
Patients with aortic stenosis may have no symptoms at all.
Aortic stenosis ranges from mild to severe. Symptoms typically develop in severe aortic stenosis.
Symptoms include:
- Shortness of breath with activity
- Angina (chest pain)
- Fainting, weakness, or dizziness
- Fatigue
- Palpitations (sensation of feeling a rapid heart beat)
How is it classified?
Aortic stenosis is classified as:
- Mild- valve area 1.1-1.9cm2 and/or transvalvular gradient <30 mmHg
- Moderate- valve area 0.8- 1.1cm2 and/or transvalvular gradient 30-50 mmHg
- Severe- valve area 0.8- 1cm2 and/or transvalvular gradient >40 mmHg
Normal valve area: 3.0- 4.0cm2 with no transvalvular gradient
What are the complications?
Complications of aortic stenosis include:
- Angina
- Syncope
- Left ventricular hypertrophy
- Heart failure
- Arrhythmias
- Endocarditis
- Cardiac arrest
Call your doctor immediately if you have any of the following:
-
Chest pain or shortness of breath
-
Weakness in the muscles of your face, arms, or legs
-
Difficulty in speaking
-
Rapid or bounding heartbeat
-
Fainting or dizziness
How is it diagnosed?
Aortic stenosis is usually diagnosed during a routine visit with your physician.
Diagnosis is based around three parameters including patient history, physical examination, and tests.
1. Patient history
Patients may complain of symptoms in severe aortic stenosis.
Symptoms of:
- Angina
- Dizziness
- Syncope
- Loss of breath
2. Physical examination
During a routine physical examination, your health care provider will notice the following:
- Faint pulse may be felt
- Changes in the quality of pulse in the neck
- During auscultation a murmur may be heard
3. Tests
The following tests may be performed:
- Electrocardiogram (EKG) - a noninvasive test performed in our clinic which determines the electrical activity of your heart. This test may be used to detect left ventricular hypertrophy (thickening) which may occur due to aortic stenosis.
Chest X-ray- a simple test performed in our office showing the size and shape of the heart to determine whether the left ventricle is enlarged. Also reveals the presence of fluid in the lungs which may occur due to aortic stenosis.
- Echocardiogram (ECHO)- a noninvasive test using sound waves to determine the size and function of the heart’s chambers and the structure and function of the heart valves.
- Cardiac catheterization- a hospital procedure used to diagnose the type and severity of your heart condition. A thin tube (catheter) is inserted into an artery in your arm or groin to reach the heart. A contrast dye will be given to view the arteries and seen under x-ray guidance. This allows the physician to see any abnormalities in the arteries including blockage that may coexist with aortic stenosis.
How is it treated?
Lifestyle changes are implemented in patients diagnosed with aortic stenosis to stop the progression of the disease.
Treatment of aortic stenosis is primarily done with surgical intervention, although medications can be used to relieve symptoms of aortic stenosis.
Lifestyle modifications
Lifestyle modifications help reduce the work load on the heart.
Lifestyle modifications include:
- Maintain a healthy weight with a BMI <25
- Limit salt intake
- Maintain cholesterol, sugar, and blood pressure
- Exercise for at least 30 minutes a day 5 days a week
- Stay physically active
- Do not smoke!
Medications
Medications are used to reduce symptoms and control heart rhythm disturbances associated with aortic stenosis.
Medications may be used to lower blood pressure or cholesterol.
- Cholesterol lowering medications- these act by decreasing the amount of cholesterol in blood, especially Lowering your low density lipoprotein (LDL- your Lousy cholesterol) and keep your high density lipoprotein (HDL- your Healthy cholesterol) High.
- Beta blockers- this lowers the heart rate and decreases blood pressure.
- Diuretics- these are sometimes referred to as ‘water pills’ which helps excrete excess water.
- Antibiotics- people who had rheumatic fever in the past may require long-term treatment with penicillin.
Surgical Procedures
Surgical valve repair or valve replacement is preferred in patients who develop symptoms; even if symptoms are not bad, surgery is still recommended.
Aortic valve replacement offers substantial improvement of symptoms and life expectancy. The only effective treatment for severe aortic stenosis is aortic valve replacement.
Therapies to repair or replace the aortic valve include:
- Balloon valvuloplasty- in a hospital setting, the physician inserts a thin tube (catheter) tipped with a balloon into an artery in the leg or arm to reach the heart. The catheter is positioned into the narrowed aortic valve and the balloon-tipped catheter is inflated. The balloon pushes the aortic valve open and stretches the valve opening, improving blood flow. Balloon valvuloplasty may relieve aortic valve stenosis and symptoms, although re-stenosis of the valve is common.
- Aortic valve replacement- is the primary treatment for severe aortic stenosis. The entire aortic valve will be replaced by a surgeon. A mechanical (metal) or tissue valve (organic) may be used to replace the stenotic valve. A mechanical valve requires life-long anticoagulation therapy such as warfarin (Coumadin) to prevent blood clot formation. Tissue valves usually come from a pig or cow. Blood does not clot easily on tissue valves so patients may only need Coumadin or aspirin for only a short time. Tissue valves may wear out faster than mechanical valves and may need to be replaced sooner.
-
Transcatheter aortic valve implantation (TAVI) - this is a less invasive approach for aortic valve replacement with a prosthetic valve. The approach can be transfemoral (femoral artery in leg) or transapical (apex of heart). TAVI approach is reserved for patients who are at an increased risk of complication from aortic valve surgery.
What is the prognosis?
Without treatment, an individual with aortic stenosis with symptoms or complications may do poorly.
Aortic stenosis can be cured with surgery although there is a risk for arrhythmias (irregular heart rhythm) which may cause sudden death. There is also a risk that the new valve will stop working and may need to be replaced.
How can I prevent this?
Aortic stenosis itself often can not be prevented, although some of the complications may be prevented.
- Prevent rheumatic fever: See a physician when you have a sore throat. Treating strep infections promptly with antibiotics can prevent rheumatic fever that causes aortic stenosis.
Reduce risk factors of coronary artery disease: Lower blood pressure, obesity, high cholesterol, and sugar. It is a good idea to maintain a healthy weight with a BMI below 25.
- Practice good oral hygiene: gum infections may also cause inflammation of the heart tissue called endocarditis.
Updated May 1, 2012 -JS
For further information please contact Cardiovascular Interventions, PA at 407-894-4880
Deep Vein Thrombosis
What is deep venous thrombosis?
Deep venous thrombosis (DVT) affects mainly the veins in the lower leg and the thigh. It involves the formation of a clot (thrombus) in the larger veins of the area. This thrombus may interfere with circulation of the area, and it may break off and travel through the blood stream (embolize). The embolus thus created can lodge in the brain, lungs, heart, or other area, causing severe damage to that organ.Risks include prolonged sitting, bedrest, or immobilization; recent surgery or trauma, especially hip surgery, gynecological surgery, heart surgery, or fractures; childbirth within the last 6 months; obesity; and the use of medications such as estrogen and birth control pills. Risks also include a history of polycythemia vera, malignant tumor, changes in the levels of blood clotting factors making the blood more likely to clot, disseminated intravascular coagulation (DIC), and dysfibrinogenia.
Deep venous thrombosis occurs in approximately 2 out of 1,000 people. The condition is most commonly seen in adults over age 60.
Prevention:
Anticoagulants may be prescribed as a preventive measure for high-risk people. Minimize immobility of the legs.
Symptoms:
- leg pain in only one leg
- leg tenderness in only one leg
- swelling of only one leg
- increased warmth of one leg
- changes in skin color of one leg, redness or bluish
- joint pain
Signs and Tests:
An examination may reveal a red, swollen, tender area of the leg. The Homans sign is positive, there is sharp pain when the foot is flexed upward.
The presence of deep venous thrombosis may be seen on:
- venography of the legs
- extremity arteriography
- blood flow studies
- Doppler ultrasound exam of an extremity
- plethysmography of the legs
Treatment:
The clot itself usually will resolve through the natural healing processes. Treatment is also aimed at relieving symptoms and preventing the clot from traveling to the lungs, heart, brain, or other areas. Treatment usually requires hospitalization, at least initially.Anticoagulants or antiplatelet medications are prescribed to prevent further clotting. Analgesics may be needed to control pain. Thrombolytics (clot dissolving medications) are rarely needed.
Bedrest may be recommended until the symptoms are relieved. The leg may be elevated to reduce swelling. Avoid prolonged sitting. Warm, moist heat to the area may help relieve pain.
After returning home, the patient may continue oral anticoagulants or antiplatelet medications for a prolonged period of time. Warm compresses may also be continued. Continue to avoid prolonged sitting or standing in one position.
Expectations:
Most DVT's disappear without difficulty. Complications may be life threatening.
Complications:
- pulmonary embolus
- stroke (rare)
- embolus in other organs (rare)
It is recommended that you call your health care provider if symptoms suggestive of DVT occur. With proper attention and care, a person with DVT can still live a long and productive life.
Updated January 29, 2009 - MJ
Enhanced External Counterpulsation (EECP)
What is EECP?
EECP is a non-invasive, FDA approved, outpatient therapy for patients having coronary artery disease with persistent symptoms of angina or heart failure who have already had the standard treatments for revascularization or those who are not eligible for surgical intervention.EECP stimulates the formation of collaterals to help create a natural bypass around narrowed or blocked arteries to improve coronary perfusion.
This increases the amount of oxygen rich blood to the heart and reduces the symptoms of chest pain, shortness of breath, and fatigue.
Who needs EECP?
- Patients with coronary artery disease who do not require surgical intervention.
- A patient with chronic angina and medical therapy alone does not provide satisfactory relief.
- Patients who have already undergone one or more invasive procedures who have persistent chest pain.
- Patients who are seeking to lower the requirements for medications.
What are the benefits of EECP?
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Lowers the requirement of medications.
- Decrease the onset and frequency of chest pain.
- Enhance the quality of life and the ability to return to daily activities.
How does EECP work?
EECP affects the dynamics of cardiovascular blood flow. A series of inflatable cuffs sequenced from the calves, thighs, and hips rapidly inflate and deflate.The timing of the inflation will be during the exact resting phase of each of your heartbeats. This timed compression in the lower extremities pushes the arterial blood backwards into the aorta and increases the coronary perfusion pressure causing stress in the coronary arteries facilitating the development of collaterals.
EECP also increases the venous return during the sequential compression of the lower extremities with a “milking” effect at the same time as the arteries, which further increases cardiac output.
Since collaterals take time to develop, EECP is given for about 1 hour a day, for 7 weeks, with a total of 35 sessions. 80% of patients usually notice improvement of symptoms with this therapy.
Patients may be ordered a nuclear stress test after the course of therapy is complete to confirm the improvement of coronary blood flow.
Can anybody with chest pain try this?
Patients who do not qualify for EECP include patients with:
-
Uncontrolled congestive heart failure
- Uncontrolled arrhythmia
- Severe pulmonary or systemic hypertension
- Mild to moderate aortic insufficiency
- Significant coagulopathy- patients on heparin or warfarin
- Severe peripheral vascular disease
- Thromboplebitis- Inflammation in the veins due to a clot
What are the risks of EECP?
EECP is a non-invasive procedure and risks are very low. Although risks are rare, patients may feel discomfort or minor pain in their legs or back.Other effects may include bruising, blistering, or skin abrasions from the inflatable cuffs.
Updated January 17th, 2012-JS
For further information please contact Cardiovascular Interventions, PA at 407-894-4880
Pulmonary Hypertension
What is it?
Pulmonary hypertension is an increase in blood pressure within the arteries of the lungs and the right side of the heart.
When the blood vessels of the lungs become narrowed, pressure builds up in these arteries, therefore called pulmonary hypertension (high blood pressure).
When the heart begins to work harder, over time the right side of the heart becomes enlarged leading to right heart failure or cor pulmonale.
What are the causes?
Pulmonary hypertension may be caused by the following:
- Autoimmune diseases i.e. scleroderma, rheumatoid arthritis, collagen vascular disease
- Familial
- Congenital heart defects
- Pulmonary embolism
- Congestive heart failure
- Heart valve disease
- Hepatic cirrhosis/ portal disease
- Infection i.e. HIV
- Chronic hypoxia i.e COPD, pulmonary fibrosis, interstitial lung disease, obstructive sleep apnea, chronic exposure to high altitudes
- Medications i.e. diet pills
- Idiopathic (unknown)
What are the risk factors?
The risk factors for pulmonary hypertension include:
- Age: older patients are more likely to have secondary hypertension, such as pulmonary hypertension
- Sex: more common in women than men
- Family history: genetic inheritance
- Weight: overweight persons are at higher risk of developing pulmonary hypertension
- Other diseases: heart, lung, or liver disease
- Other conditions: HIV infection, blood clots, sickle cell disease
- Drugs i.e. cocaine or diet pills
- Those who live at high altitudes
What are the symptoms?
Symptoms of pulmonary hypertension include:
- Shortness of breath or light-headedness during activity
- Fast heart rate (palpitations)
- Swelling of the ankles or legs
- Cyanosis (bluish discoloration of the skin)
- Dizziness or syncope
- Fatigue or weakness
- Chest pain
- Upper right quadrant abdominal pain
How is it classified?
Pulmonary hypertension can be classified into:
- Idiopathic Pulmonary Hypertension (IPH)
Unknown cause of high blood pressure in the lungs; Uncommon
- Secondary Pulmonary Hypertension
High pulmonary pressure due to other medical problems; Common
Causes include:
- Pulmonary Embolism
- COPD
- Pulmonary fibrosis
- Connective tissue disorders
- Congenital heart defects
- Sickle cell anemia
- AIDS
- Cirrhosis
- High altitude
- Drugs including cocaine, diet pills
What are the complications?
Pulmonary hypertension can lead to complications including:
- Cor pulmonale (right heart failure)- The right ventricle has to work harder therefore it becomes thickened and enlarged. Eventually the right side of the heart gives out leading to right heart failure.
- Pulmonary embolism- dislodgement of a blood clot to the lung
- Arrhythmia
- Hemoptysis- coughing up of blood
Call your doctor immediately if you have any of the following:
Call your doctor if you develop symptoms such as:
- Shortness of breath during physical activity
- Fatigue
- Chest pain
- Or other symptoms
How is it diagnosed?
Pulmonary hypertension is diagnosed in three different parameters including patient history, physical examination, and tests.
Patient history
Patients may complain of symptoms in severe pulmonary hypertension. Including symptoms of:
- Shortness of breath
- Angina (chest pain)
- Fatigue/ tiredness
- Upper right quadrant abdominal pain
- Palpitations (racing heart beat)
Physical examination
During a routine physical examination, your health care provider will notice the following:
- Cyanosis- bluish discoloration of the skin
- Weak pulse may be felt
- Distended neck veins
- During auscultation a murmur may be heard
Tests
The following tests may be performed:
- Blood tests – complete blood counts, liver function tests, PT/INR.
- Electrocardiogram (EKG) - a noninvasive test performed in our clinic which determines the electrical activity of your heart. This test may be used to detect whether your heart’s rhythm is steady or irregular. Right ventricle enlargement can also be detected.
- Chest X-ray- a simple test performed in our office showing the size and shape of the heart to determine whether the right ventricle is enlarged.

- Echocardiogram (ECHO)- a noninvasive test using sound waves to determine the size and function of the heart’s chambers and the structure and function of the heart valves. This test can estimate the pressure in the pulmonary arteries.
- Right heart catheterization- a hospital procedure used to measure the pressure in the pulmonary arteries. A thin tube (catheter) is inserted into an artery in your arm or groin to reach the right side of the heart to the pulmonary arteries.
- Chest Computed Tomography (CT) - shows pictures of the heart, lungs, and blood vessels.
- Magnetic Resonance Imaging (MRI) - shows how the right ventricle of the heart is working as well as the blood flow to the lungs.
- Pulmonary Function Tests (PTFs) – are used to measure the amount, rate, and quality of air you can breathe in and out. This helps to determine any lung disease that may be causing pulmonary hypertension.
How is it treated?
The treatment of pulmonary hypertension is aimed at early recognition, controlling symptoms, treatment of the underlying disease, and the prevention of more lung damage.
Treatment depends on what type of pulmonary hypertension is present and the severity.
Lifestyle modification:
-
Rest
-
No smoking
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Exercise: ask your physician which exercise routine is best for you. Exercise for at least 30 minutes a day, 5 days a week. This can improve symptoms of shortness of breath and fatigue
-
Avoid pregnancy and birth control pills
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Avoid traveling at high altitudes
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Control weight: maintain a healthy weight with a BMI less than 25. Extra weight makes the heart work harder. By reducing your weight you can reduce the strain on your heart.
-
Nutrition: Eat foods high in fiber such as whole grain cereals, oatmeal, and figs. Eat plenty of fruits such as apples, bananas, prunes, oranges, and pears. Include fish and legumes such as beans or chickpeas
-
Avoid fatty foods including bad saturated fat that is found in some meats, dairy products, chocolates, baked goods, and deep-fried food
-
Reduce stress
Medications:
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Vasodilators: Vasodilators dilate the blood vessels. Epoprostenol (Flolan) is the most commonly prescribed vasodilator for pulmonary hypertension. Another medication that can be prescribed is Iloprost (Ventavis), which can be inhaled and taken directly to the lungs.
-
Endothelin receptor antagonist: These medications reverse the effect of endothelin (vasoconstrictor) which makes vessels narrower. Bosentan (Tracleer) helps to improve energy and symptoms.
-
Sildenafil (Viagra) and Tadalafil (Cialis): These medications are used to treat pulmonary hypertension by opening up the blood vessels in the lungs to allow more blood flow.
-
Calcium channel blockers: Calcium channel blockers may be given in high dosage to help relax the muscles surrounding the arteries and cause the vessels to open. This increases blood flow to the lungs and lowers blood pressure. Calcium channel blockers include amlodipine (Norvasc), diltiazem (Cardizem), and nifedipine (Adalat, Procardia)
-
Ambrisenten (Lentairis): helps dilate the blood vessels
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Anticoagulants: help prevent clot formation
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Diuretics: these are sometimes referred to as ‘water pills’ that is used for patients with leg swelling or fluid in the lungs. This helps excrete excess water in the body
-
Oxygen: breathing in pure oxygen can help treat pulmonary hypertension
Surgical Procedures:
Surgery is required if medications are unable to control your pulmonary hypertension.
- Atrial septostomy: an opening is created between the left and right atriums of the heart to relieve the pressure from the right side of the heart.
- Lung transplant: is a surgery to replace a diseased lung with a healthy lung from a donor. This is reserved for patients with severe lung disease causing pulmonary hypertension.
- Heart-lung transplant: both the heart and lung are replaced with healthy organs from a donor.
What is the prognosis?
The long term outlook for pulmonary hypertension is poor, yet new treatments may lead to better results.
Death can result in patients with associated heart failure.
Pregnancy is not encouraged for patients with this condition.
Updated July 19, 2012 -JS
For further information please contact Cardiovascular Interventions, PA at 407-894-4880
Renal Artery Stenosis
What is it?
Renal artery stenosis is the narrowing of one or both of the arteries that carry blood to the kidneys.
This may cause high blood pressure and reduced kidney function.

What are the causes?
The most common cause of renal artery stenosis is atherosclerosis due to the formation of fatty plaques on the inner lining of the arteries.
- Fibromuscular dysplasia (narrowing due to thickening of internal wall of blood vessel) tends to run in families and is seen more in women below the age of 50. This usually involves the distal portion of the renal artery.
- Inflammation of the arteries known as arteritis may cause narrowing.
How does it occur?
A blockage of 50-70% of the renal artery is considered significant. As a result of decreased blood flow to the kidneys, the kidney cells falsely presume that blood flow throughout the entire body is reduced.
As a normal defense mechanism, the kidney activates a hormonal system known as the renin-angiotensin system. This response expands blood volume and elevates blood pressure throughout the entire body.
What are the risk factors?
Renal artery stenosis shares the same risk factors as atherosclerosis.
These risk factors include:
- Men have a higher incidence compared to women
- Increasing age
- Family history of cardiovascular disease
- Smoking
- Excessive alcohol intake
- Diabetes
- High blood pressure
- Chronic Inflammation: i.e. viral infections
- Increased cholesterol levels
- Compression from a mass
What are the symptoms?
Renal artery stenosis is generally not associated with any specific symptoms. Most patients commonly present with high blood pressure (hypertension).
Renal artery stenosis may be suspected in patients:
- Above the age of 50 or below the age of 30 with high blood pressure
- Who have had the need for more than 5 medications including a diuretic to control blood pressure
- Whose previously controlled blood pressure becomes uncontrollable
- With aortic or extensive peripheral vascular disease
- With worsening renal function
- With asymmetrical kidney sizes
- With flash pulmonary edema
What are the complications?
Complications of renal artery stenosis include:
-
Aortic Aneurysm
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Heart attack
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Congestive heart failure
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Kidney failure
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Stroke
How is it diagnosed?
Diagnosis is based around 3 parameters. These parameters include: Patient History, Physical Examination, and Testing.
- Patient History:
Renal stenosis may run in the family, so you might be asked about your family medical history.
History of any risk factors including diabetes, smoking, or increased cholesterol levels.
History of heart disease or peripheral vascular disease.
Severe headaches, nausea, vomiting, confusion, or blurring of vision may show symptoms of severe hypertension.
- Physical Examination:
During a physical examination, your vitals will be taken. Vitals include taking your pulse, blood pressure, and temperature. Blood pressure may be taken more than once.
A stethoscope will be placed over the side of the abdomen, to auscultate for a whooshing sound called a bruit. This may indicate the presence of renal artery stenosis.
- Testing
Laboratory investigations:
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Blood Analysis- this includes a complete blood count (CBC), electrolytes, renal function tests (urea and creatinine).
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Urine Analysis- to search for the presence of any blood in the urine.
Imaging Studies:
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Renal Ultrasound or Doppler- is a simple office procedure done to screen renal artery stenosis in high risk patients. The advantage of this test is that it is able to measure the size of a narrowing and measure the blood flow through the vessel.

- Renal Angiography- is the definitive diagnosis of renal artery stenosis. This is an invasive test done in a hospital setting, involving the insertion of a thin catheter into the groin, which passes through the aorta, then is advanced to the renal arteries. A contrast dye is given to view the arteries which are seen under x-ray guidance. This shows the exact number, location, and size of blockages. The advantage of an angiography is if a significant blockage is discovered, angioplasty or stenting may be done at the same time.

- Magnetic Resonance Angiography (MRA) - this is a test similar to an MRI, which involves a contrast dye to be injected into a vein, while pictures are taken of the renal arteries.
- Computed Tomographic (CT) Angiography: this test also involves the injection of a dye into the vein while pictures of the renal arteries are taken.
How is it treated?
The goal of treatment is to prevent the progression of renal artery stenosis, treating high blood pressure, and relieve blockages within the renal arteries.
Treatment involves 3 separate parameters including: lifestyle changes, medications, and procedures.
Life style changes
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First line of treatment is to reduce the risk factors by diet changes, exercise, and weight management.
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Eat foods high in fiber such as whole grain cereals, oatmeal, and figs. Eat plenty of fruits such as apples, bananas, prunes, oranges, and pears. Include fish and legumes such as beans or chickpeas.
-
Lower your salt intake
-
Avoid fatty foods including bad saturated fat that is found in some meats, dairy products, chocolates, baked goods, and deep-fried food.
-
Maintain a healthy weight with a BMI below 25
-
No smoking

-
Limit alcohol to one glass a day if you chose to drink
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Be more physically active
-
Relax and reduce stress
Medications:
Renal artery stenosis is associated with high blood pressure. The first and safest approach to treatment is with medication.
Medications used to treat high blood pressure include:
- Angiotensin Converting Enzyme (ACE) Inhibitors: lower blood pressure by preventing some of your natural chemicals to be made, promoting relaxation of the blood vessels and dilatation.
- Angiotensin II Receptor Blockers (ARBs): block the action of the natural chemicals that narrow blood vessels.
- Diuretics: sometimes referred to as “water pills”. These reduce sodium and water which lower blood volume.
These drugs may cause worsening of renal function. Medications and renal function will need to be closely monitored.
- Calcium Channel Blockers: helps to relax the muscles surrounding the arteries, increasing blood flow, and lowering blood pressure.
- Cholesterol lowering medications- these reduce cholesterol levels in those with conditions with atherosclerosis.
Procedure:
Intervention is required in arterial narrowing of 50-70%, uncontrolled blood pressure with medication, and worsening renal function. Interventions done to treat renal artery stenosis include:
- Renal Angioplasty and stenting: Renal artery stenting is the preferred method of treatment of renal artery stenosis. A bare metal stent is placed inside the renal artery to restore the blood flow. Once normal blood flow is restored, the kidney stops producing hormones that cause hypertension. The re-narrowing rate following renal artery stenting is 20%. Many patients are able to decrease their blood pressure medication usage or even stop them completely after this procedure.

- Endarterectomy: is a procedure in which a catheter is used to scrape the inner lining of an artery. That material which is blocking the artery is extracted by the use of a catheter.
- Renal Artery Bypass: The area of blockage in a renal artery is bypassed by attaching a vessel used from another area, to a site proximal of the blockage and attaching the other end to an area beyond the blockage.

How can I prevent this?
Healthy lifestyle habits can prevent renal artery stenosis from developing. Taking action in leading a healthy lifestyle can help keep your arteries strong, elastic, and free of plaque build up to ensure maximum blood flow.
These healthy habits include:
- NO Smoking
- Limiting alcohol consumption
- Controlling high blood pressure, cholesterol, and diabetes
- Eating a healthy diet
- Keeping physically active with the right kind of exercise
- Maintain a healthy weight with a BMI <25
- Get an annual flu shot
- Reduce stress!
Updated February 22nd, 2012 -JS
For further information please contact Cardiovascular Interventions, PA at 407-894-4880
T Wave (Repolarization) Alternans
What is it?
T wave alternans refers to a noninvasive diagnostic test to help identify patients who are at risk of life threatening heart rhythm disturbances that can lead to cardiac death.
This test refers to a beat to beat variability in the timing or shape of T waves on the surface electrocardiogram (ECG) which is not visible to the human eye. Therefore high resolution sensors are used to detect subtle changes in the ECG.
Sudden cardiac death is the nation’s number one cause of death. Most people who suffer a cardiac arrest have an underlying, pre-identifiable, electrical disorder that puts them at risk. A good way to identify this is through the measurement of TWA, a beat-to-beat alternation of a patients ECG.
Who needs it?
Patients who need a TWA are those who are at risk of sudden cardiac death (SCD). Sudden cardiac death occurs in patients who have abnormal heart rhythms or arrhythmias.
Risk of SCD typically focuses on the presence of a few factors:
- Reduced left ventricular ejection fraction (blood squeezed out from left heart chamber)
- Left ventricular dysfunction
- Ischemic cardiomyopathy
- Left ventricular hypertrophy
- Aortic stenosis
Reduced left ventricular ejection fraction is currently the primary determinate of Implantable Cardioverter Defibrillators (ICD’s), however; reduced ventricular ejection fraction lacks sensitivity.
TWA is a tool to help clarify the overall risk of arrhythmias, especially in patients who have known risks, but who fall into this ‘gray area’ in ICD eligibility.
How is it done?
TWA is performed during a treadmill stress test. The standard stress test can only identify the presence of clogged arteries, while the TWA test will identify subtle changes in electrical disturbances.
For TWA to be detected and measured at this microscopic level, the patient’s heart rate must be raised, usually by means of exercise on a treadmill.
This test is painless, noninvasive, and done on an outpatient basis.
Prior to the test, sensors are attached to the patient’s chest.
The patient begins to walk on the treadmill.
Once the patient’s heart rate reaches 90 beats per minute, the TWA test will begin.
After 2.5 minutes the test is complete.
Updated March 27, 2013 -JS
For further information please contact Cardiovascular Interventions, PA at 407-894-4880
Stroke
What is it?
A stroke is a medical emergency causing neurological injury to the brain due to ischemia or hemorrhage. Ischemia to the brain mean a portion of the brain is not receiving blood and oxygen. Hemorrhage means there is bleeding in the brain due to a ruptured blood vessel.
A stroke is due to a cardiovascular cause, yet causes neurological disorders. A stroke is a medical emergency.
What are the types?
The two types of stoke include:
- Ischemic stroke- more common (80%)
- Hemorrhagic stroke – less common (20%)
How does it occur?
A stroke occurs when blood flow to the brain stops for more than a few seconds depriving the brain of oxygen. Without oxygen for an extended period of time, permanent brain damage can occur.
Ischemic stroke may occur due to blocked or clogged arteries. A blocked artery can be due to a blood clot. A blood clot may form in an already narrowed artery (thrombotic stroke). A clot may also dislodge from another site of the body and travel to the brain (embolic stroke). Clogged arteries are due to a collection of fat, cholesterol, calcium and other substances in the interior wall of an artery.
A hemorrhagic stroke occurs in weak vessels which can rupture causing blood to leak into the brain. (i.e. aneurysm)
What are the risk factors?
A number of risk factors are present for stroke.These factors include:
- High blood pressure* (number one risk factor)
- Increasing age (>55)
- Family history of stroke
- Race (African Americans are at a higher risk)
- Diabetes
- High cholesterol
- Atrial fibrillation
- Peripheral vascular disease
- Oral contraceptives (birth control pills)
Precipitating factors:
- Obesity
- Heavy drinking
- Eating high amounts of cholesterol or salt
- Substance or drug abuse
What are the symptoms?
Stroke symptoms depend on what part of the brain has suffered from the lack of blood supply. Some people may be asymptomatic and may never know they have had a stroke.
Symptoms usually occur suddenly without any warning and can even occur while you are sleeping.
Symptoms are typically more severe when the stroke first happens, but can get worse.
Hemorrhagic stroke symptoms (bleeding within the brain) typically appear with a sudden and severe headache that can happen when lying down or while sleeping. The headache can get worse while straining, coughing, or with change of position.
Other symptoms of stroke include:
- Change in alertness
- Change in hearing
- Change in taste
- Change in the perception of touch, pain, pressure, and/or temperature
- Difficulty in swallowing, speaking, reading, writing
- Vision disturbances
- Loss of coordination
- Muscle weakness in face, arm, leg (usually on one side)
- Tingling or numbness on one side of the body
- Difficulty in walking
- Loss of balance
- Clumsiness
- Confusion/ loss of memory
- Change in mood, personality, or emotional changes
What are the complications?
A number of complications may occur with stroke. A stroke can cause temporary or permanent disabilities, depending on how long the brain has suffered without oxygen and which part of the brain was affected.Common complications include the following:
- Loss of movement or feeling in one or more parts of the body
- Loss of mobility
- Blood clots
- Difficulty eating and drinking, which increases the chance of pneumonia and malnutrition
- Difficulty in speech
- Seizures
- Urinary tract infection
- Bleeding in the digestive system
- Heart attack or heart failure
- Cardiac arrhythmia
- Bed sores
- Falls
- Dementia or thinking difficulties
- Change in behavior
How is a stroke diagnosed?
Anyone who has signs or symptoms of stroke needs immediate medical attention in an emergency department or hospital.Several tests may be used to determine your risk of stroke including:
- History & Physical Examination: Your doctor will ask you or a family member what symptoms you have been having, when they started, what you were doing when they began and if these symptoms are still present. Your doctor may ask you what medications you take and whether you have experienced any head injuries. You will be asked about your personal and family history of heart disease, TIA or stroke.
Your doctor will check your blood pressure and listen to your heart sounds and for any swooshing (bruit) sound over your carotid arteries in your neck, which may indicate atherosclerosis. An eye examination may be done using an ophthalmoscope to look for cholesterol crystals of clots in the back of your eyes.
- Blood Tests: Important tests will be ordered to see how fast your blood clots along will blood sugar levels, presence of infection, and other critical elements of blood.
- Computerized Tomography (CT) scan: Brain imaging is a key role in determining the presence of stroke and confirm the type of stroke. A CT scan uses a series of x-rays to create detailed images of your brain and the surrounding blood vessels of the head and neck.
A CT scan can reveal brain hemorrhage, tumors, strokes, and other conditions.
Doctors may inject a dye into your blood vessels to view your blood vessels for more detail known as a CT angiography.
- Magnetic Resonance Imaging (MRI): Uses radio waves and magnets to create a detailed image of your brain.
- Carotid Ultrasound: A noninvasive test using sound waves are used to determine fatty deposits (plaques) and quality of blood flow in your carotid arteries.
- Angiogram: Your doctor inserts a thin flexible tube (catheter) through a small incision in the groin and advanced up to the carotid arteries under x-ray guidance with the use of a dye to visualize the arteries.
- Echocardiogram (ECHO)- a noninvasive test using sound waves to determine the size and function of the heart’s chambers and the structure and function of the heart valves.
- Loop Recorder: for cryptogenic stroke or TIA; this is a device that is implanted into the chest which detects irregular heart rhythms (i.e. atrial fibrillations).
How is it treated?
A stroke is a medical emergency. Immediate treatment can save lives and reduce disability.Call 911 or seek urgent medical care at the first sign of a stroke.
Ischemic Stroke
The goal of treatment in ischemic stroke is to restore blood flow to the affected area of the brain as quickly as possible.Medicines used for the early treatment of stroke are used to break down clots (i.e. aspirin, anticoagulants, etc).
Results are best seen within the first 3 hours of symptoms.
- Some patients benefit from an injection into the vein of tissue plasminogen activator (TPA) which dissolves the blood clot causing stroke. TPA can also be administered directly into the brain with the use of a catheter threaded into an artery of the brain where the stroke is occurring.
- Mechanical removal of the clot can be done with the insertion of a catheter into an artery of the brain and removing the clot.
- Carotid Endarterectomy: is a procedure where a surgeon removes the fatty plaque from the carotid arteries in the neck that lead to the brain. This decreases the risk of having stroke again.
- Angioplasty and stent: Angioplasty is the stretching of an artery with a balloon to widen it, followed by stent placement. The physician inserts a long, thin tube (catheter) into the narrowed part of your artery. A wire with a deflated balloon is passed through the catheter to the narrow area. The balloon is then inflated, compressing the plaque against the artery walls so it no longer restricts blood flow. Following this a stent may be placed to prevent restenosis.
Hemorrhagic stroke
The treatment of hemorrhagic stroke depends on the cause of bleeding (i.e. high blood pressure, use of anticoagulant medications, head trauma, blood vessel malformation).
- The initial treatment is to first determine the cause of bleeding
- Control the blood pressure
- Stopping any medication that could increase bleeding. (If stroke is due to hemorrhage (bleeding), clot breaking drugs (thrombolytics) can cause more bleeding)
- Transfusion of clotting factors may be given to stop ongoing bleeding
- Measuring and controlling the pressure within the brain
Surgical blood vessel repair may be needed in cases of aneurysms or arteriovenous malformation (AVM) rupture.
Surgical options include:
- Surgical clipping: The surgeon places a clamp at the base of the aneurysm and stops blood flow to it.
- Coiling (endovascular embolization): The surgeon guides a detachable coil into the aneurysm and fills the aneurysm with the coil. This blocks blood flow into the aneurysm and causes the blood to clot.
- Surgical AVM removal: The surgeon removes the AVM if possible, to eliminate the risk of rupture and lower the risk of a hemorrhagic stroke.
- Decompressive craniotomy: When a patient’s life is threatened due to the high pressure effects of a clot in the brain, the physician may open the skull and/or remove the blood.
Once the bleeding within the brain stops, treatment usually involves bed rest and supportive medical care.
What is the prognosis?
The outlook depends on:
- The type of stroke
- How much brain tissue is damaged
- What body functions have been affected
- How quickly you get treated
Some patients may recover completely or some may remain with permanent loss of function.
Over half of the patients are able to function and live at home. Others may not be able to care for themselves.
If treatment with clot dissolving drugs is successful, the symptoms of stroke may go away. However, patients who do not receive treatment soon enough may not get the same results.
Patients who have ischemic stroke (due to blood clot) have a better chance of surviving than those with hemorrhagic stroke (bleeding).
Risk of a second stroke is highest during the weeks or months after the initial stroke.
How can I prevent this?
Know your stroke risk factors. Identifying your risk factors makes prevention of stroke easier.Follow your doctor’s recommendations and adopt a healthy lifestyle.
Some lifestyle recommendations include:
- Controlling high blood pressure
- Lowering the amount of cholesterol and saturated fat in your diet
- No smoking
- Control diabetes (high sugar levels)
- Maintain a healthy body weight with a BMI <25
- Eat a diet rich in fruits and vegetables
- Exercise regularly
- Drink alcohol in moderation, if at all
- Treat obstructive sleep apnea, if present
- Reduce stress
*Ask your doctor about taking low dose aspirin!
Taking a small dose of aspirin thins the blood and helps prevent blood from clotting.
Updated March 26, 2013 -JS
For further information please contact Cardiovascular Interventions, PA at 407-894-4880
Cardioversion
What is it?
Cardioversion is a procedure used to treat abnormal and rapid heart rhythms known as cardiac arrhythmias. Cardioversion is most commonly used for atrial fibrillation.
Cardioversion delivers an electrical ‘shock’ to the heart to restore the heart to its normal rate and rhythm.
The energy delivered can be given in one of two ways. The electrical energy can be delivered externally with the use of electrodes attached to the chest, or directly to the heart with the use of paddles to the heart during open heart surgery.
An alternative mode of delivery is by the use of a permanent implantable device known as an implantable cardioverter defibrillator (ICD).
How does a normal heart beat?
Heart muscle cells are activated by electrical impulses that cause them to contract regularly and in sync. This contraction produces a heartbeat, allowing blood to be pumped out to the entire body.
Electrical impulses originate from specialized cells called the sinoatrial (SA) node, which is the hearts natural pacemaker. The SA node is located in the upper right chamber of the heart, the right atrium. From the SA node, the impulse spreads across the upper chambers of the heart to reach the atrial ventricular (AV) node located between the atria and lower ventricles. After leaving the AV node, impulses spread across the pumping chambers of the heart, the ventricles. As impulses are spread along the heart, the cardiac muscle cells are stimulated to contract, producing a heartbeat.

What is an arrhythmia?
During a rapid cardiac arrhythmia, an abnormal electrical mechanism overrides the action of the sinoatrial node. Some arrhythmias start in the atria, while others are produced by the ventricles.
Sometimes, cardiac tissue can form an electrical loop or short-circuit. If the hearts electrical energy goes through this loop, it will cycle over and over again, repeating itself indefinitely causing a re-entrant loop. Each loop through this cycle causes the heart to contract.
Arrhythmias occurring by a re-entrant loop are usually regular, organized, and can be treated with cardioversion. Atrial flutter and ventricular tachycardia are examples of organized arrhythmias. Rapid, disorganized, chaotic arrhythmias include atrial fibrillation.
Cardioversion involves a high energy shock to the heart muscle which activates all of the cardiac muscle and conduction tissue simultaneously. This interrupts and breaks the re-entrant loop ceasing the arrhythmia, triggering the sinoatrial node to fire again and restoring a normal heart rhythm.

What to do before the procedure?
Prior to a cardioversion, if not already on anticoagulation therapy, your physician will determine your risk of blood clot formation and choose an anticoagulant medicine (i.e. Coumadin) to prevent the risk of stroke or heart attack.
Arrive at least one hour before your procedure to allow for prep time.
Make arrangements to have someone drive you to and from the clinic. You will not be permitted to drive after the procedure.
What happens during the procedure?
An external cardioversion is common and is performed in a specially equipped procedure room in our office.
The patient’s heart rate and rhythm, blood pressure, breathing rate, and oxygen levels are monitored.
An I.V. medication is given to sedate the patient to avoid any pain for when the shock is given. Patients will be asleep during the cardioversion and most will not recollect the procedure.
Two pads will be applied to the skin. One pad will be placed near the breast bone and the second pad on the back, under the left shoulder blade.
After the patient is sedated, the doctor will deliver the shock given through these pads.
The cardioversion itself only takes a few seconds.
The patient usually wakes up within 5 to 10 minutes.

What happens after the procedure?
The patient will be monitored for at least one hour after the procedure.
You may also be required to wear a Holter monitor for 24 hours to monitor the electrical activity of the heart after the procedure.
What are the benefits?
The benefits of cardioversion include:
- The return of normal heart rhythm
- Improvement of the heart’s ability to effectively pump blood
- Decrease symptoms including palpitations, chest pain, light headedness, difficulty breathing etc.
- Decrease the risk of clot formation in the heart
What are the complications?
Several complications can occur with cardioversion. These include:
- Provocation of other arrhythmias or heart block
- Embolus
- Myocardial necrosis
- Skin burns
Updated April 3, 2013 -JS
For further information please contact Cardiovascular Interventions, PA at 407-894-4880
Peripheral Vascular Disease
What is it?
Peripheral vascular disease is a disease of the arteries of the circulatory system due to atherosclerosis. Atherosclerosis is a gradual build up of fat in the arterial walls depriving the limbs of the oxygenated blood flow it needs.
Atherosclerosis is not confined to one artery, but may involve other areas as well.
Most commonly peripheral vascular disease is seen in the legs and is noticed by intermittent pain while walking.
Similarly, when atherosclerosis affects the heart it is called coronary artery disease.
What causes peripheral vascular disease?
At a young age, fat can start to deposit within the blood vessel walls. Fat builds up over the years that can cause injury to the blood vessel walls. Not only fat, but a number of other substances in blood like inflammatory cells, cellular waste products, proteins, and calcium begin sticking to the inside of vessel wall. All of these substances collectively form what is called Plaque. The formation of plaque is known as atherosclerosis.
Many of the plaque deposits have a soft inner center and hard exterior. If the hard outer surface breaks or tears, the soft inner center will become exposed to our body’s platelets. The platelets will come to the area of breakage and form a clot around the plaque. This causes the artery to narrow even more. A clot may dislodge and permit blood flow again, but the dislodging of a clot can also be dangerous.
The reduced blood flow deprives the tissues of oxygen and nutrients. There is more of a demand for oxygen during activities such as walking, climbing stairs, or exercise. During these times, symptoms usually occur.



Although atherosclerosis is the most common, other causes of peripheral vascular disease include:
- Congenital: some individuals develop structural defects in the blood vessels at birth.
- Autoimmune: people with autoimmune conditions can develop inflammation of the blood vessels known as vasculitis.
- Thrombosis: a blood clot can narrow or obstruct a blood vessel.
- Diabetes: can cause damage to the blood vessels which can make them more likely to be weakened or narrowed.
- Infective: certain infections like syphilis can cause inflammation and scarring leaving a blood vessel weak or narrow.
- Trauma
What are the risk factors?
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Genetic: inherited tendency within the family
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Age: increasing age, above 50 years
-
Sex: men are slightly more at risk than women
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Family history of peripheral vascular disease, heart disease, or stroke
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Smoking
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Diabetes
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Obesity (BMI > 30)
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High blood pressure
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High cholesterol
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Sedentary lifestyle
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Coronary artery disease
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High levels of homocysteine
What are the symptoms?
Many people with peripheral vascular disease have mild or no symptoms.
The first symptom is claudication. This is a pain or cramping in the calf while walking or during exercise. Pain usually subsides at rest, unless the blockage is severe and will also appear at rest.
Other symptoms include:
- Pain or cramping in the hip or thigh during walking or climbing up stairs
- Tingling or numbness in the legs
- Feet that feel cold
- Slow healing of cuts or sores on the feet and legs

- Pale, shiny, or bluish skin
- Brittle and slow growing nails
- Loss of hair over the limbs
- Absent or weak pulse in the legs or feet
- Erectile dysfunction
These symptoms are NOT a normal part of aging.
Do not be afraid to speak up and let Dr. Jamnadas know of any symptoms you are having.
What are the complications?
If left untreated, peripheral vascular disease can develop:
- Permanent tingling, numbness, or weakness in the legs or feet
- Permanent burning or aching pain which can lead to an excruciating pain
- Leg or foot ulcers
- Gangrene – death of a part of the body due to lack of blood supply. Treatment will be amputation of the affected part.
- Stroke and heart attack- people with peripheral vascular disease are at higher risk of heart attack and stroke
- Renal artery disease or stenosis- blocking the blood flow to the kidneys.
How is it diagnosed?
Dr. Jamnadas will know if you have peripheral vascular disease by:
- Discussing with you about your symptoms, risk factors, medical and family history.
- On physical examination- weak or absent peripheral pulses along with other signs of peripheral vascular disease will be looked for.
- Blood tests- measuring your lipid profile and glucose levels in case diabetic.
By performing tests including:
- Ankle Brachial Index (ABI) – this is a non-invasive test measuring the ratio of blood pressure in the ankle to your arm. If the value is lower than expected, this may indicate a vascular problem. You may be required to walk on a treadmill and have readings taken before and after exercise.
- Ultrasound Doppler Test- helps to evaluate the blood flow through a vessel and identify the site of blocked or narrowed arteries.
- Angiogram- allows Dr. Jamnadas or Dr. Kelly to locate the exact anatomical site of blockage by injecting a dye within the arteries, which is visualized on x-ray showing the amount of blood flow to an area, and the number, size, and location of any blockages.
How is it treated?
It is very important to reduce any risk factors in order to manage the symptoms and progression of peripheral vascular disease.
Lifestyle changes
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Quit smoking!!

-
Limit alcohol intake if you chose to drink
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Lower blood sugar levels
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Maintain a normal blood pressure and cholesterol
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Eat a well balanced diet
-
Eat foods high in fiber such as whole grain cereals, oatmeal, and figs. Eat plenty of fruits such as apples, bananas, prunes, oranges, and pears. Include fish and legumes such as beans or chickpeas.
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Lower your salt intake
-
Avoid fatty foods including bad saturated fat that is found in some meats, dairy products, chocolates, baked goods, and deep-fried food.
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Exercise regularly for 30 minutes at least 3-4 time
s a week and be more physically active
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Maintain a healthy weight with a BMI below 25
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If you have diabetes, practice proper foot care and prevent injury to the foot. Avoid walking barefoot and wear proper shoes. Maintain toenails and skin care. Be sure to inspect the soles of your feet regularly.
Medical treatment
-
Cholesterol lowering medications- these act by decreasing the amount of cholesterol in blood, especially Lowering your low density lipoprotein (LDL- your Lousy cholesterol) and keep your high density lipoprotein (HDL- your Healthy cholesterol) High
-
High blood pressure medications- such as Beta blockers that slow your heart rate and reduce blood pressure which decreases the oxygen demand of the heart
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Antiplatlet medication- such as aspirin or plavix which prevents platelets clumping together. This thins the blood and reduces the chances of a blood clot narrowing or obstructing the arteries. This is not appropriate for all patients including patients with bleeding disorders or already taking a blood thinner.
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Controlling of blood sugar
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Cilostazol (Pletal) - used for claudication and pain relief by increasing the blood supply to the limbs by dilating blood vessels and preventing clots.
-
Pentoxifylline (Trental) - this is an alternative medication which is less effective than Cilostazol, but has fewer side effects of headache and diarrhea.
Supplements
Food and supplements rich in omega 3 fatty acids help reduce inflammation throughout the body, lower blood pressure, and the risk of heart attack.
- Fish and Fish Oil- are high in omega 3 fatty acids, especially in fish like salmon, herring, and tuna. Fish oil supplements are also high in omega 3 fatty acids.
- Flax and flaxseed oil- contain omega 3 fatty acids and fiber
- Greensoul- is a natural supplement that helps to maintain a healthy cholesterol level, supports the body’s immunity, and the ability to fight free radicals.
- Coenzyme Q10- helps maintain a healthy cholesterol level, boosts immunity, and energy.
- Perfusion SR- helps enhance blood flow, maintain a normal blood pressure, and enhance the elasticity of large arteries.
Procedures to restore blood flow

-
Angioplasty and stent placement: this is a procedure considered as non-surgical because it is done by Dr. Jamnadas or Dr. Kelly, who accesses the artery by inserting a long, thin tube (catheter) into the narrowed part of your artery while being visualized under x-ray. A wire with a deflated balloon is passed through the catheter to the narrow area. The balloon is then inflated, compressing the plaque against the artery wall and widening it, so it no longer restricts blood flow.
Blocked arteries in the renal, iliac, and legs can easily be treated with stenting or arthrectomy.
Unlike stenting done in the coronary arteries, only bare metal stents are used in the peripheries.
Bare metal stents act as a scaffolding to keep an artery open but suffer a 30% re-stenosis rate. This is due to a process called intimal hyperplasia which is attributed to a keloid. A keloid is an overgrowth of the inner lining of the blood vessel that covers over the stent. However, this reparative process lasts no more than 6 minutes. After 6 minutes, if an artery has not re-narrowed at the site of the stent, it is unlikely to re-narrow.
If symptoms were to reoccur, it would be due to New blockages at another site within the arteries.



- Atherectomy: is a procedure where a catheter is used to scrape the inner lining of an artery. That material which is blocking the artery is extracted and then taken out of the artery using the catheter. The procedure is Best suited for blocked arteries in the legs. Laser Arthrectomy has been used in the legs also, but Dr. Jamnadas does not favor it at this time due to the high re-stenosis rate.
**Patients will need to improve risk factor modifications to prevent New blockage formation.
- Bypass Surgery: is performed by removing a portion of a small blood vessel and sewing or ‘grafting’ one end of the bypass proximal to the area of blockage and the other end beyond the area of blockage, therefore bypassing the affected area.
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Bypassing artery in the thigh |
Bypassing artery in the leg |
If your disease is very severe and these procedures are not recommended for you, your foot or leg may need to be removed (amputated). This is usually necessary if you have very little to no blood flow leading to death of the tissues which can cause risk of a life threatening infection.
Amputation is a last resort, but if peripheral vascular disease is not controlled, it is a possible result.
How can I prevent this?

-
Do not smoke!
-
Maintain a healthy weight with a BMI <25
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Maintain a normal blood pressure, cholesterol and sugar levels
-
Eat nutritious, low fat foods and avoid foods high in cholesterol
-
Exercise regularly for 30 minutes at least 3-4 times a week
Updated January 23, 2012 -JS
For further information please contact Cardiovascular Interventions, PA at 407-894-4880
Venous Insufficiency
What is it?
Venous insufficiency is a condition which affects the veins of the legs. In healthy veins, blood is pumped from the legs up to the heart. Blood is pumped upwards due to a series of valves within the veins directing blood flow from the superficial veins to the deep veins leading back to the heart. Contraction of the calf muscles also aid in pumping the blood flow back up to the heart.
When the valves within the veins are damaged or not working properly, blood flows backwards resulting in a pooling of blood in the legs. Pooling of blood in the legs increases the pressure in the veins leading to venous hypertension. This increase in pressure can cause mild symptoms of leg heaviness and aching or in chronic cases may progress to severe symptoms.


What are the symptoms?
Symptoms of venous insufficiency include:
- Dull aching, heaviness, or cramping in the legs
- Pain relieved with leg elevation and aggravated while standing
- Edema (swelling of the legs)

- Skin discoloration
- Prominent veins (varicose veins)
- Skin ulcers
- Itching, tingling, burning, or throbbing sensation in the legs or feet
- Leg weakness
What are the causes?
Several causes may lead to venous insufficiency including:
- Congenital- born with weak vein walls or abnormal valves
- Deep vein thrombosis (DVT) – blocks the blood flow through a vein. This increases pressure in the vein and may lead to permanent vein or valvular damage.
- Superficial Phlebitis- infection of the superficial vein in the legs can cause damage to the veins and valves.
- Direct leg injury to the veins or valves
- Increase in body weight (i.e. pregnancy, obesity) - increases pressure in the veins
- Weakness in the leg muscles- standing or sitting for long periods of time without walking can decrease the draining of blood from the legs, resulting in increase pressure and pooling of blood.
What are the risk factors?
Those at risk of developing venous insufficiency include:
- Family history of venous insufficiency
Sex: women are more prone than men
- Increasing age
- Varicose veins
- History of deep vein thrombosis
- Obesity
- Pregnancy
- Prolonged sitting or standing
- Muscle weakness
- Trauma to the legs
- Cancer
What are the complications?
If venous insufficiency is left untreated, complications may arise due to the high pressure within the vein and reduced clearance of metabolites from the legs.
Complications include:
- Pain
- Infection
- Thrombophlebitis
- Swelling of the legs
- Non-healing venous skin ulcers
- Recurrent cellulitis
- Stasis dermatitis
How is it diagnosed?
Venous insufficiency is diagnosed based around 3 parameters.
These parameters include: Patient History, Physical Examination, and Testing.
A. Patient History:
Symptoms give a strong clue in diagnosing venous insufficiency.
Typical symptoms include:
- Swelling, throbbing, cramping, heaviness, and burning in the legs
- Development of leg pain after standing or sitting for long periods of time
- Pain that is relieved by elevating the legs
- Warmth aggravates symptoms while cold relieves them
B. Physical Examination:
The appearance of the leg gives an idea of whether venous insufficiency is present.
Skin discoloration, swelling, stasis dermatitis, cellulitis, and dilated veins may be noticed in the legs.
Venous skin ulcers if present are typically located over the inner aspect of the ankle, but can also be present over the leg.
A manual maneuver known as The Trendenlenburg test may help in distinguishing insufficiency due to valvular damage. This is done by elevating the leg to empty the veins and applying pressure to occlude the junction where reflux is occurring from the deep to superficial veins. While maintaining this occlusion, the patient is asked to stand. If the lower portion of the vein remains empty or slowly fills, the occlusion is removed. If this is followed by rapid filling, the junction has been correctly identified. If filling occurs with the occlusion, this may indicate a valvular problem in the deep veins.
C. Tests
Laboratory investigations:
Blood analysis: Elevated platelets levels increase the susceptibility to clot formation.
Diagnostic Studies:
-
Venous Reflux Study: this is a non-invasive test carried out in our clinic used to detect the presence of venous insufficiency
-
Venous Doppler: is a non-invasive test done in our clinic showing the direction of blood flow and detects thrombus (clot) formation
-
Magnetic Resonance Venography (MRV): is an imaging study used to provide a detailed picture of the deep and superficial veins of the legs using radio waves within a strong magnetic field
-
Direct Contrast Venography: is an invasive test used to detect venous and nonvascular causes of leg pain and edema
How is it treated?
The goal in treatment is to reduce symptoms and correct the underlying problem.
Conservative treatment:
-
Weight loss: losing weight can boost the blood flow in your legs.
-
Leg Elevation: Legs are elevated just above the level of your heart for about 30 minutes and should be done three to four times a day. Elevation improves the drainage of blood and metabolites from the lower extremities. This helps relieve symptoms in patients with mild venous insufficiencies.


-
Exercise: Foot and leg exercises help to improve symptoms. Movement helps prevent stasis of blood and also helps the calf muscles pump blood up towards the heart.

- Graduated Compression: this is the cornerstone of treatment.

Compression stockings can be used in all patients with venous insufficiency and is available in our clinic. Compression stockings apply a greater amount of pressure toward the ankle while pressure tapers as it goes up the leg. Compression stockings prevent pooling of blood and help to improve blood flow from the lower extremities.

Compression bandages are used for patients with severe symptoms including severe leg swelling or ulcers. Ulcers must have a dressing on before the application of a compression bandage. Application should be done by trained personnel. The bandage must stay dry and covered while bathing. If the bandage becomes wet, the bandage should be removed due to development of potential infection. The bandage may be changed once to twice a week.
Procedures:
The goal of these procedures is to improve the venous circulation by correcting insufficiencies by removing the reflux pathways.
- Sclerotherapy: is a chemical ablation done by injecting a sclerosing agent into the vein, causing the vein to collapse.
- Thermal Ablation: is a minimally invasive technique using intense thermal energy to irreversibly destroy an incompetent or diseased vein. This procedure is carried out in our clinic.

- Vein ligation or Stripping: is a surgical procedure done to remove veins requiring the use of incisions. Ligation and stripping are becoming outdated due to ablation techniques.
How can I prevent this?
-
Lose weight
-
Avoid prolonged standing or sitting
-
Be more physically active by walking or running to promote the calf pumping function
-
Elevate your legs to improve circulation
-
Correct the underlying problem to prevent progression
-
Change your sitting or standing position regularly
-
Protect your legs from injury
Updated February 15th, 2012 -JS
For further information please contact Cardiovascular Interventions, PA at 407-894-4880
Venous Ablation
What is it?
Venous ablation is a minimally invasive technique using intense thermal energy to irreversibly destroy an incompetent or diseased vein.
A catheter with heat is inserted into the targeted vein and closes off the vein. The vein is closed off, but not removed.
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Damaged valve causing venous insufficiency |
Damaged vein becomes tortuous (rope like) |
Pooling of blood due to damages valve |
Who needs it?
Venous ablation is indicated in patients with symptoms of venous disease with reflux.
- Dilated, engorged, or tortuous veins (varicose veins)
- Skin discoloration

- Burning or itching sensation in the legs or feet or around the veins
- Swelling in the legs, ankles, or feet (edema)
- Heavy feeling or weakness of the legs
- Cramping or throbbing in the lower legs

- Skin ulcers
- Leg pain, especially after sitting or standing for a long period of time
- Spider veins
A venous Doppler ultrasound is used to confirm the presence of venous obstruction, reflux, valvular incompetence, or clot formation.
Who does not qualify as a candidate?
Patients who do not qualify for venous ablation include patients with:
- Thrombus (blood clots) in the affected vein
- Infection in the vein (phlebitis)
- A combination of infection and clot known as thromboembolism
- Pregnancy
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Clot formation |
Why do I need it?
The goal of venous ablation is to treat and reduce the symptoms and signs of venous disease.
Venous ablation is also useful in preventing the risk of complications from venous disease (blood clot formation).
How to prepare for the procedure:
Before the procedure:
This procedure is done here in our clinic. Arrive at the clinic at the time of your appointment.
You may eat and drink before and after the procedure. This procedure is not effecting by eating.
This procedure does not require any alteration in medications, so take your morning medications as prescribed.
Arrange for someone to drive you home after the procedure. You may not be permitted to drive immediately after the procedure.
The procedure lasts about 1-2 hours.
During the procedure:
The procedure will be carried out in our clinic in our procedure room.
During the procedure you will lay on a table positioned on your back.
Under ultrasound guidance, the exact location of the vein will be determined.
A guide wire is inserted through a small opening in the skin into the targeted vein.
Once the guide wire is in, an introducer sheath is passed over the guide wire. The guide wire can then be removed. Under ultrasound guidance, the catheter is passed through the sheath and is advanced up the vein until it reaches 2cms below a junction called the saphenofemoral junction.
Under sterile precautions, a local anesthetic along with sodium bicarbonate will be injected into the tissue along the length of the vein to ensure that you do not feel any heat from the catheter. This compress the vein from two sides and also separates the vein from other structures including nerves.
After the catheter is in place, the tip of this will send out a radiofrequency which is converted into heat that reaches 120oC. When the catheter is slowly withdrawn, the heat causes the vein to collapse and close off. Once the vein is closed off, blood is re-routed into other healthy veins.

After the procedure:
While you are on the table, an ultrasound will be done over the ablated vessel to confirm that the vessel is collapsed as well as an absence of blood flow in that vein. 
Following the procedure, a bandage will be placed over the insertion site. A compression bandage will be wrapped around the entire length of the leg and should be worn for 2-3 days.
A compression bandage is important because it helps prevent bruising and tenderness as well as reduces the risk of blood clot formation and dislodgment. A compression stocking is required to be worn for up to 4 weeks.
You are encouraged to walk for at least 30 minutes a day after the procedure to prevent deep vein thrombosis.
Avoid heavy or strenuous exercise for a few days.
Avoid prolonged sitting or standing.
Wear compression stockings for up to 2 weeks.
An ultrasound follow up is required 3 days after the procedure to rule out any deep vein thrombosis (clot formation).
After 4 weeks of the procedure, an ultrasound follow up is required to ensure the complete closure of the vein and no reflux.
What are the possible risks and complications?
Venous ablation is generally a safe procedure, but with any surgical procedure it carries a risk.
If they occur are usually minor and temporary.
Complications include:
- Bruising
- Pain
- Infection
- Skin burn
- Blood clot formation
- Perforation of vessel
- Nerve irritation or damage
What are the benefits?
The benefits of venous ablation include:
- Relief of symptoms
- Outpatient procedure
- Well tolerated procedure
- High success rate
- Less invasive, less pain, tenderness, and few complications compared to surgical venous procedures
- Does not require general anesthesia
- Quick healing time
- Can return to normal daily activities within 1-2 days
- Good cosmetic results
- Greater efficacy compared to venous surgical procedures with a positive prognosis of 10 years
Updated February 13th, 2012 -JS
For further information please contact Cardiovascular Interventions, PA at 407-894-4880
Syncope
What is it?
Syncope is defined as a brief loss of consciousness with an inability to maintain postural tone that is followed by a rapid and complete recovery.
Syncope is common and about one third of the population experiences a syncopal episode in their life. Syncope may be an alarming sign in some people who have a serious underlying medical condition. In a younger age group, more than 75% of cases are not associated with an underlying medical problem.

What are the symptoms?
Syncope is often preceded by symptoms of ‘faintness’ including:
- Light headedness or dizziness
- Blurring of vision
- Heaviness in the lower limbs and swaying
- Loss of consciousness
- Drowsiness
- Pale skin
- Sweating
- Nausea
What are the causes?
A number of causes can lead to syncope. To remain conscious, oxygen rich blood must supply the brain without delay. A delay of blood to the brain for even 3-5 seconds can result in syncope.
Consciousness is regained after falling or lying down because blood returns to the brain once gravity is removed. A number of medical conditions can cause syncope.
The most common causes include:
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Sight of blood |
A. Vasovagal Syncope
This is the most common type of syncope. This is triggered by a number of conditions including physical or emotional stress, fear (i.e. sight of blood), dehydration, bleeding, pain, anemia, fever, or fasting.
These conditions or ‘triggers’ stimulate a nerve to slow the heart rate and dilate the blood vessels, which causes blood to pool in the legs, resulting in low blood pressure (hypotension). This leads to a decrease or delay in blood flow to the brain resulting in syncope.
Symptoms include nausea, sweating, stomach pain, hyperventilation, weakness, and confusion.
B. Cardiac Syncope:
Cardiac syncope can be due to arrhythmia or obstruction to cardiac outflow.
-
Alteration of the heart rate and rhythm can produce cardiac syncope. The heart rate and rhythm are controlled by the natural pacemaker of the heart known as the Sinoatrial Node.
This regulates the electical activity of the heart which follows through a conduction pathway.
Syncope can occur due to problems at several places in this pathway.
When this is altered the condition is termed as arrhythmia.
Arrhythmias are produced by:
- Bradycardia: this means the heart rate is slower than normal. A slow heart rate does not allow enough blood flow to reach the brain.
- Heart Block: The transmission of impulses from the sinoatrial node are blocked. This interrupts the heart rate, making it slow and delaying blood flow to the brain.
- Ventricular Tachycardia: this refers to a fast heart rate. The ventricles in the heart send out their own rapid electrical impulses, over taking the normal rhythm produced from the sinoatrial node. This causes an inadequate amount of blood flow to the brain.
- Supraventricular Tachycardia: this also includes a fast heart beat, although the generation of rapid electrical impulses originates above the ventricles.

- Obstruction to cardiac outflow may also result in a sudden loss of consciousness. The most common causes of obstruction are due to hypertrophic cardiomyopathy (exertional syncope) and aortic stenosis.
- Hypertrophic Cardiomyopathy: is a thickening of the left ventricular muscle wall. This can provide inadequate blood flow from the heart to reach the brain.
- Aortic Stenosis: is the thickening of the aortic valve in the heart leads to inadequate blood flow leaving the heart and can also lead to hypertrophic cardiomyopathy.
Other obstructive causes include mitral stenosis, pulmonary stenosis, pulmonary embolus, left atrial myxoma, and pericardial tamponade.
C. Orthostatic Syncope (Postural Syncope):
This refers to a low blood pressure after suddenly getting up from a laying position or from standing for a long period of time.
The heart rate slows and the blood vessels in the legs dilate causing a pooling of blood in the legs, leading to a lowering of blood pressure. This can decrease the blood flow to the brain and cause a syncopal episode.
The causes of orthostatic hypotension include blood or fluid loss, illnesses affecting the nervous system (i.e. diabetes, Parkinson disease), alcohol, carotid sinus hypersensitivity, and certain medications used to treat arrhythmias and lower blood pressure.
These medications include:
Angiotensin Converting Enzyme (ACE) Inhibitors |
Nitrates |
Beta Blockers |
Quinidine |
Calcium Channel Blockers |
Amidarone |
Diuretics |
Tricyclic Antidepressants |
D. Other Causes:
Other causes of syncope include low blood sugar, anemia, hyperventilation, seizures, heart attack, cardiac tumor, or pulmonary embolism.
Differential Diagnosis of a syncopal attack:
Endrocrine |
Cardiovascular
|
Adrenal Insufficiency/Crisis |
Abdominal Aortic Aneurysm |
Hypoglycemia |
Acute Massive Myocardial Infarction |
Aortic Dissection |
|
Metabolic |
Aortic Stenosis |
Hyponatremia |
Asystole |
Atrial Fibrillation |
|
|
CNS |
Brugada Syndrome |
Subarachnoid Hemorrhage |
Obstructive Cardiomyopathy |
Heart block |
|
Toxicity |
Left Atrial Valve Thrombus or Myxoma |
Antidepressant Toxicity |
Long QT Syndrome |
Antiarrhythmic Toxicity |
Mitral Stenosis |
Beta-Blocker Toxicity |
Multifocal Atrial Tachycardia |
Calcium Channel Blocker Toxicity |
Pulmonary Embolism |
Cocaine Toxicity |
Pulmonary Valvular Stenosis |
Tricyclic Antidepressant Toxicity |
Pulmonary Hypertension |
Sinus Bradycardia |
|
Sick Sinus Syndrome |
|
Tetralogy of Fallot |
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Torsade de Pointes |
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Tachycardia |
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Wolff-Parkinson-White Syndrome |
How is it diagnosed?
Dr. Jamnadas or Dr. Kelly will better understand your syncope based around 3 parameters. 
These parameters include: Patient History, Physical Examination, and Testing.
A. Patient History:
Obtaining patient history is the most important diagnostic tool in syncope. 85% of the diagnosis comes from the history given from the patient.
- Dr. Jamnadas or Dr. Kelly will ask of the events which occurred before, during, and after a syncopal attack.
- You will be asked of precipitating factors such as sleep or food deprivation, if you were in a warm atmosphere, if alcohol was consumed, if u had any pain, or had strong emotional stress before the occurrence of syncope.
- Warning signs before the attack might be discussed such as sweating or dizziness, which gives a clue towards a vasovagal syncopal attack.
- If the event was sudden without warning signs, it is more likely to be a heart rhythm problem.
- If the syncopal attack occurred during exertion, it is most likely an obstructive cause.
- If the event occurred after postural changes or standing for a long period of time.
- Details may be given from a witness present at the time of syncope and be able to estimate the duration of unconsciousness.
- A witness may also provide history of how you were after the episode. If postevent confusion was present, it is a clue to determining weather the episode was syncope or a seizure. Postevent confusion is suggestive of a seizure.
- You will also be asked if you have any preexisting medical conditions like heart disease or diabetes.
- History will be taken from patients who have taken medication prior to the event, including blood pressure lowering drugs.
- You will also be asked of any other personal and family history of cardiac disease.
B. Physical Examination:
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During a physical examination, your vitals will be taken. Vitals include taking your pulse, blood pressure, and temperature. Blood pressure may be taken more than once, in the laying and sitting position.
- Dr. Jamnadas or Dr. Kelly will listen to the sounds of your heart to determine weather any high-grade valvular defects are present. They will also listen for any carotid artery bruits, indicating a narrowing of the blood vessel supplying the brain.
- Carotid sinus massage may be done. The carotid artery (artery in neck) is firmly massaged while your heart rate is monitored. This can help in diagnosing a condition known as carotid sinus syncope.
- You will also be examined for any injuries that may have been sustained secondary to syncope such as head injury, lacerations, or fractures.
C. Testing:
A number of medical tests can help determine the cause of syncope. These tests include:
Laboratory investigations:
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Blood Analysis- this includes a CBC to reveal signs of anemia, serum glucose levels, electrolytes and renal function tests. Cardiac enzymes may be checked in patients whom cardiac origin is high.
- Urine Analysis- to search for bacteria causing urinary tract infection, which may precipitate syncope.
Diagnostic Studies:
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Electrocardiogram (EKG) - this is a common and easy test to perform which records the electrical activity of the heart. This detects any irregular heart rhythms and may show evidence of a preexisting heart attack.
- 24-Hour Holter monitor- records the electrical events of the heart during your normal daily activities. This helps in detecting signs of arrhythmias. It is important to accurately record your activities and symptoms so Dr. Jamnadas or Dr. Kelly can compare them to the Holter monitor findings.
- Event monitor- If symptoms of arrhythmia are infrequent and do not occur within 24 hours, an event recorder may be ordered for you for up to 30 days.
- Echocardiogram (ECHO) - shows images of the heart determining the size and shape of the heart, and shows whether the heart walls and pumping activity are normal or performing weakly.
- Tilt Table Test- In this test you will be asked to lay flat on a table. The table will be tilted in various angles while your heart rate and blood pressure are monitored. This test is useful in revealing abnormal cardiovascular reflexes that produce syncope.

- Stress test- helps access the blood flow to the heart at rest and during stress. Will detect if any areas are receiving less blood flow.
Other Studies:
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Chest x-ray- to assess the size and shape of the heart in congestive cardiac failure, infections within the lung causing pneumonia, or any fluid or mass can be seen in the lungs.
- Computerized tomography (CT scan) - to look for signs of abdominal aortic aneurysm or aortic dissection.
- T-wave alternans test (TWA)- detects abnormalities in electrical conduction that predicts sudden death.
How is it treated?
Syncope is treated depending on the underlying cause. The goal of treatment is to prevent recurrences and the progression of serious problems.
Vasovagal syncope:
Vasovagal syncope can be treated by educating the patient of the precautions to be taken as well as avoiding potential triggers to minimize the potential risk of harm.
For example:
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If you have carotid sinus syncope, you will be instructed to not wear tight collars, to use a razor instead of electric razor, and drink plenty of fluids.
- If you usually faint at the sign of blood, Lie Down immediately and elevate the legs once you feel any symptoms of syncope. If you are unable to lie down, sit down with your head between your knees.

Cardiac Syncope:
Cardiac syncope is treated with antiarrhythmic drugs or pacemaker/ ICD placement. A pacemaker is a small device that is implanted under the skin of the chest. This connects wires to the heart which transmits impulses to regulate the heart rate and rhythm.

Patients with life-threatening ventricular arrhythmias may need an implantable cardioverter defibrillator (ICD). This does the same as the pacemaker, but also administers an electric shock to correct the electrical problem that can prevent a person from dying.
Cardiac outflow obstruction can be treated with Beta Blockers to decrease the workload of the heart. Cardiovascular intervention procedures may be required or valvular correction surgeries.
Orthostatic Syncope:
Orthostatic syncope treatment also involves patient education. Certain techniques are designed to decrease the pooling of blood in the legs that allows blood pressure to drop once you stand. These techniques include the contraction of the leg muscles before and during standing. You will also be instructed to and rise slowly, in stages to stand.
Additional therapy may be given such as increasing salt in your diet, foot exercises, compression stockings for the legs, and consumption of plenty of fluids can be useful in the treatment of syncope. 
It is important to Stay Hydrated.
Antihypertensive medications, especially diuretics are notorious for causing orthostatic syncope and many need adjustment.
Medications used:
- Fludrocortisone- can be given to increase the amount of blood volume.
- Midodrine- is a medication that can also be given to constrict the blood vessels to increase the blood volume.
What is the prognosis?
Vasovagal syncope and orthostatic syncope have an excellent prognosis and do not increase the risk of death.
Patients with cardiac syncope may be restricted from their daily activities.
Without treatment the prognosis for people with cardiac conditions is usually poor and has a mortality of 25%.
How can I prevent this?

Patient education is very important!
Education may have a vital impact on prevention and recurrence of syncope.
Certain maneuvers can be tried which may stop or delay vasovagal syncope.
These maneuvers include:
- Crossing the legs while tensing the leg muscles, abdominal muscles, and buttock
- Clenching your fists or gripping a stress ball
- Gripping your arms while trying to pull them apart
- Avoid standing up too quickly
- Sit near the aisle so you can leave if you feel faint
- Increase your salt and potassium intake
- Drink plenty of fluids
- Do not skip meals
- Do not stand for a long period of time
- Avoid alcohol and caffeine
- Review your medications with your doctor
Safety Issues:
Patients who present with syncope are instructed not to drive until cleared by the doctor to do so.
This is recommended mostly for patients with no warning signs of syncope.
Updated February 2, 2012 -JS
For further information please contact Cardiovascular Interventions, PA at 407-894-4880
Valve Disease
- Mitral Regurgitation
- Mitral Valve Prolapse
- Mitral Stenosis
- Bacterial Endocarditis Guidelines
- Indications for Bacterial Endocarditis Prophylaxis
Mitral Regurgitation
What is it?
Mitral regurgitation is a common valve disorder in which blood flows backwards through the mitral valve during the contraction of the heart and is commonly referred to as a ‘leaky valve’. This reduces the amount of blood flow that is pumped out to the body.
If the leak is small and does not progress, the leak has no significant complication, although severe mitral regurgitation can lead to symptoms of fatigue, breathlessness, and complications.

What are the causes?
The causes of mitral regurgitation include:
- Mitral valve prolapse- occurs when the mitral valve leaflet tissue is deformed and elongated, therefore the leaflets to not approximate together normally
- Infective endocarditis- an infection of the heart valves caused by bacteria, fungi, or other organisms. These organisms stick to the heart valves and grow into vegetations. Vegetations prevent the valve from closing properly, allowing blood to leak backwards
- Rheumatic fever- condition in which bacteria named group A Streptococcus, the bacteria causing Strep throat, goes untreated leading to inflammation of the heart valves
- Trauma- breakage of the chords which hold the mitral valve leaflets in place
- Congenital heart abnormalities
- Coronary artery disease and Heart attack
- Chronic hypertension (high blood pressure)
- Heart tumors
- Mitral annular calcification
- Systemic lupus erythematosus, Marfan’s syndrome, ankylosing spondylitis
- Cardiomyopathies
How does a normal heart work?
The heart consists of four chambers. The top two chambers (atria) receive blood while the bottom two chambers (ventricles) pump blood out.
Deoxygenated venous blood returns to the right atrium of the heart. Blood flows from the right atrium to the right ventricle by the opening of the tricuspid valve. Blood is prevented from going backwards into the atrium by the closure of the tricuspid valve. The right ventricle then contracts and pumps blood out through the pulmonary valve into the pulmonary artery. The pulmonary valve is closed and the blood is sent to the lungs where it picks up oxygen.
Oxygenated blood returns from the lungs by the pulmonary veins and enters the left atrium. From the left atrium, blood enters the left ventricle by the opening of the mitral valve. Blood is prevented from flowing back into the left atrium by the closure of the mitral valve. Blood is then pumped out the left ventricle to the aorta by the opening of the aortic valve. Blood is prevented from flowing back into the left ventricle by the closure of the aortic valve. From the aorta, oxygenated blood is pumped out to the entire body.


What happens in mitral regurgitation?
The mitral valve is a funnel shaped valve with the apex in the left ventricle. The mitral valve consists of two leaflets (cusps) which connect to a ring called the mitral annulus. Heart valves only open in one direction. Once all of the blood has emptied out of the left atrium, the mitral valve tightly closes to ensure no backflow of blood.
In mitral regurgitation the leaflets become weakened, preventing the valve to completely close, causing a back flow of blood into the left atrium. The complete emptying of oxygenated blood from the left atrium is impaired, increasing pressure in the atrium, pulmonary vasculature, and the right side of the heart. Blood is limited from entering the ventricle; therefore blood is limited to fill the rest of the body with oxygenated blood, which can result in symptoms of fatigue and shortness of breath.

What are the risk factors?
Several factors can increase the risk of mitral regurgitation.
Risk factors include:
- Age- by middle age, many people have some mitral regurgitation caused by the normal wear and tear process of the valves. Symptoms are seen in only a small amount of older patients
- History of mitral valve prolapse or mitral stenosis
- Previous heart attack
- History of rheumatic fever and recurrent strep infections
- Congenital heart defects- born with an abnormal mitral valve
What are the symptoms?
Patients with mitral regurgitation may have no symptoms at all. Mitral regurgitation ranges from mild to severe.
Symptoms typically do not occur in mild to moderate mitral regurgitation, or even in severe mitral regurgitation. Symptoms may develop if complications occur.
Symptoms include:
Cough
- Weakness
- Fatigue
- Light-headedness
- Shortness of breath, especially with increased physical activity or when lying flat
- Swollen feet and ankles (pedal edema)
What are the complications?
Complications of mitral regurgitation include:
- Atrial fibrillation
- Blood clots (systemic thromboembolism)
- Infective endocarditis
- Heart failure
- Pulmonary hypertension
Call your doctor immediately if you have any of the following:
Call your doctor if you develop symptoms which are suggestive of mitral regurgitation or another problem associated with your heart.
Most signs and symptoms of mitral regurgitation are due its complications, including arrhythmias or heart failure.
These symptoms include:
- Fatigue
- Shortness of breath during physical activity
- Heart palpitations
- Chest pain
How is it diagnosed?
Mitral regurgitation is usually diagnosed during a routine visit with your physician.
Diagnosis is based around three parameters including patient history, physical examination, and tests.
1. Patient history
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Patients may give history of rheumatic fever, although many may not recall a history of rheumatic fever usually because mitral defects manifests later on in life
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Patients may complain of symptoms of shortness of breath in states with an increased heart rate
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History of hoarseness of voice may be told due to compression of a nerve by an enlarged heart
2. Physical examination
During a routine physical examination, your health care provider will notice the following:
- Distended neck veins may be noticed
- During auscultation a specific type of murmur will be heard caused by the sound of turbulent blood flowing backwards through the mitral valve.
3. Tests
The following tests may be performed:
- Electrocardiogram (EKG) - a noninvasive test performed in our clinic which determines the electrical activity of your heart. This test may be used to detect left atrial enlargement, and commonly atrial fibrillation.
- Chest X-ray- a simple test performed in our office showing the size and shape of the heart to determine whether the left atrium is enlarged. Also reveals the presence of fluid in the lungs which may occur due to mitral regurgitation.
- Cardiac color-Doppler study- use to confirm the presence and determine the severity of mitral regurgitation and cause of mitral regurgitation.
- Echocardiogram (ECHO)- a noninvasive test using sound waves to determine the size and function of the heart’s chambers and the structure and function of the heart valves.
- Transesophageal echocardiogram (TEE)
How is it treated?
Mitral valve regurgitation treatment depends on how severe your condition is.
People with significant (moderate to severe) mitral regurgitation should have routine monitoring to determine if and when treatment is required.
The goal of treatment is to improve the heart function, minimize signs and symptoms and avoid complications.
Lifestyle modification
Lifestyle modifications are used to minimize the factors contributing to mitral regurgitation (i.e. high blood pressure).
This includes:
- Salt restriction- a low-salt diet is required to control blood pressure and reduce pulmonary congestion if present
- Maintain a healthy weight- with a BMI <25
- Limit alcohol
- Do not smoke
- Exercise- for at least 30 minutes a day 5 days a week
The 2006 American College of Cardiology guidelines state that no exercise restrictions are needed for people who have the following:
- No symptoms of mitral regurgitation
- Normal heart rhythm
- Normal size left ventricle and left atrium
- Normal pulmonary artery pressure
Medications
Medications will not correct a defective mitral valve, although medications can reduce the symptoms by reducing the workload of the heart and regulating the heart rhythm.
The goal of medical treatment is to reduce the recurrence of rheumatic fever, provide prophylaxis for infective endocarditis, reduce symptoms of pulmonary congestion, control the heart rate and rhythm, and prevent complications.
- Diuretics- these are sometimes referred to as ‘water pills’ which helps excrete excess water
- Anticoagulants- these are sometimes referred to as “blood thinners” which prevent blood clots from forming and traveling to other parts of the body
- Beta blockers- this lowers the heart rate and decreases blood pressure
- Anti-arrhythmic medications- used to treat atrial fibrillation or other rhythm disturbances associated with mitral regurgitation
- Cholesterol lowering medications- these act by decreasing the amount of cholesterol in blood, especially Lowering your low density lipoprotein (LDL- your Lousy cholesterol) and keep your high density lipoprotein (HDL- your Healthy cholesterol) High
- Antibiotics- some patients who have had rheumatic fever may need long-term treatment with penicillin
Procedures
To treat mitral regurgitation, valve repair or valve replacement may be necessary.
The treatment of choice for people with severe mitral regurgitation is surgical repair or replacement of the mitral valve.
- Mitral valve repair: repair of the mitral valve is preferred over mitral valve replacement. This procedure improves ventricular function and does not require lifelong therapy with an anticoagulant when compared to valve replacement. During valve repair, the valve will be reshaped to prevent or reduce the backwards flow of blood.
- Mitral valve replacement: valve replacement is done when repair is not possible. The entire mitral valve will be replaced by a surgeon. A mechanical (metal) or tissue valve (organic) may be used to replace the impaired valve. A mechanical valve requires life-long anticoagulation therapy such as warfarin (Coumadin) to prevent blood clot formation. Tissue valves usually come from a pig or cow. Blood does not clot easily on tissue valves so patients may only need Coumadin or aspirin for only a short time. Tissue valves may wear out faster than mechanical valves and may need to be replaced sooner.
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Mechanical Valve |
Tissue Valve |
What is the prognosis?
The outcome varies depending on the condition of the patient. Symptoms are usually controlled with medications.
The clinical outcomes are greatly improved in patients with severe mitral regurgitation who undergo surgical or percutaneous interventions.
There is also a risk that the new valve will stop working and may need to be replaced.
How can I prevent this?
Mitral regurgitation itself often can not be prevented, although some of the complications may be prevented.
- Prevent rheumatic fever: See a physician when you have a sore throat. Treating strep infections promptly with antibiotics can prevent rheumatic fever that causes mitral regurgitation.
- Reduce risk factors of coronary artery disease: Lower blood pressure, obesity, high cholesterol, and sugar. It is a good idea to m




























